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오리엔탈나리의 球根 收穫時期와 低溫處理 期間이 生育에 미치는 影響
金熙峻,金正萬,金致善,柳汀,崔泳根,文炳永 한국화훼연구회 2001 화훼연구 Vol.9 No.1
This study was carried out to determine the effect of the duration of chilling treatment and the lifting time of bulbs on growth of Lilium Oriental hybrids for product of cut flower in the type of forcing or semiforcing culture in Korea. "Casablanca", "Acapulco" and "Le Reve" in lilium Oriental hybrids were used in these experiment and carried out from 1999 to 2000 in Iksan, Korea. The bulbs for experiment were lifted on August 27, September 27 and October 27 and the all of them were chilled at 5±1℃ for 0,8,10,12 weeks. Development of the bulbs lifted on October 27 was superior than that of lifted bulbs on August 27 in the three cultivars. The bulbs lilted lately and chilled for a long time were sprouted more earlier and grown faster. In order to increase bulb sprouting until 100%, the duration of chilling treatment of bulbs was need for 12 weeks on August 27, 10 weeks on September 27 and above 8 weeks on October 27. The flowering according to the duration of chilling treatment and the lifting time of bulbs was same above. Consequently, the bulbs should be chilled at 5±1℃ for chilled for 10 weeks at late September and 8 weeks in producted bulbs at late October in earlyㆍMidseason variety fur product of cut flower hut bulbs of late variety should be chilled for 12 weeks at late September and 10 weeks at late October.
한국형 보행자 사고재현모형의 실제 사고 적용을 통한 충돌지점 규명에 대한 연구
류태선(Ryu, Tae-Seon),이홍기(Lee, Hong-Gi),장영채(Jang, Yeong-Chae) 대한교통학회 2007 대한교통학회 학술대회지 Vol.55 No.-
현재까지 보행자사고의 재현 및 분석에 관한 연구는 주로 외국에서 인체모형(Durnmy) 또는 시체(Cadaver)의 실차실험(Full Scale test)을 통해 보행자의 충돌속도와 전도거리간의 관계식만을 제시하고 있는 수준으로 이루어져 왔다. 그러나 이러한 관계식은 실제 사고에서의 주된 쟁점사항인 보행자의 충돌지점을 규명하는 데에 있어서 대개의 교통사고에서 그렇듯이 자동차의 충돌속도를 정확히 산정하기 어렵기 때문에 사고해석에 별반 도움을 주지 못하고 있는 실정이며, 또한 외국산 자동차와 외국 보행자를 기준으로 실험식이 유도되어 무분별한 국내 보행자사고에의 적용은 심각한 오류를 낳을 수도 있다.
Lee, Seon-Yeong,Kwok, Seung-Ki,Son, Hye-Jin,Ryu, Jun-Geol,Kim, Eun-Kyung,Oh, Hye-Jwa,Cho, Mi-La,Ju, Ji Hyeon,Park, Sung-Hwan,Kim, Ho-Youn BioMed Central 2013 Arthritis research & therapy Vol.15 No.1
<P><B>Introduction</B></P><P>Fibroblast-like synoviocytes (FLSs) are a major cell population of the pannus that invades adjacent cartilage and bone in rheumatoid arthritis (RA). The study was undertaken to determine the effect of interleukin-17 (IL-17) on the survival and/or proliferation of FLSs from RA patients and to investigate whether signal tranducer and activator of transcription 3 (STAT3) is implicated in this process.</P><P><B>Methods</B></P><P>Bcl-2 and Bax expression in FLSs was determined using the real-time PCR and western blot analysis. The expression of Bcl-2 and phosphoSTAT3 in synovial tissues was investigated by confocal microscope. Apoptosis of FLSs was detected by Annexin V/propidium iodide staining and/or phase contrast microscopy. The proliferation of FLSs was determined by CCK-8 ELISA assay.</P><P><B>Results</B></P><P>The pro-apoptotic Bax is decreased and anti-apoptotic Bcl-2 is increased in FLSs from RA patients compared with those from patients with osteoarthritis (OA). IL-17 upregulated the expression of Bcl-2 in FLSs from RA patients, but not in FLSs from OA patients. STAT3 was found to mediate IL-17-induced Bcl-2 upregulation in FLSs from RA patients. Additionally, IL-17 promoted the survival and proliferation of FLSs from RA patients. Most importantly, treatment with STAT3 inhibitor reversed the protective effect of IL-17 on FLSs apoptosis induced by sodium nitroprusside (SNP).</P><P><B>Conclusions</B></P><P>Our data demonstrate that STAT3 is critical in IL-17-induced survival of FLS from RA patients. Therefore, therapeutic strategies that target the IL-17/STAT3 pathway might be strong candidates for RA treatment modalities.</P>
