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중국 내 순차적 직접투자와 경영 전략적 특성에 관한 연구
임형록 ( Hyung Rok Yim ),정원진 ( Won Jin Chung ) 국제지역학회 2014 국제지역연구 Vol.18 No.3
본 연구는 순차적 직접투자가 단일 직접투자에 비해 창출하는 경쟁우위를 이론적으로 규명한후 이를 실증적으로 검증하고자 했다. 이는 대(對)중국 직접투자를 시행하는 우리나라 기업들이 주로 일회성 투자에 그치지 않고 후속투자를 거쳐 중국 시장 내 생산거점을 확보하려 한다는 전략적 행동에 기반 한 것이다. 이를 이론적으로 접근하기 위해 수량경쟁모형을 구축한 후명제들을 추출해 냈다. 미래가치를 포함 해 도출된 균형점들은 첫째, 순차적 직접투자는 모기업들의 생산을 증대시키는 효과를 발생시킬 것이고, 둘째, 일회성 투자에 그치는 경우에 비해순차적 투자는 큰 미래가치를 창출할 수 있으며, 셋째, 대(對)중국 순차적 직접투자는 장기적으로 진행될수록 기업단위에서 보다 큰 생산효과를 거둘 수 있다는 점을 제시한다. 이는 중국시장에서의 노하우가 축적될수록 경쟁우위가 강화될 것임을 의미한다. 이러한 이론적 명제들을 검증하고자 대(對)중국 직접투자를 시행한 우리나라 모기업들을 대상으로 회귀분석을 시도했는데, 그 결과 수량경쟁 모형으로부터 도출된 명제들이 지지되었다. 주요 결과를 정리하면 첫째, 순차적 투자는 모기업의 생산력을 증대시키는 것으로 나타났고, 둘째, 중국 내 활동기간에 비례해 중국 내 총 자회사 수가 증가함을 알 수 있다. 셋째, 중국 투자 이전 해외진출경험은 대(對)중국 순차적 직접투자를 시행한 모기업의 경영성과를 유의적으로 개선시키고,대(對)중국 순차적 직접투자가 증가할수록 모기업의 생산성이 동반 상승한다. A noticeable aspect of Korean firms’ outward sequential FDIs to China is that they occur sequentially, which means that they implement the outward FDIs to China with along-term perspective. To analyze the strategic advantages of sequential investment, we introduce Cournot type quantity competition model. According to the model, three important implications are derived. First, sequential FDIs enhances the Korean parents’production capabilities. Second, the parents are more likely to establish new Chinese subsidiaries as they stay longer in China. Third, the production effect of sequential investments incurs more sequential investments. Some regression models are tested for verifying the predictions. According to empirical results, three important results are found. First, initial entry mode affects the size expansion of the Korean parents. Second, the longer the duration of intial subsidiary in China, the more the sequential investment will be. Third, sequential investments are positively associated with the productivity of the Korean parents.
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Prostaglandin 이 백서의 임의형 피부판에 미치는 영향에 대한 실험적 연구
유재덕,이상헌,이영호,신극선,정원진 大韓成形外科學會 1984 Archives of Plastic Surgery Vol.11 No.4
As to survive, the flap must maintain the blood supply to its own. In recent years, many attempts have been made clinically and experimentally to improve the blood supply in ischemic random pattern flaps. It is reasonable to speculate that the mechanical stimuli during the construction of skin flap may result in release of certain kinds of prostaglandins which cause vasoconstruction, thrombus formation and even vasodilatation. It is probably one of the major detrimental factors responsible for the failure of the constructed skin flap. This study was designed to clarify the certain facts of prostaglandin synthesis following the flap construction and reconfirm the actions of various prostaglandins in regard with the flap survival. A total of 40 male white rats was grouped into 4; the control, group Ⅰ treated with flurbiprofen as prostaglandin inhibitor, group Ⅱ treated with flurbiprofen and prostaglandin E₂ as vasodilator, and group Ⅲ treated with flurbiprofen and prostaglandin F₂α as vasodilator. Each animal had a flap on the back measuring 2×8cm and was observed for the flap survival daily for one week and the following results were summerized; 1. Group Ⅰtreated with flurbiprofen which inhibit prostaglandin and thromboxane synthesis showed significant increase of the area of flap survival compared with the control. 2. The flap survival was best improved in the group Ⅱtreated with flurbiprofen and vasodilating prostaglandin(PGE₂) and worse in the group Ⅲ treated with vasoconstricting prostaglandin(PGF₂α). 3. It is suggested that prostaglandins act on distal part of the flap because no vascular changes were noted at proximal part of the flaps on histological study.
