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위암 및 Helicobacter pylori 감염의 위험인자로서 염분 섭취의 역할
경태영(Tae Young Kyong),김학양(Hak Yang Kim),채경수(Gyeoung Soo Chae),유희승(Hee Seung Yoo),박수종(Soo Jong Park),김종혁(Jong Hyeok Kim),장웅기(Woong Ki Chang),김용범(Yong Bum Kim),박충기(Choong Kee Park),남은숙(Eun Sook Nam),유재영( 대한내과학회 1998 대한내과학회지 Vol.55 No.2
Objectives: Gastric cancer is one of the most important malignancy of the gastrointestinal tract. High salt intake has been suggested as a risk factor of gastric cancer and promoting Helicobocter pylori infection. Few studies have been addressed about the relation between excessive salt intake and gastric cancer. This study was performed to investigate the relation of high salt intake to H. pylori infection and gastric cancer. Methods . Between May 1996 and July 19%, hospitalized patiaits with epigastric pain or dyspepsia were prospectively recruited. Patients with underlying diseases which could reduce kidney function were excluded. Salt consumption was estimated by the urine sodium to creatinine ratio(U-Na/Cr) on fresh single urine sample. Gastric cancer was diagnosed by endoscopic biopsies. H. pylori infection was evaluated by CLOtest. Gastric cancer was classified histologically by Lauren classification. Results: Ninety seven patients, 52patients with gastric cancer and 45patients with chronic gastritis were enrolled. There was no statistically significant difference in the sodium to creatinine ratio between gastric cancer group(173.60±123, range; 21-665.2) and chronic gastritis group(164.02±138, range; 20.4-482.7)(p=0.361). In gastric cancer, the sodium to creatinine ratio was not different between CLOtest positive and negative group(p=0.201), and among intestinal, diffuse and mixed type(p=0.419), either. Conclusion: This study does not support the causal relation of high salt diet on gastric carcinogenesis. There is no significant difference in H, pylori infection rate between CLOtest positive group and negative group.
Helicobacter pylori 감염의 박멸후 발생한 역류성 식도염
이준호,김학양,유재영,박충기,김종혁,이종민,김용범,서지영,김재삼,유희승 대한소화기내시경학회 1998 Clinical Endoscopy Vol.18 No.5
Helicobacter pylori infection causes chronic gastritis and its well documented con- sequences are peptic ulcer disease and gastric neoplasia. As duodenal ulcer is often associated with gastroesophageal reflux disease and antral gastritis is a frequent finding in patients with reflux disease, H. pylori infection may be a common cause of both conditions. Recent studies reveal that H. pylori has no role in the pathogenesis of reflux esophagitis. Furthermore, there are some arguments on whether H. pylori infection may have a protective role in reflux esophagitis. We have experienced two cases of reflux esophagitis after cure of H. pylori infection. H. pylori eradication therapy was performed in two patients who have gastric and/or duodenal ulcer with omeprazole and two anti-biotics (clarithromycin and amoxicillin). After cure of H. pylori infection, reflux esophagitis was demonstrated in these patients by endoscopy.
Helicobacter pylori 감염을 동반한 T 림프구 기원의 원발성 위 림프종 1 예
남은숙,김학양,유재영,박충기,김종혁,김용범,박수종,유희승,김성균,허필석 대한소화기내시경학회 1999 Clinical Endoscopy Vol.19 No.2
The primary T-cell lymphoma of the stomach is a extremely rare disease, only about 33 cases were described in the literature since 1986. Recent studies have proved a strong relation between Helicobacter pylori infection and several gastric diseases such as peptic ulcer, chronic gastritis, gastric cancer, and lymphoma. We report a case of primary gastric T-cell lymphoma with H. pylori infection in a 63-year-old male who had a history of epigastric fullness for 2 years. Gastroscopy showed a round ulcer of 1cm in size on anterior side of gastric angle. The gastroscopic biopsy revealed diffuse large cell malignant non-Hodgkin' s lymphoma on light microscopic examination, and T-cell immunophenotype with immunohistochemical study. The result of CLOtest was positive. No lymphadeno-pathy was found with chest roentgenogram and computed omography of abdomen and pelvis. Bone marrow examination showed normal finding.