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최홍복,신응배,황경엽 漢陽大學校 環境科學硏究所 1996 環境科學論文集 Vol.17 No.-
본 연구는 2차 슬러지의 전처리가 혐기성 소화에 미치는 영향에 관한 연구로 슬러지를 기계적으로 분사 및 충돌시키는 전처리공법에 대한 연구이다. 본 전처리공법의 핵심은 슬러지를 구성하고 있는 미생물 세포를 파괴하는 것으로 이를 위하여 슬러지를 기계적으로 가압하에 분사, 충돌시킨 물리적 힘으로 세포막을 파손시켜 내부 단백질을 노출시켰다. 실험결과 충돌 전후 시료의 단백질 농도 변화가 뚜렷이 나타났다. 단백질 농도는 분사압력이 증가 할 수록 또한 분사횟수가 증가할 수록 높아졌다. 전처리한 슬러지를 회분식 혐기성소화 실험한 결과 체류시간 2∼26일에서 13∼50%의 VS제거효율을 나타냈다. 반면에 같은 운전조건에서 전처리하지 않았을 경우 2∼35%의 VS제거효율을 나타내서 하수의 2차슬러지를 기계적으로 전처리 할 경우에 월등히 높은 혐기성 처리효율을 보이고 있는 것으로 나타났다. This research investigates the effectiveness of sludge pretreatment as it affects the subsequent anaerobic digestion of waste-activated sludge(WAS). The key of this pretreatment process is that bacteria cells in the WAS are ruptured by mechanical jet and smash under pressurized conditions. The protein concentrations in the sludge vary significantly before and after pretreatment. Protein concentration increases according to pressure and smash times. In batch experiments, the removal efficiencies of volatile solids (VS) were 13∼50% when the WAS is pretreated once under 30 bar fed into an anaerobic digester with 2∼26day retention time. Under the same operating conditions, when intact WAS is fed into the digester, VS removal efficiencies are 2∼35%. It is observed that higher digestion efficiencies of sludge can be obtained through mechanical pretreatment of sludge.
황경엽,이윤용,이병철,김재덕 한국화학공학회 1989 Korean Chemical Engineering Research(HWAHAK KONGHA Vol.27 No.4
20-50℃, 100-307bar의 초임계 이산화탄소에 대한 달맞이꽃씨유의 용해도를 측정하고 추출수율에 미치는 온도와 압력의 영향 및 물질전달속도를 구하였다. 용해도는 온도를 일정하게 유지하면 압력이 높아짐에 따라 증가하였다. 그러나 용해도에 미치는 온도의 영향은 압력에 따라 다르게 나타남을 알 수 있었다. 107-200bar일 때는 온도가 높아짐에 따라 용해도는 감소하였고 250bar일 때는 거의 일정하다가 300bar에서는 온도가 높아짐에 따라 증가하였다. 추출수율에 미치는 온도와 압력의 영향도 용해도에서의 경향과 일치하였다. 이산화탄소의 유량이 3.5-4.0ℓ/min(STP)일 때, 본 연구의 실험범위에서 최적 추출조건은 50℃, 300bar이며, 이때 추출수율은 약 2l wt%였다. 유지기준 추출수율이 약 50%될 때까지 loading량은 거의 일정하였고, 그 후 급격한 감소가 일어났으며, 평균 loading량은 체류시간에 따라 증가하였다. 또한 물질전달 parameter, k_(c)a는 이산화탄소의 선속도에 따라 선형적으로 증가하였다. Solubilities of evening primrose oil(EPO) in supercritical carbon dioxide were measured at temperatures of 20-50℃ and pressures of 100 to 300 bars. Solubilities of EPO were found to increase with increasing pressure at constant temperature. When pressure vas kept constant. solubilities of EPO decreased with increase in temperature at pressures of 100 to 200 bars, while they increased with higher temperatures at 300 bar. Solubilities at 250 bar were of a constant value. The effects of temperature and pressure on the extraction yield coincided with the tendency in solubility. Under the range covered in this study the highest yield of extraction was 21 wt% at the temperature of 50℃ and the pressure of 300 bar with the flow rate of carbon dioxide of 3.5-4.0 ℓ/min (STP). Loading of oil in supercritical carbon dioxide was constant until the extraction yield based on available oil was less than about 50%, and then it decreased sharply. The average loadings increased with increase in residence time. Mass transfer parameter, k_(c)a, increased linearly with the superficial velocity of carbon dioxide in the extractor.
Rg3-enriched red ginseng extract promotes lung cancer cell apoptosis and mitophagy by ROS production
Soon-Kyung Hwang,Yun-Jeong Jeong,Hyun-Ji Cho,Yoon-Yub Park,Kwon-Ho Song,Young-Chae Chang 고려인삼학회 2022 Journal of Ginseng Research Vol.46 No.1
Background: Red Ginseng has been used for many years to treat diseases. Ginsenoside Rg3 has documented therapeutic effects, including anticancer and anti-inflammatory activities. However, the anticancer effect of Rg3-enriched red ginseng extract (Rg3-RGE) and its underlying mechanisms have not been fully explored. We investigated whether Rg3-RGE plays an anti-tumor role in lung cancer cells. Methods: To examine the effect of Rg3-RGE on lung cancer cells, we performed cell viability assays, flow cytometry, western blotting analysis, and immunofluorescence to monitor specific markers. Results: Rg3-RGE significantly inhibited cell proliferation and induced mitochondria-dependent apoptosis. Furthermore, Rg3-RGE also increased expression of mitophagy-related proteins such as PINK1 and Parkin. In addition, treatment with Rg3-RGE and mitophagy inhibitors stimulated cell death by inducing mitochondria dysfunction. Conclusions: Rg3-RGE could be used as a therapeutic agent against lung cancer.