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비알코올성 지방간의 중증도에 따른 혈중 Ghrelin 농도 변화
유태우 ( Tae Woo Yoo ),조용균 ( Yong Kyun Cho ),강문수 ( Mun Su Kang ),오세용 ( Se Yong Oh ),박정호 ( Jung Ho Park ),김홍주 ( Hong Joo Kim ),박동일 ( Dong Il Park ),손정일 ( Chong Il Sohn ),전우규 ( Woo Kyu Jeon ),김병익 ( Byung I 대한내과학회 2006 대한내과학회지 Vol.71 No.1
Background: Recently, ghrelin has been reported to be associated with insulin resistance. Nonalcoholic fatty liver disease (NAFLD) is a condition in which insulin resistance relatively plays a pivotal role. The aim of this study was to evaluate the change of serum ghrelin concentration according to severity of hepatosteatosis. Methods: Sixty five apparently normal male adults who underwent health screen examinations were classified into three groups, Group I: normal (27 subjects), Group II: mild (24 subjects) and Group III: moderate to severe fatty liver (14 subjects), according to ultrasonographic findings of liver. We analyzed the association between serum ghrelin concentration and severity of hepatosteatosis by ANOVA test. And the independent correlation between serum ghrelin concentration and insulin resistance related factors, HOMA (homeostatic model assessment), BMI (body mass index), WC (waist circumference), HC (hip circumference), WHR (waist to hip circumference ratio) were analyzed by multiple linear regression analysis. Results: Serum ghrelin concentration tended to decrease according to severity of hepatosteatosis (Group I: 230.9±94.3, Group II: 195.2±97.2, Group III: 164.3±71.4 pmol/L). But this was statistically insignificant (p=0.081). The independent correlation between serum ghrelin concentration and insulin resistance related factors were not observed. Conclusions: Our study did not prove the correlation between insulin resistance related factors and serum ghrelin concentration in NAFLD according to severity of hepatosteatosis. However, we found a tendency to decrease serum ghrelin concentration according to severity of hepatosteatosis. So, further studies are required for certification these correlations.(Korean J Med 71:45-51, 2006)
유태우(Tae Woo Yoo),송윤미(Yun Mi Song) 한국의학교육학회 1991 Korean journal of medical education Vol.3 No.2
In an effort to evaluate the effectiveness of teaching behaviors and to compare the qualitative and quantitative methods of evaluating clinical preceptors, we performed a study with questionnaires to survey students and preceptors who participated in preceptorship in our department of Family Medicine over 6 months from May, 1991 to September, 1991. Students reported humanistic orientation behaviors to be most contributing to their clinical medical education. However, preceptors favored student centered instructional strategies. An analysis and comparison of the qualitative and quantitative evaluation of preceptors suggest that qualitative approach identifies individual preceptor strengths and weaknesses more clearly than quantitative approach and may be more useful for providing feedback to stimulate and guide change.
독성 간손상 관련 한국인의 약물복용 실태와 건강비용 조사 : 독성 간손상의 진단 및 보고체계 구축을 위한 다기관 공동연구
유태우 ( Tae Woo Yoo ),김병익 ( Byung Ik Kim ),김진봉 ( Jin Bong Kim ),김동준 ( Dong Joon Kim ),김재우 ( Jae Woo Kim ),백순구 ( Soon Koo Baik ),김광석 ( Kwang Seok Kim ),천갑진 ( Gab Jin Cheon ) 대한간학회 2007 Clinical and Molecular Hepatology(대한간학회지) Vol.13 No.1
한국인 성인에서 혈당 및 혈압 정도에 따른 인슐린저항성 및 혈중 hsCRP 농도의 비교
김영춘 ( Young Choon Kim ),이은정 ( Eun Jung Rhee ),유태우 ( Tae Woo Yoo ),황상태 ( Sang Tai Hwang ),오세용 ( Se Yong Oh ),전한별 ( Han Byul Chun ),강정묵 ( Jeung Mook Kang ),류승호 ( Seung Ho Ryu ),이원영 ( Won Young Lee ),김선우 대한내과학회 2005 대한내과학회지 Vol.68 No.2
Background : Insulin resistance is a strong contributor to cardiovascular diseases. The increasing prevalence of diabetes and the subsequent complications confers a great importance to the early detection and intervention of diabetes. However, the exact r
Mongolian gerbil에서 Helicobacter pylori 감염 후 발생되는 Microsatellite Instability에 관한 연구
박동일 ( Dong Il Park ),김민경 ( Min Kyung Kim ),김재준 ( Jae J. Kim ),유태우 ( Tae Woo Yoo ),박정호 ( Jung Ho Park ),김홍주 ( Hong Joo Kim ),조용균 ( Yong Kyun Cho ),손정일 ( Chung Il Sohn ),전우규 ( Woo Kyu Jeon ),김병익 ( Byung 대한내과학회 2006 대한내과학회지 Vol.71 No.1