Ok, Seon-Yeong,Cho, Kwon-Koo,Kim, Ki-Won,Ryu, Kwang-Sun Royal Swedish Academy of Sciences 2010 Physica scripta Vol.2010 No.t139
<P>Well-ordered TiO<SUB>2</SUB> nanotube arrays were fabricated by the potentiostatic anodic oxidation method using pure Ti foil as a working electrode and ethylene glycol solution as an electrolyte with the small addition of NH<SUB>4</SUB>F and H<SUB>2</SUB>O. The influence of anodization temperature and time on the morphology and formation of TiO<SUB>2</SUB> nanotube arrays was examined. The TiO<SUB>2</SUB> nanotube arrays were applied as a photoelectrode to dye-sensitized solar cells. Regardless of anodizing temperature and time, the average diameter and wall thickness of TiO<SUB>2</SUB> nanotube arrays show a similar value, whereas the length increases with decreasing reaction temperature. The conversion efficiency is very low, which is due to a morphology breaking of the TiO<SUB>2</SUB> nanotube arrays in the manufacturing process of a photoelectrode.</P>
Lee, Seon-Yeong,Jung, Young Ok,Ryu, Jun-Geol,Oh, Hye-Jwa,Son, Hye-Jin,Lee, Seung Hoon,Kwon, Jeong-Eun,Kim, Eun-Kyung,Park, Mi-Kyung,Park, Sung-Hwan,Kim, Ho-Youn,Cho, Mi-La Society for Leukocyte Biology 2016 Journal of Leukocyte Biology Vol.100 No.3
<P>EGCG attenuates arthritis and osteoclastogenesis by inhibition of Th17 and reciprocal induction of T-reg. The green tea polyphenol epigallocatechin-3-gallate is a potent antioxidant. Here, we describe the effects of epigallocatechin-3-gallate on T cell differentiation and osteoclast differentiation in an animal model of arthritis. Mice with collagen-induced arthritis were injected intraperitoneally with epigallocatechin-3-gallate, 3 times/wk after the primary immunization. Surface markers of T helper 17 cells and regulatory T cells were analyzed by flow cytometry. Flow cytometry, Western blotting, and enzyme-linked immunosorbent assays were used to evaluate the effect of epigallocatechin-3-gallate on cell signaling in the collagen-induced arthritis model. Epigallocatechin-3-gallate decreased the arthritis index and showed protective effects against joint destruction in collagen-induced arthritis mice. The expression of cytokines, oxidative stress proteins, and phosphorylated-signal transducer and activator of transcription-3, 705 and 727, were significantly less in mice treated with epigallocatechin-3-gallate than it was in controls. Epigallocatechin-3-gallate reduced the expression of osteoclast markers in vitro and in vivo relative to the control, and the antiosteoclastic activity was observed in epigallocatechin-3-gallate-treated, interferon- knockout mice. The proportion of forkhead box protein 3-positive regulatory T cells was increased in the spleens of mice treated with epigallocatechin-3-gallate compared with control mice, whereas the proportion of T helper 17 cells was reduced. In vitro, the expression of nuclear respiratory factor 2, heme oxygenase-1, and extracellular signal-regulated kinase was increased significantly by epigallocatechin-3-gallate. We demonstrated that the administration of epigallocatechin-3-gallate attenuated the symptoms of arthritis, inhibited osteoclastogenesis and T helper 17 cell activation, and increased the number of regulatory T cells. At the molecular level, the antiarthritic effects of epigallocatechin-3-gallate may be due to induction of phosphorylated-extracellular signal-regulated kinase, nuclear respiratory factor 2, and heme oxygenase-1 and inhibition of signal transducer and activator of transcription-3 activation.</P>