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정장한 ( Jang Han Jung ),강민규 ( Min Kyu Kang ),이한나 ( Han Na Lee ),권미혜 ( Mi Hye Kwon ),정청일 ( Chung Il Joung ) 대한내과학회 2011 대한내과학회지 Vol.81 No.1
Behcet`s disease is a multisystem autoimmune disease with vasculitic features, and major vascular involvement occurs in 7.7-60% of patients. Venous lesions are more common than arterial lesions and arterial thrombotic events are relatively rare. Wereport a patient with Behcet`s disease who developed a splenic infarct associated with splenic thrombotic arteritis. A 44-year-oldman who had been diagnosed with Behcet`s disease 5 years earlier presented with left flank pain lasting for 5 days. Laboratory tests revealed an elevated erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP). Computed tomography (CT) and three-dimensional CT showed a wedge-shaped splenic infarct and thrombus in the splenic artery. We treated him with low-molecular-weight heparin and prednisolone. The symptoms improved within 6 days of hospitalization, after which we stopped the heparin and added methotrexate and azathioprine. Splenic infarct should be ruled out if patients with Behcet`s disease complain of new left-sided abdominal pain. (Korean J Med 2011;81:126-129
Parallel NPN BJT로 인한 높은 홀딩 전압을 갖는 SCR 기반 양방향 ESD 보호 소자에 관한 연구
정장한(Jang-Han Jung),우제욱(Je-Wook Woo),구용서(Yong-Seo Koo) 한국전기전자학회 2021 전기전자학회논문지 Vol.25 No.4
본 논문에서는 기존의 LTDDSCR의 구조를 개선하여 기생 NPN BJT의 낮은 전류이득으로 높은 홀딩전압을 갖는 새로운 ESD 보호 소자를 제안한다. 제안된 보호 소자는 Synopsys사의 TCAD simulation을 이용하여 HBM simulation으로 전기적 특성을 분석하였고 current flow와 impact ionization 및 recombination Simulation으로 추가된 BJT가 동작하는 것을 확인하였다. 또한, 설계변수 D1, D2로 홀딩전압 특성을 최적화하였다. Simulation 수행결과, 새로운 ESD 보호 소자는 기존의 LTDDSCR과 비교하여 높은 홀딩전압을 갖는 것이 검증되었고 대칭적인 양방향 특성을 갖는 것이 확인되었다. 따라서 제안된 ESD 보호 소자는 IC에 적용될시 높은 면적 효율성을 가지며 IC의 신뢰성을 향상시킬 것으로 기대된다. In this paper, we propose a new ESD protection device with high holding voltage with low current gain of parasitic NPN BJT by improving the structure of the existing LTDDSCR. The electrical characteristics of the proposed protection device were analyzed by HBM simulation using Synopsys’ TCAD simulation, and the operation of the added BJT was confirmed by current flow, impact ionization and recombination simulation. In addition, the holding voltage characteristics were optimized with the design variables D1 and D2. As a result of the simulation, it was verified that the new ESD protection device has a higher holding voltage compared to the existing LTDDSCR and has a symmetrical bidirectional characteristic. Therefore, the proposed ESD protection device has high area efficiency when applied to an IC and is expected to improve the reliability of the IC.
