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History of Bioelectrical Study and the Electrophysiology of the Primo Vascular System
Park, Sang Hyun,Kim, Eung Hwi,Chang, Ho Jong,Yoon, Seung Zhoo,Yoon, Ji Woong,Cho, Seong-Jin,Ryu, Yeon-Hee Hindawi Publishing Corporation 2013 Evidence-based Complementary and Alternative Medic Vol.2013 No.-
<P><I>Background</I>. Primo vascular system is a new anatomical structure whose research results have reported the possibility of a new circulatory system similar to the blood vascular system and cells. Electrophysiology, which measures and analyzes bioelectrical signals tissues and cells, is an important research area for investigating the function of tissues and cells. The bioelectrical study of the primo vascular system has been reported by using modern techniques since the early 1960s by Bonghan Kim. This paper reviews the research result of the electrophysiological study of the primo vascular system for the discussion of the circulatory function. We hope it would help to study the electrophysiology of the primo vascular system for researchers. This paper will use the following exchangeable expressions: Kyungrak system = Bonghan system = Bonghan circulatory system = primo vascular system = primo system; Bonghan corpuscle = primo node; Bonghan duct = primo vessel. We think that objective descriptions of reviewed papers are more important than unified expressions when citing the papers. That said, this paper will unify the expressions of the primo vascular system.</P>
단일공법 복강경하 질식자궁절제술 110예의 고찰 및 다공법과의 비교
박병준 ( Byung Joon Park ),김용욱 ( Yong Wook Kim ),노덕영 ( Duck Yeong Ro ),김태응 ( Tae Eung Kim ),류기성 ( Ki Sung Ryu ),김장흡 ( Jang Heup Kim ) 대한산부인과학회 2010 Obstetrics & Gynecology Science Vol.53 No.7
Objective: To evaluate the safety and feasibility of single-port access laparoscopically assisted vaginal hysterectomy (SPA-LAVH) using conventional laparoscopic instruments compared to multi-port access laparoscopically assisted vaginal hysterectomy (MPA-LAVH). Methods: We reviewed the medical records of 220 patients with uterine leiomyoma or adenomyosis who underwent 110 SPA-LAVH and 110 MPA-LAVH in Incheon St. Mary`s Hospital between April 2007 and November 2009. We performed SPA-LAVH with conventional rigid straight laparoscopic instruments in all cases. We also performed a new vaginal cuff closure method, Kim`s Vaginal Vault Suspension Method, named after the operator (Kim, YW) in both SPA-LAVH and MPA-LAVH. Results: There was no significant difference in patients` age, operating time, uterine weight, hemoglobin change, frequency of blood transfusion, and incidence of postoperative fever between the two groups. The patients` mean age was 46.1±7.0 years (SPA-LAVH) and 45.5±6.3 years (MPA-LAVH). The mean operating time was 87.2±21.0 minutes (SPA-LAVH) and 83.3±20.3 minutes (MPA-LAVH). The mean uterine weight was 261.4±139.7 g (SPA-LAVH) and 257.8±132.9 g (MPA-LAVH). The mean hemoglobin change was 1.1±0.7 g/dL (SPA-LAVH) and 1.2±0.6 g/dL (MPA-LAVH). Neither bowel injury nor urinary tract injury occurred during the operation in the two groups. One of the SPA-LAVH and one of the MPA-LAVH cases were converted to abdominal total hysterectomy. The mean hospital stay time was shorter with SPA-LAVH (2.6±0.6 days [SPA-LAVH] and 3.3±0.7 days [MPA-LAVH], P<0.05). Conclusion: SPA-LAVH using conventional rigid straight laparoscopic instruments can be offered as a safe and feasible alternative to MPA-LAVH.
