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Physiological responses of yellow‑horn seedlings to high temperatures under drought condition
Hua Jin,Jixiang Zou,Linlin Li,Xinlei Bai,Tong Zhu,Junbao Li,Bing-Cheng Xu,Zhi Wang 한국식물생명공학회 2020 Plant biotechnology reports Vol.14 No.1
Drought and hot stresses are the primary limiting factors for the growth and seed production of yellow-horn (Xanthoceras sorbifolia Bunge), especially in Xinjiang Province of China. A better understanding of its physiological and biochemical characteristics under drought–hot stress is requisite for its efficient cultivation in the arid and semi-arid areas. In this study, the physiological and biochemical responses of 3-month-old yellow-horn seedlings were evaluated after 7 days of treatments of five temperatures (25 °C/20 °C, 30 °C/25 °C, 35 °C/25 °C, 40 °C/25 °C and 45 °C/25 °C) under two watering conditions [adequate water supply (HW): 80 ± 5% FC (field capacity); low water supply (LW): 40 ± 5% FC] by a pot experiment. The increased temperature (above to 35 °C) markedly aggravated the damage on biomass accumulation and membrane integrity of yellow-horn seedlings under the LW. The accumulation of soluble sugars and proteins exhibited a growing trend as the temperature increased from 25 to 40 °C, which declined when the temperature was higher than 40 °C except for the soluble protein accumulation under HW. In addition, captured responsive characteristics of the gas-exchange parameters and chlorophyll fluorescence have indicated that the combinations of high temperature (above to 35 °C) and LW induced significant decrease in photosynthetic activities of yellow-horn seedlings. All these results showed that the hot stress significantly aggravated the drought damage on yellow-horn plant growth, especially when the temperature increased above 35 °C under drought stress.
N-doped porous carbon encapsulated MnFe2O4 nanoparticles as advanced anodes for Li-ion batteries
Taolin Zhao,Xinlei Zhang,Zezheng Liu,Qingyuan Gu,Xiaoyu Jin,Saihu Xie,Shuai Liu 대한금속·재료학회 2024 ELECTRONIC MATERIALS LETTERS Vol.20 No.3
Transition metal oxide MnFe 2 O 4 is considered a promising anode material for Li-ion batteries owing to its high theoreticalspecifi c capacity. However, this material has two bottleneck problems, i.e., poor conductivity and serious volume expansionduring cycling. In this work, MnFe 2 O 4 nanoparticles were successfully encapsulated in the matrix of N-doped porouscarbon via a sol–gel method. As a result, the N-doped carbon matrix enhances the electronic conductivity of the composites. The special porous structure increases the contact area between the electrode material and the electrolyte and facilitates therapid infi ltration of the electrolyte. At a calcination temperature of 400 °C, the MnFe 2 O 4 /C composite shows a high initialdischarge specifi c capacity of 1207.0 mAh g −1 at 0.2 A g −1 and retains a reversible specifi c capacity of 1100.1 mAh g −1after 200 cycles. The simple design of metal oxide nanomaterials encapsulated in N-doped porous carbon provides a newdirection for improving the electrochemical performance of electrode materials for Li-ion batteries.
Park, Sunho,Kim, Seung-Jun,Yu, Donghyeon,Peñ,a-Llopis, Samuel,Gao, Jianjiong,Park, Jin Suk,Chen, Beibei,Norris, Jessie,Wang, Xinlei,Chen, Min,Kim, Minsoo,Yong, Jeongsik,Wardak, Zabi,Choe, Kevin Oxford University Press 2016 Bioinformatics Vol.32 No.11
<P>Motivation: Identification of altered pathways that are clinically relevant across human cancers is a key challenge in cancer genomics. Precise identification and understanding of these altered pathways may provide novel insights into patient stratification, therapeutic strategies and the development of new drugs. However, a challenge remains in accurately identifying pathways altered by somatic mutations across human cancers, due to the diverse mutation spectrum. We developed an innovative approach to integrate somatic mutation data with gene networks and pathways, in order to identify pathways altered by somatic mutations across cancers. Results: We applied our approach to The Cancer Genome Atlas (TCGA) dataset of somatic mutations in 4790 cancer patients with 19 different types of tumors. Our analysis identified cancer-type-specific altered pathways enriched with known cancer-relevant genes and targets of currently available drugs. To investigate the clinical significance of these altered pathways, we performed consensus clustering for patient stratification using member genes in the altered pathways coupled with gene expression datasets from 4870 patients from TCGA, and multiple independent cohorts confirmed that the altered pathways could be used to stratify patients into subgroups with significantly different clinical outcomes. Of particular significance, certain patient subpopulations with poor prognosis were identified because they had specific altered pathways for which there are available targeted therapies. These findings could be used to tailor and intensify therapy in these patients, for whom current therapy is suboptimal.</P>