높은 홀딩 전압으로 인한 래치업 면역을 갖는 양방향 구조의 ESD 보호회로에 관한 연구
정장한(Jang-Han Jung),도경일(Kyung-Il Do),진승후(Seung-Hoo Jin),고경진(Kyung-Jin Go),구용서(Yong-Seo Koo) 한국전기전자학회 2021 전기전자학회논문지 Vol.25 No.2
본 논문에서는 일반적인 SCR의 구조를 개선하여 높은 홀딩 전압으로 인한 래치 업면역 특성을 가지는 새로운 ESD 보호회로를 제안한다. 제안된 ESD회로의 특성검증을 위하여 Synopsys사의 TCAD를 이용하여 시뮬레이션을 진행하였으며, 기존 ESD 보호회로와 비교하여 제시하였다. 또한 설계변수 D1을 이용하여 전기적 특성의 변화를 검증하였다. 시뮬레이션 수행 결과 제안된 ESD 보호회로는 기존의 ESD 보호회로에 비해 높은 홀딩 전압특성과 양방향 방전특성을 확인하였다. 또한, Samsung의 0.13㎛ BCD 공정을 이용하여 설계 후 TLP 측정을 통해 전기적 특성을 검증하였다. 이러한 과정을 통해 본 논문에서 제안된 ESD 보호회로 설계변수의 최적화를 진행하였고 향상된 홀딩 전압으로 래치 업 면역을 갖는다는 점에서 고전압 어플리케이션에 적용하기에 매우 적합함을 검증하였다. In this paper, we propose a novel ESD protection device with Latch-up immunity properties due to high holding voltages by improving the structure of a typical SCR. To verify the characteristics of the proposed ESD circuit, simulations were conducted using Synopsys TCAD and presented compared to existing ESD protection circuits. Furthermore, the variation of electrical properties was verified using the design variable D1. Simulation results confirm that the proposed ESD protective circuit has higher holding voltage properties and bidirectional discharge properties compared to conventional ESD protective circuits. We validate the electrical properties with post-design TLP measurements using Samsung’s 0.13㎛ BCD process. And we verify that the proposed ESD protection circuit in this paper is well suited for high voltage applications in that it has a latch-up immunity due to improved holding voltage through optimization of design variables.
김진호(Kim Jin-Ho),한석윤(Han Suk-Yoon),정장용(Jung Jang-Yong),문제우(Mun Je-Woo) 한국철도학회 2008 한국철도학회 학술발표대회논문집 Vol.- No.-
The construction of concrete slab track system is increased recently but the noise level on slab track is 3dB(A) higher than the one on ballast track since most of noise is reflected on slab track. Currently, concrete slab track systems has been designed for Gyungbu high speedline stage 2 and Honam high speed line. For those tracks, noise absorbing blocks are considered. In this paper, noise absorbing blocks were installed at Hwang Hak tunnel for evaluation of noise absorbing blocks and the indoor noise of KTX was measured. The results have done comparative analysis.
Hep3B 세포내 PPARγ/PGC-1α/HNF-4α 연쇄작용을 통한 Atorvastin의 FXR과 CYP1A1 활성화 유도
이진 ( Jin Lee ),홍은미 ( Eun Mi Hong ),정장한 ( Jang Han Jung ),박세우 ( Se Woo Park ),이상표 ( Sang Pyo Lee ),고동희 ( Dong Hee Koh ),장현주 ( Hyun Joo Jang ),계세협 ( Sea Hyub Kae ) 대한소화기학회 2021 대한소화기학회지 Vol.77 No.3
Background/Aims: PPARγ, farnesoid X receptor (FXR) and CYP7A1 are associated with solubility of bile. This study was performed to understand a mechanism and interactions of statin-induced PPARγ, PGC-1α and HNF-4α related to the statin-induced activation of FXR and CYP7A1, and verify whether the mevalonate pathway is involved in the mechanism. Methods: MTT assays were performed using cultured human Hep3B cells to determine the effect of atorvastatin on the cell proliferation. Expression levels of indicated proteins were measured using Western blotting assays by inhibiting the protein expression or not. Results: Atorvastatin increased expression of PPARγ, PGC-1α, HNF-4α, FXR, and CYP7A1 in Hep3B cells. PPARγ ligand of troglitazone upregulated the expression of PGC-1α, HNF-4α, FXR, and CYP7A1 in Hep3B cells. Silencing of PPARγ, PGC1α, and HNF4α using respective siRNA demonstrated that atorvastatin-induced FXR and CYP7A1 activation required sequential action of PPARγ/PGC-1α/HNF-4α. The silencing of PPARγ completely inhibited atorvastatin-induced PGC-1α expression, and the PGC1α silencing partially inhibited atorvastatin-induced PPARγ expression. The inhibition of HNF4α did not affect atorvastatin-induced PPARγ expression, but partially inhibited atorvastatin-induced PGC-1α expression. Besides, mevalonate completely reversed the effect of atorvastatin on PPARγ, PGC-1α, HNF-4α, FXR, and CYP7A1. Conclusions: Atorvastatin induces FXR and CYP7A1 activation as a result of sequential action of PPARγ/PGC-1α/HNF-4α in human hepatocytes. We propose that atorvastatin enhances solubility of cholesterol in bile by simultaneously activating of FXR and CYP7A1. (Korean J Gastroenterol 2021;77:123-131)