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방제(方劑)에서 행인(杏仁)의 역할에 관한 문헌적 고찰
변성희,김한균,지선영,이병욱,김상찬,Byun Sung-Hui,Kim Han-Kyun,Jee Seon-Young,Lee Byung-Wook,Kim Sang-Chan 대한한의학방제학회 2003 大韓韓醫學方劑學會誌 Vol.11 No.2
Armeniacae Amarum Semen was well-known as a theraputic agent of asthma by flowing downward of qi and moistening of lung in Oriental Medicine. But there are many other effect in Armeniacae Amarum Semen such as, loosening the bowel to relieve constipation, removing stagnation of qi and food, dispersing accumulation of pathogen, parasiticide. We have been studying what the meaning of Armeniacae Amarum Semen in herbal formula and usage of Armeniacae Amarum Semen is.
왕앙(汪昻)의 약물이론(藥物理論)에 관한 연구(硏究) (-수치이론(修治理論)에 관(關)하여-)
변성희,서부일,Byun Sung-Hui,Seo Bu-il 대한한의학방제학회 1999 大韓韓醫學方劑學會誌 Vol.7 No.1
We studied medicinal theory on processing drugs of Wang-Ang. Wang-Ang used various methods of medicinal processing. He used such as soaking(浸), maceration(泡), washing(洗), elutriation(淘), mixing together(拌), refining powder with water(水飛) and floating(水漂) in medicinal processing with water. He used such as making hot(?), roasting in ashes(?), stir-frying with liquid(炙), parching(炒), heating(燒), baking(焙), stirring-baking(?) and distilling(煉) in medicinal processing with fire. He used such as steaming(蒸), boiling(煮), making hot and soaking(??) in medicinal processing with water and fire. And He used such as cooking(熟), making drugs into gelatin(制膠), making drugs into frostlike powder(制霜法), making drugs into bead(制珠), fermentation(生衣) and compound medicinal processing(複制法).
사묘용안탕(四妙勇安湯) 물추출물이 DNCB로 유발된 접촉성피부염에 미치는 영향
변성희,이병욱,김상찬,Byun, Sung-Hui,Lee, Byung-Wook,Kim, Sang-Chan 대한한의학방제학회 2005 大韓韓醫學方劑學會誌 Vol.13 No.2
Samyoyongantang (SMY), a herbal medicine, has been used as cure for gangrene. To evaluate anti-inflammatory effect of samyoyongantang, we treated samyoyongantang extract in animal model system induced contact hypersensitivity. Contact hypersensitivity, a local inflammatory response of the skin, was induced by 1% DNCB on the right ear of BALB/c mouse. Samyoyongantang was prepared as water extract and administrated everyday for 2 weeks per oral. A right ear of mouse was potently swelled by 1% of DNCB treatm ent, but a mouse ear thickness was significantly reduced by samyoyongantang after 2 we eks treatment. Samyoyongantang reduced IgG in serum obtained from blood of 1 % DNCB-treated mouse. IgE in serum was not changed by samyoyongantang treatment. From these results, anti-inflammatory effect of samyoyongantang, especially reduction of ear swell ing, might be partly due to reduction of IgG in serum.
변성희 ( Sung Hui Byun ),박상미 ( Sang Mi Park ),김상찬 ( Sang Chan Kim ),조일제 ( Il Je Cho ) 대한본초학회 2013 大韓本草學會誌 Vol.28 No.4
Objectives : Mori Folium was popularly used as one of the traditional medicinal herbs. Although M. Folium has been cultivated for rearing silkworm historically, it`s use has been expanded as natural therapeutic agent for the treatment of filariasis, diabetes and dropsy in East Asia. However, little has been known about the effect of M. Folium on liver fibrosis. Therefore, we would like to explore an anti-fibrogenic potential of M. Folium extract (MFE) using immortalized human hepatic stellate cell line, LX-2 cells. Methods : We examined the effects of MFE on the transforming growth factor β1 (TGFβ1)-induced liver fibrosis in LX-2 cells. Cell viability, Smad binding element-driven luciferase activity, phosphorylations level of Smad 2/3, and expression level of TGFβ1-dependent target genes were monitored in the MFE-treated LX-2 cells. Results : Up to 30 μg/ml MFE treatment did not show any possible toxic effect in LX-2 cells. MFE inhibited TGFβ1-inducible Smad binding element-driven luciferase activity and decreased the TGFβ1-inducible phosphorylations of Smad 2 and Smad 3 in hepatic stellate cell in a dose dependent manner. Furthermore, increases of plasminogen activator inhibitor type 1, TGFβ1 and matrix metalloproteinases 2 genes by TGFβ1 were also attenuated by MFE treatment. Conclusions : These findings suggested that MFE would be used as a potential therapeutic agent for the treatment liver fibrosis, which might be mediated by the inhibition of TGFβ1-inducible Smad 2/3 transactivation and target genes expression.