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The Effect of Gender Leadership with Political Capital in the CSR Performance of China Listed Firms
Tzu-Yu LIN,Sheng-Hsiung CHIU,Ruijun WU,Ziyu XIAO 한국유통과학회 2018 KODISA ICBE (International Conference on Business Vol.2018 No.-
This paper investigates the effect of gender leadership with political connection on CSR performance using annual data of Chinese firms who listed in China A-share stock market and had CSR rating assessed by Rankins from 2009 to 2015. In addition, we also examine whether the foregoing question is under the influence of particular ownership structure in China. Our empirical results suggest that female chairman or CEO would not perform well in CSR activities, while leader’s political capital acts an exacerbating force. Specifically, the negative and statistically significant of interaction term female leadership with political capital is obviously identified for the Non-SOEs. The mandatory CSR reporting would not encourage firms to make more effort on CSR activities to the purpose on generating positive social externalities, while the advantages of CSR rating in firms are not obviously experience to coordinate the conflict of interest between stakeholders.
Li Cui,Weiquan Bu,Jie Song,Liang Feng,Tingting Xu,Dan Liu,Wenbo Ding,Jianhua Wang,Changyang Li,Binge Ma,Yi Luo,Ziyu Jiang,Chengcheng Wang,Juan Chen,Jian Hou,Hong-mei Yan,Lei Yang,Xiao-bin Jia 대한약학회 2018 Archives of Pharmacal Research Vol.41 No.3
Alantolactone is a sesquiterpene lactone isolatedfrom Inula helenium L. Although alantolactone possessesanti-inflammation and apoptosis-induction activities, theunderlying mechanism of anti-cancer effect on humanbreast cancer cells remains largely unknown. In this study,we explored the possibility of alantolactone as an apoptosis-inducing cytotoxic agent using MDA-MB-231 cells asin vitro model. Alantolactone significantly induced itsapoptosis, demonstrated by cell cycle analysis, annexinV-APC/7-AAD double staining and dUTP nick end labeling. Additionally, alantolactone triggered the mitochondrial-mediated caspase cascade apoptotic pathway, whichwas confirmed by increased Bax/Bcl-2 ratio, loss of MMP,release of cytc from mitochondria to cytoplasm, activationof caspase 9/3, and subsequent cleavage of PARP. Z-VADFMKpartially prevented apoptosis induced by alantolactone. Alantolactone provoked the production of ROS, whileNAC (a scavenger of ROS) reversed alantolactone-mediateddepolarization of MMP and apoptosis. Alantolactonemodulated the activities of MAPKs. As expected, cotreatmentwith SB203580, SP600125 or U0126 could reducedthe apoptotic rate. Furthermore, alantolactone decreasedthe protein expressions of p-NF-kB p65 and p-STAT3,increased p-c-Jun level in a dose-dependent manner. Thesefindings suggested that alantolactone possessed anticanceractivity via ROS-mediated mitochondrial dysfunctioninvolving MAPK pathway, and had an effect on the transcriptionfactors of NF-kB, AP-1 and STAT3.