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횡좌굴을 (橫挫屈) 고려한 H 형강보의 회전 능력에 관한 기초적 연구
오경환(Kyung Hwan Oh),이명재(Myung Jae Lee),김덕재(Duk Jae Kim) 한국강구조학회 1991 韓國鋼構造學會誌 Vol.3 No.2
The study aims at investigating the rotational capacity of H-shaped steel beams analytically and experimentally, of which strength are dependent on the lateral buckling. Secant modulus concept is used to calculate the moment-curvature relations of buckling strength and tangent modulus theory is used. Numerical analysis results are compared with test results on the loading condition of pure bending and 3-point bending.
원발성 담즙성 간경변증에 병발된 전신성 홍반성 루푸스의 임상소견을 갖는 쇼그렌 증후군
이장교 ( Chang Gyo Lee ),장현규 ( Hyun Kyu Chang ),김승용 ( Seung Yong Kim ),강호형 ( Ho Hyung Kang ),김덕재 ( Duk Jae Kim ),장진근 ( Chin Kun Chang ),맹주희 ( Ju Hi Maeng ),이희섭 ( Hui Sup Lee ),박배근 ( Pae Gun Park ),정재걸 ( 대한류마티스학회 2001 대한류마티스학회지 Vol.8 No.1
Primary biliary cirrhosis (PBC) is a chronic inflammatory disease characterized by fibrous obliteration of intrahepatic bile ductules. Although the etiopathogenesis of PBC remains unclear, several studies suggest that a disordered immune response may be involved. This disease is commonly associated with various autoimmune diseases, such as Sjogren`s syndrome, scleroderma, rheumatoid arthritis, Hashimoto`s thyroiditis, polymyositis, and pernicious anemia. More than 80% of cases with PBC are accompanied by at least one autoimmune disorder, and 40% by two or more. Sjogren`s syndrome, defined by dry eyes and xerostomia, may be found in 69 to 81% of PBC patients. However, concurrent coexistence of PBC and systemic lupus erythematosus (SLE) has been rarely described in the literature. We report a 46-year-old female with PBC and Sjogren`s syndrome who also satisfied a classification criteria of SLE.
부신피질 호르몬이 사람골수기질세포의 Apoptosis에 미치는 영향
김하영,김덕재,이시열,홍정수,김동관,김기수 대한내분비학회 2002 Endocrinology and metabolism Vol.17 No.1
Background: Osteoporosis is one of the most serious side effects of long-term glucocorticoid therapy, but the mechanism of glucocorticoid-induced bone loss remains poorly defined. Glucocorticoid induces decreased bone formation and death of isolated segments of bone (osteonecrosis) suggesting that glucocorticoid excess may affect the birth or death rate of bone cells and thereby reduce their numbers. It has been known that reduction in bone formation is due to reduced proliferation in osteoblast precursor cells and reduced matrix synthesis in mature osteoblast. Here, we present evidence for dexamethasone-induced apoptosis on human bone marrow stromal cells (hBMSC). To understand the mechanism of glucocorticoid-induced osteoporosis, we investigated the effects of glucocorticoid on primary cultured hBMSC. Methods: Treatment with dexamethasone at the concentration of 10^-9 M for 3∼5 days significantly decreased cleavage tetrazolium salt WST-1 level/concentration by mitochondrial dehydrogenase in viable cells. Greater decrease was observed with higher concentration of dexamethasone (10^-7 M, and 10^-5 M). Apoptosis was measured by annexin V binding/propidium iodide using fluorescence-activated cell sorter (FACS) analysis and nuclear morphology stained with the fluorescence dye, Hoechst 33342. Results: The level/concentration of apoptotic hBMSC (annexin V positive / PI negative) was increased with 10^-9 M dexamethasone (1.2% to 5.3%) and further increased with 10^-7 M, and 10^-5 M concentration (11.7% and 12.5%, respectively). The same result was observed with Hoechst 33342 staining. Conclusion: These results indicate that glucocorticoid induces apoptosis on osteoblast precursor cell, hBMSC, and may contribute to decrease bone formation
정읍지역 폐경후 여성에서 혈청 렙틴 농도와 골초음파지표와의 상관관계
김하영,김덕재,김기수,김상욱,고정민 대한내분비학회 2002 Endocrinology and metabolism Vol.17 No.1
Background: Obese postmenopausal women usually have a tend to have greater bone mineral density than lean women. This has been attributed to either the mechanical effects of their excessive weight on bone tissue or to their high body fat content. A recent study demonstrated that leptin, the hormone produced in adipocytes, acts on bone metabolism. These findings have prompted speculations on the possible role of leptin in the protective effect of obesity on bone. Methods: We studied the relationship between serum leptin levels and quantitative ultrasound (QUS) values of calcaneus in 94 postmenopausal Korean women who were randomly selected from the population of the Chung-Up osteoporosis prevalence study. QUS values, broadband ultrasound attenuation and speed of sound; were measured at the calcaneus. Results: Leptin values were strongly correlated with body mass index (r = 0.478, p < 0.001), confirming a positive relationship between leptin levels and fat mass. In contrast, no significant correlations were observed between serum leptin levels and calcaneal QUS values. Conclusion: Our results suggest that circulating plasma leptin does not have a significant influence on QUS values of calcaneus in Korean postmenopausal women