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남궁경영,강명진,임홍목,김미성,고병성,안현택,이종률,김종옥 대한간학회 2004 Clinical and Molecular Hepatology(대한간학회지) Vol.10 No.2
Metastasis of hepatocellular carcinoma occurs at a relatively late stage of the disease. Hematogenous and lymphatic metastases are the most common routes for dissemination of tumor cells. Hepatocellular carcinoma also extends into the adjacent portal vein and bile ducts. Since there is no peritoneum between the body of the gallbladder and the liver fossa, gallbladder cancer can easily cross the boundary. Gallbladder invasion of hepatocellular carcinoma, however, is quite rare. We report a case of hepatocellular-cholangiocarcinoma in a non-cirrhotic liver that invaded the gallbladder mimicking the gallbladder carcinoma complicated by cholecystitis and liver abscess. (Korean J Hepatol 2004;10:148-153)
남궁경영,강명진,임홍목,김미성,고병성,안현택,이종률,김종옥 대한간학회 2004 Clinical and Molecular Hepatology(대한간학회지) Vol.10 No.2
Metastasis of hepatocellular carcinoma occurs at a relatively late stage of the disease. Hematogenous and lymphatic metastases are the most common routes for dissemination of tumor cells. Hepatocellular carcinoma also extends into the adjacent portal vein and bile ducts. Since there is no peritoneum between the body of the gallbladder and the liver fossa, gallbladder cancer can easily cross the boundary. Gallbladder invasion of hepatocellular carcinoma, however, is quite rare. We report a case of hepatocellular- cholangiocarcinoma in a non-cirrhotic liver that invaded the gallbladder mimicking the gallbladder carcinoma complicated by cholecystitis and liver abscess.
NADPH Oxidase Mediates b-Amyloid Peptide-Induced Neuronal Death in Mouse Cortical Cultures
최기오,남궁경영,황신애,김종근,배춘상 전남대학교 의과학연구소 2017 전남의대학술지 Vol.53 No.3
b-Amyloid peptide (Ab) is the main component of senile plaques in patients with Alzheimer's disease, and is known to be a main pathogenic factor of the disease. Recent evidence indicates that activation of NADPH oxidase (NOX) in microglia or astrocytes may be a source of Ab-induced reactive oxygen species (ROS). We investigated the role of neuronal NOX in Ab-induced neuronal death in mouse mixed cortical cultures. Cell death was assessed by measuring lactate dehydrogenase efflux to bathing media 24 or 48 hr after exposure to Ab25-35, a fragment of Ab with an equivalent neurotoxic effect. Ab25-35 induced neuronal death in concentration- and time- dependent manners with apoptotic features. Neuronal death was significantly attenuated, not only by anti-apoptotic drugs, such as z-VAD-fmk and cycloheximide, but also by antioxidants, such as trolox, ascorbic acid, and epigallocatethin gallate. We also demonstrated that treatment with 20 mM Ab25-35 increased fluorescent signals in mixed cortical cultures, but produced only weak signals in pure astrocyte cultures in the presence of 2’,7’-dichlorofluorescin diacetate (DCF-DA), an indicator for intracellular ROS. Increased DCF-DA fluorescence was markedly inhibited, not only by trolox, but also by selective NOX inhibitors, such as apocynin and AEBSF. Western blot analyses revealed that Ab25-35 increased the expression of gp91phox, a main subunit of NOX in cells. The above antioxidants, apocynin, and AEBSF significantly attenuated neuronal death induced by Ab25-35. Furthermore, the gp91phox-specific siRNA-based knockdown of NOX significantly inhibited neuronal death. These results suggest that activation of neuronal NOX is involved in Ab25-35-induced neuronal death.
춘계학술대회 : 포스터전시 ; B형 간염 보유와 동반된 자가면역성 간염 1예
안현택,강명진,남궁경영 대한간학회 2005 Clinical and Molecular Hepatology(대한간학회지) Vol.11 No.3(S)
배경/목적: 자가 면역성 간염은 원인 미상의 지속적인 간세포염증과 섬유화를 보이는 질환으로 조직학적으로 경계면 간염, 고감마글로부린증, 자가항체 발현을 특징으로 하는 질환으로 만성 간염의 원인으로 감별해야 할 질환 중의 하나이다. 진단은 International AIH group에서 정한 기준에 따라 진단하게 된다. 이 질환은 유전적인 자가면역 경향이 있으며 환경적인 인자나 감염이 자가면역 과정을 유발한다고 추정되고 있으며 이에 관여된 바이러스로 C형