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백서피부 Fibroblast 배양에서 Triamcinolone Acetonide 가 Collagen대사에 미치는 영향
정원진,신극선,이영호,유재덕,김윤수 大韓成形外科學會 1990 Archives of Plastic Surgery Vol.17 No.4
The process of wound healing of skin tissue is closely regulation of collagen metabolism and is a complicate biological phenomenon. Therefore. the study to clarify the basic mechanism of it is an active field. in addition. there are many active investigations of the mechanism of keloid or hypertrophic scar formation after wound healing. It has been reported that histopathological changes such as excess accumulation of collagen, a connective tissue component and biochemical changes such as increased activities of prolyl hydroxylase and galactosylhydroxylysyl glucosyltransferase were observed in keloid tissue. Now, clincal use of glucocorticoids on the local dermal tissues to decrease collagen synthesis gives a good response, but reports of the effect of synthetic glucocorticoids on the growth and collagen metabolism of skin fibroblasts are controversial. In the present study. cultured fibroblasts of rat skin were treated with triamcinolone acetonide in various concentrations and the amousnts of collagen and collagen specific mPNA in fibroblasts were measured. fibroblated in media containing low concentrations of rtiamcinolone acetonide(0.5.1,2,4uM) or high concentration of triamcinolone acetonide (50,100,150uM) for 10 days and were used for the experiment. The results are as follows. 1. The amount of collagen was measured indirectly by the measurement of hvdroxproline which is present specifically in collagen. The amounts of hydrexyproline in the culture flasks treated with 0.5,1,2, and 4 uM triamcinolone acetonide were 179.1,200.5,229.7 and 171.9% of the control respectively, and the amounts of hydroxyprolime per ug DNA were 147.6 166.7, 224.0 and 133.1% of the control, indicating the increased collagen content by the treatment of low concentration of triamcinolone acetonide. DNA contents of fibroblasts treated with 0.5,1,2, and 4uM triamcinolone acetonide were 121.4, 120.3, 102.6 and 129.2% of the control. 2. However, the amounts of hydroxyproline in culture flasks treated with high concentrations of triamcinolone acetonide. 50.100 and 150uM were 45.1, 43.8 and 39.3% ofthe control, respectively. The amounts of hydroxyproline per ug DNA were also 42.4, 37.7 and 34.0% of the conrtol, respectively, indicating a decreased collagen content by High concentrations of triamcinolone acetonide treatments. The DNA contents in each treatment group were 107.8,117.6 and 115.2% of the control, indicating that even 150uM of triamcinolone acetonide did not inhibit the cell growth but increased it slightly. 3. Treatment with I uM of triamcinolone acetonide caused a slight decrease in the content of mRNA for the αI chain of type I collagen, but no great changes were observed . However, treatments with 50uM and 100uM of triamcinolone acetonide resulted in a decrease of mRNA for the α Ichain of type I collagen to a quarter of the control. This suggests that the increase of collagen content in rat skin fibroblasts treated with low concentrations of triamcinolone acetonide might concentrations of triamcinolone acetonide might result from selective control in transcription step, as evidenced by the decreased amount of mRNA for αI(1) collagen in conjugation with the decreased collagen content.
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