Kim, Sol,Lee, Jungwoon,Kim, Ja Young,Lim, Bobae,Shin, Eung-Kyun,Han, Yong-Mahn,Kim, Sung-Su,Song, Jin-Ho,Kim, Jungho Wiley Subscription Services, Inc., A Wiley Company 2009 International journal of cancer: Journal internati Vol.124 No.10
<P>The EWS-Oct-4 protein is a chimeric molecule in which the amino terminal domain (NTD) of the EWS becomes fused to the carboxy terminal domain (CTD) of the Oct-4 transcription factor. It was identified in human bone and soft-tissue tumors associated with t(6;22)(p21;q12). Using in vitro and in vivo systems, we found that the EWS-Oct-4 protein self-associates. The major domains required for self-association mapped to the EWS NTD (amino acids 70–163) and the POU DNA-binding domain. EWS-Oct-4 protein also associated with EWS-Oct-4 (V351P), which contains a mutation in the POU DNA-binding domain. Using electrophoretic mobility shift assays, we found that the EWS-Oct-4 (V351P) mutant interfered with wild-type EWS-Oct-4 DNA-binding activity. In addition, we found that EWS-Oct-4-mediated transcriptional activation was inhibited by EWS-Oct-4 (V351P) protein in vivo. Thus, this mutation in the POU DNA-binding domain results in a dominant negative protein. These findings suggest that the biological functions of the EWS-Oct-4 oncogene can be modulated by the dominant negative mutant EWS-Oct-4 (V351P). © 2008 Wiley-Liss, Inc.</P>
Kim, Tae Eung,Park, Jin Man,Sohn, Sung Woo,Kim, Do Hyang,Kim, Won Tae,Stoica, Mihai,Kü,hn, Uta,Eckert, Jü,rgen The Japan Institute of Metals 2010 MATERIALS TRANSACTIONS Vol.51 No.4
<P>The effect of carbon addition on the microstructure and mechanical properties of Fe-Zr and Fe-Zr-Nb ultrafine eutectic-dendrite composites has been investigated. Addition of carbon leads to the formation of iron solid solution strengthened by carbon interstitials and carbide particles encapsulated by α-Fe dendrites preferentially nucleated on the carbides. Consequently, the ultrafine eutectic Fe-5Zr-5Nb-0.3C (mass%) alloy exhibits high compressive strength (∼1.2 GPa) and large plastic strain (∼24%). The present study suggests another effective way to enhance the mechanical properties of Fe-based nano-eutectic alloys.</P>
Kim, Sun-Gyun,Jang, Soo-Jeong,Soh, Jaemog,Lee, Keesook,Park, Jin-Ki,Chang, Won-Kyong,Park, Eung-Woo,Chun, Sang-Young Association for the Study of Internal Secretions 2009 Endocrinology Vol.150 No.8
<P>Ectodermal neural cortex (ENC) 1, a member of the kelch family of genes, is an actin-binding protein and plays a pivotal role in neuronal and adipocyte differentiation. The present study was designed to examine the gonadotropin regulation and action of ENC1 during the ovulatory process in immature rats. The levels of ENC1 mRNA and protein were stimulated by LH/human chorionic gonadotropin (hCG) within 3 h both in vivo and in vitro. In situ hybridization analysis revealed that ENC1 mRNA was localized not only in theca/interstitial cells but also in granulosa cells of preovulatory follicles but not of growing follicles in pregnant mare's serum gonadotropin/hCG-treated ovaries. LH-induced ENC1 expression was suppressed by a high dose of protein kinase C inhibitor RO 31-8220 (10 microM) but not by low doses of RO 31-8220 (0.1-1.0 microM), suggesting the involvement of atypical protein kinase C. ENC1 was detected in both nucleus and cytoplasm that was increased by LH/hCG treatment. Both biochemical and morphological analysis revealed that LH/hCG treatment increased actin polymerization within 3 h in granulosa cells. Interestingly, ENC1 physically associated with actin and treatment with cytochalasin D, an actin-depolymerizing agent, abolished this association. Confocal microscopy further demonstrated the colocalization of ENC1 with filamentous actin (F-actin). The present study demonstrates that LH/hCG stimulates ENC1 expression and increases F-actin formation in granulosa cells. The present study further shows the physical association of ENC1 and F-actin, implicating the role of ENC1 in cytoskeletal reorganization during the differentiation of granulosa cells.</P>
Eung Chang Kim,Myeong Jong Lee,Sang Yep Shin,Geun Hee Seol,Seung Ho Han,Jaeyong Yee,Chan Kim,Sun Seek Min 대한생리학회-대한약리학회 2013 The Korean Journal of Physiology & Pharmacology Vol.17 No.1
