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Sung Nae Cho,Kyu Sang Kim 한국물리학회 2013 Current Applied Physics Vol.13 No.7
Effect of Si-doping on InGaN layers below the quantum wells (QWs), which cause different levels of charge concentration in the depletion region, have been investigated for InGaN light emitting diodes (LEDs). Four groups of InGaN LEDs with different levels of Si-doping on InGaN/GaN layers below quantum-wells have been produced for the experiment (i.e., 0.5 x 1017 cm-3 for group A, 1 x 1017 cm-3for group B, 5 x 1017 cm-3 for group C, and 1 x 1018 cm-3 for group D.) The reverse leakage current of LED can be significantly decreased and the light output power of LED can be enhanced by lowering the background charge concentration in the depletion region of LED. Such result enables us to improve the device lifetime by inhibiting the degradation of the GaN-based LED.
The Prevalence of Colonic Neoplasm in Cryptogenic Pyogenic Liver Abscess: A Prospectively Enrolled
( Nae-yun Heo ),( Tae Oh Kim ),( Young Soo Moon ),( Sung Yeun Yang ),( Seung Ha Park ),( Jong Ha Park ),( Joon Hyuk Choi ),( Sung-min Kim ),( Ki Tae Yoon ),( Young Mi Hong ),( Mong Cho ) 대한간학회 2016 춘·추계 학술대회 (KASL) Vol.2016 No.1
Aims: Several studies suggested that pyogenic liver abscess (PLA) was associated with colon neoplasm. Thus, colonoscopic exam for cryptogenic PLA might present the hidden colonic neoplasm, through which the intestinal flora can transmit into the liver. However, there is no prospectively enrolled cross-sectional data for colonic neoplasm in cryptogenic PLA, yet. Methods: The patients with PLA were prospectively enrolled in two university hospitals. Among them, in case of cryptogenic PLA, the all patients were recommended to perform the colonoscopic exam for detection of colonic neoplasm. Results: One hundred eighty three patients with PLA were enrolled for 22 months. Among them, 101 (55.2%) patients did not have a definite cause of liver abscess at initial evaluation. The maximal diameter of the largest lesion was 5.7 (1.0-14.0) cm, and 74.3% of the patients were treated by percutaneous abscess drainage. 91% of the patients who had an identified pathogen yielded Klebsiella. Sixty two patients had colonoscopic exam, and no one have a colonic neoplasm. Fifty patients had esophagogastroduodenoscopic exam, and 9 have a gastric ulcer and one did esophageal ulcer, and another one did hemorrhagic gastritis. Conclusions: The prevalence of colonic neoplasm among the patients with cryptogenic PLA was not as high as the previous studies. The further well-designed and large scale studies are required to confirm the association of the colon neoplasm and cryptogenic PLA.
Detection of Mycobacterium tuberculosis Antigens in Sputum Samples from Tuberculosis Patients
( Sang Nae Cho ),( Sun Hee Baik ),( Sun Park ),( Joon Chang ),( Se Kyu Kim ),( Sung Kyu Kim ),( Won Young Lee ),( Yun Sop Chong ),( Chul Ho Cho ),( Jin Joo Kim ),( Joo Deuk Kim ) 대한결핵 및 호흡기학회 1994 Tuberculosis and Respiratory Diseases Vol.41 No.6
Cho, Jang-Eun,Kim, Yoon-Suk,Park, Sang-Jung,Cho, Sang-Nae,Lee, Hye-Young Korean Society for Molecular and Cellular Biology 2010 Molecules and cells Vol.29 No.1
Chemokines function in the migration of circulating leukocytes to regions of inflammation, and have been implicated in chronic inflammatory conditions including myco-bacterial infection. We investigated whether Leukotactin-1 (Lkn-1), a novel member of the CC-chemokines, is involved in the immune response of macrophages against Myco-bacterium tuberculosis (MTB). In PMA-differentiated THP-1 cells, MTB infection increased mRNA expression of Lkn-1 in a dose-dependent manner. Lkn-1 induction peaked 12 h after infection, then declined gradually and returned to its basal level at 72 h. Secretion of Lkn-1 was elevated by MTB infection. The increase in expression and secretion of Lkn-1 caused by MTB was reduced in cells treated with inhibitors of phosphatidylinositol 3-kinase (PI3-K), 3-phosphoinositide-dependent kinase 1 (PDK1) and Akt. MTB-induced Akt phosphorylation was blocked by treatment with a PI3-K inhibitor or a PDK1 inhibitor, implying that PI3-K, PDK1, and Akt are associated with the signaling pathway that up-regulates Lkn-1 in response to MTB. These results suggest that Lkn-1 is novel member of the group of chemokines that is released by macrophages infected with MTB.