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Oxidation of tetralin to 1-tetralone over CrAPO-5
사미란,이유리,안화승 한국화학공학회 2017 Korean Journal of Chemical Engineering Vol.34 No.3
A chromium-incorporated microporous aluminophosphate, CrAPO-5, was tested as a catalyst for the oxidation of tetralin using either a combination of aldehyde and O2 (1 atm) or tert-butyl hydroperoxide as an oxidant system. The former resulted in significantly improved catalytic performance (69.6% yield to 1-tetralone) over the latter (38.5% yield to 1-tetralone) at 80 oC in 8 h, and conversion increased in the sequence of acetaldehyde<<isobutyl aldehyde<pivalaldehyde. CrAPO-5 also exhibited good activity and high selectivity (53.7% yield to 1-tetralone) after 24 h using pivalaldehyde and O2 at room temperature. Hot filtration and recycle experiments established that oxidation takes place mostly on Cr sites in CrAPO-5. A plausible reaction mechanism involving acylperoxy radicals and oxo-chromium(V) species on CrAPO-5 was proposed for the tetralin oxidation.
A 24 ㎓ High Gain, High Efficient Stagger-Tuned CMOS Power Amplifier
Tahesin Samira Delwar(델웨 타헤신 사미라),Abrar Siddique(아브라르 시디크),Manas Ranjan Biswal(비스왈 마나스 란잔),Prangyadarsini Behera(프랑기다르시니 베헤라),Yeji Choi(최예지),Habibulloyev F. A. Ugli(하비불로예브 파흐리딘 압두하림 우그리),Bo-Yeong Park 대한전자공학회 2022 대한전자공학회 학술대회 Vol.2022 No.6
Morin Protects Normal Human Dermal Fibroblasts from Ultraviolet B-induced Apoptosis
박정언,진오현,박미경,강경아,페르난도 핀카 디바게 사미라 마두샨,헤라스 무디야세라게 우다리 라크미니 헤라스,현진원 한국생명과학회 2023 생명과학회지 Vol.33 No.4
Ultraviolet B (UVB) irradiation causes skin diseases by inducing cellular oxidative stress, photoaging, and inflammation. This study aimed to investigate the protective effects of morin against UVB-induced oxidative stress in normal human dermal fibroblasts (NHDFs). Morin has been reported to be a potential therapeutic candidate for oxidative stress-mediated diseases, neurodegenerative diseases, and inflammation. Since morin has been identified as a potential antioxidant, we speculated that morin could alleviate UVB-induced apoptosis in NHDFs. Cell viability and intracellular reactive oxygen species (ROS) levels were measured using the MTT assay, H2DCFDA, and the DHE staining method, respectively. Lipid peroxidation and protein carbonyl formation were tested using ELISA kits. DNA fragmentation and comet assay were used to assess DNA damage. Apoptotic bodies were analyzed using Hoechst 33342 staining and TUNEL assay. The expression of apoptosis-related proteins was examined using Western blot analysis. Morin showed a cyto-protective effect by scavenging UVB-induced ROS, increasing the expression of antioxidant-related proteins and inhibiting UVB-induced oxidative alterations such as lipid peroxidation, protein carbonylation, and DNA damage. Morin protects against UVB-induced cell apoptosis by inhibiting Bcl-2-associated X protein, caspase-9, and caspase-3 expression, while increasing the expression of the anti-apoptotic protein Bcl-2. These effects of morin were conferred through decreased phosphorylation of p38 and c-Jun N-terminal kinase 1/2. The results demonstrated that morin may be developed as a preventive/therapeutic drug to be used to prevent UVB-induced skin damage.