Many intracellular proteins and signaling cascades contribute to the sensitivity of N-methyl-D- aspartate receptors (NMDARs). One such putative contributor is the serine/threonine kinase, protein kinase C (PKC). Activation of PKC by phorbol 12-myristate 13-acetate (PMA) causes activation of extracellular signal-regulated kinase (ERK) and promotes the formation of new spines in cultured hippocampal neurons. The purpose of this study was to examine which PKC isoforms are responsible for the PMA-induced augmentation of long-term potentiation (LTP) in the CA1 stratum radiatum of the hippocampus in vitro and verify that this facilitation requires NMDAR activation. We found that PMA enhanced the induction of LTP by a single episode of theta-burst stimulation in a concentration- dependent manner without affecting to magnitude of baseline field excitatory postsynaptic potentials. Facilitation of LTP by PMA (200 nM) was blocked by the nonspecific PKC inhibitor, Ro 31-8220 (10ՌM); the selective PKCՄ inhibitor, rottlerin (1ՌM); and the PKCՅ inhibitor, TAT-ՅV1-2 peptide (500 nM). Moreover, the NMDAR blocker DL-APV (50ՌM) prevented enhancement of LTP by PMA. Our results suggest that PMA contributes to synaptic plasticity in the nervous system via activation of PKCՄ and/or PKCՅ, and confirm that NMDAR activity is required for this effect.
Kim, Hyeon Soo,Yumkham, Sanatombi,Choi, Jang Hyun,Kim, Eung-Kyun,Kim, Yong Sik,Ryu, Sung Ho,Suh, Pann-Ghill Korean Society for Molecular Biology 2006 Molecules and cells Vol.22 No.1
<P>Haloperidol is a classical neuroleptic drug that is still in clinical use and can lead to abnormal motor activity following repeated administration. However, there is little knowledge of how it triggers neuronal impairment. In this study, we report that it induced calcium ion influx via L-type calcium channels and that the elevation of calcium ions induced by haloperidol appeared to render hippocampal cells more susceptible to oxidative stress. Indeed, the level of cytotoxic reactive oxygen species (ROS) and the expression of pro-apoptotic Bax increased in response to oxidative stress in haloperidol-treated cells, and these effects were inhibited by verapamil, a specific L-type calcium channel blocker, but not by the T-type calcium channel blocker, mibefradil. These findings indicate that haloperidol induces calcium ion influx via L-type calcium channels and that this calcium influx influences neuronal fate.</P>
( Sung Ha Lim ),( Eun Jung Kim ),( Chung Hyuk Lee ),( Gi Hyun Park ),( Kang Min Yoo ),( Sung Ju Nam ),( Kyong-oh Shin ),( Kyungho Park ),( Eung Ho Choi ) 대한피부과학회 2020 대한피부과학회 학술발표대회집 Vol.72 No.1
Background: The stratum corneum (SC) consists of corneocytes, intercellular lipids, and corneodesmosomes. Most of the previous studies have focused on a ceramide with a single chain fatty acid, not with diverse chain lengths. Objective: We evaluated whether a lipid mixture enriched by ceramide NP with fatty acids of diverse chain lengths can restore the skin barrier impaired by topical corticosteroid. Methods: 27 healthy adult males were recruited. Topical corticosteroid was applied on both the forearms and, either the test cream that contains a lipid mixture or a vehicle cream was applied on the left or right forearm, respectively. The functional parameters of the skin barrier, epidermal differentiation markers, hyaluronic acid synthase 3 (HAS3), cytokines, and the lipid profiles in the SC were analyzed. Results: The barrier recovery rate, SC integrity, and SC hydration were significantly improved in the test cream-applied site. Filaggrin and HAS3 levels were significantly higher, Interleukin-1α (IL-1α) levels were increased, and IL-2, IL-6, IL-10, and IL-13 levels decreased in the test cream-applied site. Lipid analysis showed that C18, C20, and total ceramide NP levels increased in the test cream-applied site. C16, C18, C20, C24, and total ceramide NP levels were elevated in the test cream-applied site after acute barrier disruption. Conclusion: A lipid mixture enriched by ceramide NP with fatty acids of diverse chain lengths could recover the barrier impaired by topical corticosteroid.