Background: During Helicobacter pylori (HP) gastritis, the organisms cause an impairment of DNA repair that results in accumulation of mutations in the genome of epithelial cells and an increased risk of gastric carcinoma. The aim of this study was to determine whether microsatellite instability (MSI) could be induced by chronic HP infection in the Mongolian gerbil model. Methods: Seven-weeks old, specific pathogen-free male Mongolian gerbils were infected with the HP strain CA20. They were sacrificed at the each time points (1, 2, 3, 6, 9, 12 months). Areas of gastritis were carefully microdissected. DNA from tissues was analyzed for the presence of MSI by genomic DNA amplification with five gerbil microsatellite markers per sample. Gerbil markers labelled with Fam or Hex were obtained to perform MSI analysis using the 310 Genetic Analyzer. Results: MSI was more frequent in the area of gastritis than in that of normal (p<0.05). Six showed MSI-low and one showed MSI-high within the area of gastritis (p<0.05). Conclusions: This study shows that chronic HP infection in the Mongolian gerbil induces MSI. This finding indicates that HP infection causes a decrease in DNA MMR proteins in epithelial cells of the stomach that may reach critically low levels, allowing for the accumulation of mutations such as those seen in microsatellite regions.(Korean J Med 71:17-23, 2006)
궤양성 대장염 환자에서 5 - Aminosalicylic Acid 로 유발된 급성 췌장염
이현영(Hyun Young Lhee),김병익(Byung Ik Kim),유태우(Tae Woo Yoo),이호철(Ho Chul Lee),정찬희(Chan Hee Jung),신헌섭(Hun Sub Shin),박승하(Seung Ha Park),김정욱(Jung Uk Kim),조용균(Yong Kyun Cho),이준행(Jun Haeng Lee),박창영(Chang Young 대한소화기학회 2002 대한소화기학회지 Vol.40 No.4
Acute pancreatitis is a rare complication of 5-aminosalicylic acid (5-ASA) treatment. We recently experienced a case of acute pancreatitis secondary to 5-ASA in a patient with ulcerative colitis (UC). A 25-year-old man was admitted for bloody diarrhea and lower abdominal pain. Mesalazine was used under the diagnosis of ulcerative colitis, but the symptoms were not improved. After symptomatic improvement by steroid treatment, mesalazine was restarted for maintaining the remission. On the next day, he had a sudden severe epigastric pain with elevation of serum amylase and lipase levels. Drug- induced pancreatitis was suspected, and the mesalazine was discontinued. His abdominal pain disappeared, and his serum amylase and lipase levels returned to normal. After the administration of sulfasalazine, however, pancreatitis was relapsed. In conclusion, we suggest that drug-induced pancreatitis should be suspected in any patient who develops new abdominal pain while receiving 5-ASA. (Korean J Gastroenterol 2002; 40: 282-285)
한국 남성 비알코올성 지방간에서 혈청 adipokine 농도 변화에 영향을 미치는 요인
오세용 ( Se Yong Oh ),조용균 ( Yong Kyun Cho ),유태우 ( Tae Woo Yoo ),박정호 ( Jung Ho Park ),김홍주 ( Hong Joo Kim ),박동일 ( Dong Il Park ),손정일 ( Chong Il Sohn ),전우규 ( Woo Kyu Jeon ),김병익 ( Byung Ik Kim ),정찬희 ( Chan H 대한내과학회 2006 대한내과학회지 Vol.71 No.1
Background: Adipokines are associated with various metabolic disorders including insulin resistance, obesity and dyslipidemia. Metabolic disorders have also been reported to be associated with nonalcoholic fatty liver disease (NAFLD). We aimed to estimate changes in serum adipokines levels according to the degrees of steatosis and to determine independent factors influencing serum adipokines levels in Korean male patients with NAFLD. Methods: 65 Korean male patients were subjected. The degrees of steatosis were stratified into the three groups, Group I: normal liver (27 subjects), Group II: mild fatty liver (24 subjects) and Group III: moderate to severe fatty liver (14 subjects), according to ultrasonographic liver findings. The anthropometric parameters, fasting serum adipokine levels including leptin, adiponectin and resistin were measured in all subjects. The level of insulin resistance was estimated using the HOMA-IR. Results: Serum leptin levels were significantly different among the three groups (mean±SD: Group I (2.052±1.071), Group II (2.879±1.016), Group III (4.457±1.965 ng/mL), p<0.001). Serum adiponectin and resistin levels were not significantly different among the three groups (p=0.184, p=0.649, respectively). BMI and HOMA-IR were independent factors of changes in serum leptin levels (p=0.026, p=0.001, respectively), but independent factors of changes in serum adiponectin and resistin levels were not observed. Conclusions: Our study support a indirect role to induce metabolic disorder for leptin in the pathogenesis of NAFLD, but do not support roles for adiponectin and resistin in the pathogenesis of NAFLD. BMI and HOMA-IR were only independent factors of changes in serum leptin levels.(Korean J Med 71:58-66, 2006)