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Phillip A. Alviola,Marnelli S. Alviola,Kirk J. Taray,Cristian C. Lucañas,Anna Pauline O. de Guia,Aimee Lynn B. Dupo,Virginia C. Cuevas,Nelson M. Pampolina,Ireneo L. Lit Jr. 국립중앙과학관 2023 Journal of Asia-Pacific Biodiversity Vol.16 No.3
Food habits of eight insectivorous bat species from Puting Bato Cave Complex, Polillo Island, wereexamined. Fecal samples collected from eight species of cave-dwelling insectivorous bats containedculled fragments from seven prey taxa (six insect orders and one fish prey). Lepidoptera, Coleoptera, andHymenoptera were the most consumed group in both percentage volume and percentage frequency. Thediet of Hipposideros diadema, H. pygmaeus, Rhinolophus arcuatus, and R. philippinensis mostly concurswith previous studies but with varying proportions. Baseline information on the diets of H. coronatus,M. paululus, R. macrotis, and R. rufus is provided in this study.
EBSD and reconstruction of pre-transformation microstructures, examples and complexities in steels
Abbasi, M.,Kim, D.I.,Nelson, T.W.,Abbasi, M. Elsevier 2014 Materials characterization Vol.95 No.-
Electron backscattered diffraction has provided a quantitative tool to study micro/nano-structures in large scales. A recent application of electron backscattered diffraction is the reconstruction of pre-transformed phases in polymorphic systems, especially when there is no retained pre-transformed phase at room temperature. This capability has been demonstrated by various researchers utilizing different approaches towards grain structure and orientation recovery. However, parameters affecting reconstruction have not been investigated systematically. Factors such as post-transformed microstructures (morphology and crystallography), lattice strain (deformation), pattern and sample quality are among the affecting factors. Two-dimensional datasets of different steels have been reconstructed along with a limited 3-dimensional dataset in the current paper. Preliminary results intended for large-scale automatic reconstructions have been presented. They indicate that the successfulness of reconstruction is strongly dependent on the post-transformed microstructure. Factors such as morphology, grain size, variant selection, and deformation play roles. Few examples of reconstruction complexity at prior austenite boundaries leading to uncertain results are presented. Lastly, reconstructions are discussed in terms of meaningfulness and if they correctly represent pre-transformed grains and orientations.
Baek, Ji Yeon,Morris, Shelli M.,Campbell, Jean,Fausto, Nelson,Yeh, Matthew M.,Grady, William M. Wiley Subscription Services, Inc., A Wiley Company 2010 International journal of cancer: Journal internati Vol.127 No.5
<P>Hepatocellular carcinoma (HCC) results from the cumulative effects of deregulated tumor suppressor genes and oncogenes. The tumor suppressor and oncogenes commonly affected include growth factors, receptors and their downstream signaling pathway components. The overexpression of transforming growth factor alpha (TGF-α) and the inhibition of TGF-β signaling are especially common in human liver cancer. Thus, we assessed whether TGF-α overexpression and TGF-β signaling inactivation cooperate in hepatocarcinogenesis using an in vivo mouse model, MT1/TGFa;AlbCre/Tgfbr2<SUP>flx/flx</SUP> mice (“TGFa;Tgfbr2<SUP>hepko</SUP>”), which overexpresses TGF-α and lacks a TGF-β receptor in the liver. TGF-β signaling inactivation did not alter the frequency or number of cancers in mice with overexpression of TGF-α. However, the tumors in the TGFa;Tgfbr2<SUP>hepko</SUP> mice displayed increased proliferation and increased cdk2, cyclin E and cyclin A expression as well as decreased Cdkn1a/p21 expression compared to normal liver and compared to the cancers arising in the TGF-α overexpressing mice with intact TGF-β receptors. Increased phosphorylated ERK1/2 expression was also present in the tumors from the TGFa;Tgfbr2<SUP>hepko</SUP> mice and correlated with downregulated Raf kinase inhibitor protein expression, which is a common molecular event in human HCC. Thus, TGF-β signaling inactivation appears to cooperate with TGF-α in vivo to promote the formation of liver cancer that recapitulates molecular features of human HCC.</P>