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Cytokine and Tumor Environments
Jin Tae Hong1*, Ju Kyung Song, Sun Mi Kwon, Byung Woo Ahn, Youngsoo Kim, Sang Bae Han 충북대학교 동물의학연구소 2012 Journal of Biomedical and Translational Research Vol.13 No.1
Cytokines are known to function as regulatory molecules that can be produced by virtually every nucleated cell type in the body, including lymphocytes, monocytes/macrophages, epithelial cells, fibroblasts, and many others. Cytokines include lymphocyte-derived factors (lymphokines), monocyte-derived factors (monokines), hematopoietic factors (colony-stimulating factors), connective tissue/growth factors, and chemotactic chemokines. Cytokines released in response to infection can affect tumor development in different ways. When exposed to infectious agents, cytokines are secreted by sentinel cells, such as macrophages and dendritic cells. These cytokines include interleukin 1 (IL-1) and tumor necrosis factor-α, as well as others, such as IL-6, IL-12, and IL-18. When released in sufficient quantities, these molecules can cause inflammation. Chronic inflammation is highly associated with tumor initiation, promotion, and progression. In this article, we review the roles and mechanisms of cytokines in tumor development.
Chun, Yoon-Keun,Ha, Joo-hun,Hong-Jung-Woo,Oh, Soo-Myung,Kim, Sung-Soo 경희대학교 동서의학연구소 1999 INTERNATIONAL SYMPOSIUM ON EAST-WEST MEDICINE Vol.1999 No.1
Yoon-Keun Chun¹,Joohun Ha□Hong-Jung Woo□, Soo Myung Oh□,Sung Soo Kim□ ¹Department of Molecular Biology, College of Medicine,²Department of Surgery, college of Medicine,³Department of Internal Medicine, College of Oriental Medicine,and ⁴East-Weat Medical Reserch Institute,Kyung Hee University, Seoul, Korea. The HBV DNA Amounts in Serum Have No relationship with ALT level and Hetergeneous Population Coexits in Chronic Hepatitis B Virus Infection. Proceedings of International Symposium on East-West Medicine, Seoul. 212-230, 1999. -Hepatitis B is caused by hepadnavirus. Hepatitis B virus replicates through 3.5kb pregenomic RNA intermediate which is regulated by core promoter. Pathogenesis of hepatitis B virus has been bilieved the result of host immune response. But recently many studies have reported that high level of viral replication caused by mutation in core promoter might result in severs hepatitis. But these studies were performed in vitro, not in vivo. So there is yet debate about which factor, viral of host factor, is more important in pathogenesis of hepatitis B virus. So we measured real viral replication level in 204 chronic hepatitis B patients by quantifying HBV DNA from sera by our novel PCR-based more sensitive method, and compared these results with ALT level measured from same sera, which indicates liver cell damage. Surprisingly there are no significant correlation between HBV DNA quantity and ALT level. Then we cloned core promoter region. In SSCP, we found that many viral mutants coexist in one patient. Base on SSCP result, we chose main viral core promoter type in each patients, which is thought to determine overall viral replication level in this patient. Main type of core promoter region of each 41 patients were directly sequenced. And with these we measured promoter activity by luciferase assay system and compared promoter activity with on another. We found tha there were some differences in promoter activity according to core promoter sequences. And we constructed replication-competent viral constructs with core promoter from 41 patients and Transfected these into HepG2 cell and measured HBV DNA by southern blot. There were also differences in HBV DNA quantity according to core promoter sequences. On these all results we investigated correlation between the effect of HBV core promoter on viral replication in vitro and HBN DNA quantity, ALT level from sera of each patients. We found there is no significant correlation among them. As a result, we concluded that in determining severity chronic hepatitis B patients, host factors of each patient is more important rather than replicative activity of virus itself.
DETERMINATION OF EFFICIENCY OPTIMAL OPERATING POINT FOR VANE-SPEED FLOW CONTROL SYSTEM
Sung-Kwang Hur,Hong-Woo Rhew,Min-ho Park 전력전자학회 1989 ICPE(ISPE)논문집 Vol.- No.-
It cannot always be said that applying varlable speed drives to parallel operation fans are the best choice or the most cost-effective. Depending on the conditions of operating and drive capacity, the vane - speed control will be more favorable than others. In this case, there are lots of combination operating points between the vane opening and the fan speed. The efficiency optimal operating point for a vane - speed parallel fan system is calculated and tested to the forced draft fans in a power plant boiler The results ascertain the validity of the theoretical analysis.<br/>
Poster Session 1 : Role of carboxy-terminus of p23 in the regulation of its physiological functions
( Sang Hyeok Woo ),( Sung Kwan An ),( Hyeon Ok Jin ),( Hyung Chahn Lee ),( Sung Keum Seo ),( Doo Hyun Yoo ),( Chang Hun Rhee ),( Seok Il Hong ),( In Chul Park ) 한국생화학분자생물학회 (구 한국생화학회) 2005 생화학분자생물학회 춘계학술발표논문집 Vol.2005 No.-
Modeling and Control of IGBT Converter-Based High-Voltage Direct Current System
Hong-Woo Kim,Suk-Whan Ko,Hae-Joon An,Gil-Soo Jang,Hee-Sang Ko 한국조명·전기설비학회 2011 조명·전기설비학회논문지 Vol.25 No.7
This paper presents modeling and control for the emerging IGBT converter-based high-voltage direct-current system (IGBT-HVDC). This paper adds to the representation of the IGBT-HVDC system in the dq-synchronous reference frame and its decoupled control scheme. Additionally, since the IGBT-HVDC is able to actively support the grid due to its capacity to control independently active and reactive power production, a reactive power control scheme is presented in order to regulate/contribute to the voltage at a remote location by taking into account its operational state and limits. The ability of the control scheme is assessed and discussed by means of simulations using ahybrid power system, which consists of a permanent magnetic synchronous-generator (PMSG) based wind turbine, an IGBT-HVDC, and a local load.
Inhibitory Effects of B-HT 920 on Gastric Acid Secretion Induced by Vagal Stimulation in Rat
Hong, Sung-Cheul,Park, Mi-Sun,Chung, Joon-Ki,Kang, Maeng-Hee,Choi, Su-Kyung,Kim, Myung-Woo The Pharmaceutical Society of Korea 1989 Archives of Pharmacal Research Vol.12 No.4
Effects of B-HT 920 on the vagally stimulated gastric acid secretion were studied in anesthetized and gastric fistula rats. When the gastric acid secretion was increased by stimulation of the vagus nerve, B-HT 920 was partially attenuated by prazosin, $\alpha_1-$adrenoceptor antagonist and virtually abolished by yohimbine, $\alpha_2-$adrenoceptor antagonist. On the other hand, when the gastric acid secretion was increased by the infusion of bethanechol, a muscarinic parasympathetic stimulant, B-HT 920 had no effect on the bethanechol-induced gastric acid secretion. These results suggest that B-HT 920 inhibits vagally induced gastric acid secretion by activation of presynaptic $\alpha-$adrenoceptors located on the vagally stimulated pathways in the gastric wall and this effect of B-HT 920 is more related to $\alpha_2-$adrenoceptors than $\alpha_1-$adrenoceptors.