http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
이계호,이효지,정문교 한국농화학회 1971 Applied Biological Chemistry (Appl Biol Chem) Vol.14 No.3
As a series on the soy-bean protein and their related substances 9 samples were collected from 9 places such as straws (Rice) to obtain bacterial strains which produce protease. From these samples total of 23 strains were isolated by the use of dilution pour plate method. For all isolated strains primary screening of productivity of protease was performed and useful straines with regard to protease productivities were identified. Optimum conditions for enzyme action of protease from isolates D_9, F_(20) strains were pH 7.5 and 40℃. Chung-Kook-Jang is one of the characteristic foods in Korea made from soy-bean by fermentation. The chief bacterium is Bacillus subtilis and the chief change which takes place in soy-bean during fermentation is degradation of protein. Three kinds of Chung-Kook-Jang were prepared using three different strains of Bacillus natto, D_9 and F_(20) from isolated. Water soluble-N, TCA soluble-N, amino-N and peptide-N were measured about the steamed soybean, Chung-Kook-Jang prepared with three strains of bacteria. Water soluble-N decreased very largely in steamed soybean, but in Chung-Kook-Jang it increased to 85% of raw soy-bean.
Identification of AFLP Marker Linked to a SCN Resistant Gene in Soybean
Ko,Mi-Suk,Kim,Myung-Sik,Han,Soung-Jin,Chung,Jong-Il,Kang,Jin-Ho 한국자원식물학회 2002 Plant Resources Vol.5 No.3
The soybean cyst nematode (Heterodera glycines Inchinoe; SCN) is a devastating pest of soybean and is responsible for significant losses in yield. The use of resistant cultivars is the effective method to reduce or eliminate SCN damage. The objective of this research is to identify AFLP markers linked to the SCN resistant genes. Bulked genomic DNA was made from resistant and susceptible genotypes to SCN and a total of 19 primer combinations were used. About 31 fragments were detected per primer combination. The banding patterns were readily distinguished in resistant and susceptible bulked genotypes. Polymorphic fragments were detected between resistant and susceptible bulked genotypes in the primer combination of CGT/GGC, CAG/GTG and CTC/GAG. In primer combinations of CGT/GGC and CAG/GTG, bulked resistant genotype produced a polymorphic bands. However, in primer of CTC/GAG, bulked susceptible genotype produced a polymorphic fragments. Three AFLP markers identified as a polymorphic fragments between bulked genomic DNA were mapped in 85 F2 population. Among them, only two markers, CGT/GGC and CTC/GAG, was linked and was mapped. Broad application of AFLP marker would be possible for improving resistant cultivars to SCN.
김충호,고신웅 경성대학교 공학기술연구소 2005 공학기술연구지 Vol.12 No.-
This investigation performed monotonic incremental loading test and repeating loading test of the beams repaired with Carbon Fiber Sheet(CFS) after occurring the crack corresponding to 0.75P_(y) representing the damages. Experiments confirmed that growth of the special pre-crack under the loading point was a direct cause of a delamination of the CFS and it led a failure of the beams. From the test results, it is concluded that the failure of beams repaired by CFS will occur at pre-crack under the loading point.
Adriamycin 및 Epiadriamycin 투여가 Mouse 심장근의 Na+, K+ - ATPase 활성에 미치는 영향
곽진구,이군자,정호삼,이규식 대한해부학회 1988 Anatomy & Cell Biology Vol.21 No.1
Adriamycin and epiadriamycin, which are anthracyclin antibiotics isolated from Streptomyces peucetius caesius, inhibit DNA and RNA synthesis, and have been widely used as anticancer drugs. Adriamycin and epiadriamycin have so little specificity toward the normal and tumor cells that they cause cellular damage by inhibiting protein synthesis and by peroxidation of membrane unsaturated fatty acids. Therefore the authors undertook the present study to pursue the effect of adriamycin and epiadriamycin on the cardiac muscle cells. Albino mice, ICR strain, weighing 20 gm were used as experimental animals. The experimental animals were killed at 12, 24, 48 and 72 hours after administration of 30 mg/kg of adriamycin and 36 mg/kg of epiadriamycin, respectively. The specimens obtained from left ventricle of the heart were frozen in the cryostat and stained by Guty and Albers' method for Na^(+), K^(+)-ATPase activity. The result obtained were as follows: 1. The Na^(+), K^(+)-ATPase activity in the left ventricular cardiac muscle cells was weakly' positive in the 12-hour and 24-hour adriamycin treated groups and trace positive in the 72-hour adriamycin treated group. 2. The Na^(+), K^(+)-ATPase activity in the left ventricular cardiac muscle cells was moderately posoitive in the 12-hour and 48 hour epiadriamycin treated groups and weakly positive in the 72-hour epiadriamycin treated group. Consequently, it is suggested that adriamycin and epiadriamycin inhibit Na^(+), K^(+)-ATPase activity, the inhibition being due to cytotoxic effect of the drug and that the cardiac toxicity of epiadriamycin appears to be less than that of adriamycin.