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Openflow & sFlow-based Flows Visualization
Gwan-Woo Kang,Muhammad Afaq,,Jea-Rin KIM,Wang-Cheol Song 중소기업융합학회 2015 중소기업융합학회 국제학술대회논문집 Vol.2 No.1
In Data center network, the overall traffic consists of large flows and small flows which gives rise to bottleneck like situation when small flows are stuck behind large flows. In this paper, we propose an approach by means of which flows in the network can be visualized and eventually can be very easily rescheduled.
화학공정에서의 안전성 자동 분석기 AHA 의 개발에 관한 연구
강병관(Byoung Gwan Kang),강경욱(Kyung Wook Kang),이병우(Byung Woo Lee),서정철(Jung Chul Suh),윤인섭(En Sup Yoon) 한국화학공학회 1999 Korean Chemical Engineering Research(HWAHAK KONGHA Vol.37 No.3
There are many kinds of complicated equipment in chemical plants. So, the chemical plants always have high possibility of accidents. Hazard analysis is one of the basic tasks to ensure the safety of chemical plants, but it is an arduous, tedious, time-consuming work and requires multidisciplinary knowledge, and demands considerable cognitive load from the analysts. To overcome these problems, there have been various attempts to automate this work by utilizing computer technology, particularly knowledge-based technique. However, many of the past approaches are lacking in properties: safeguard consideration, accident diversity, cause and consequence diversity, pathways leading to accidents, and various hazard analysis reasoning. Therefore, in this study, deviation analysis algorithm, malfunction analysis algorithm, and accident analysis algorithm were proposed using multimodel approach of representing a chemical process adequately for hazard analysis in view of function, behavior, structure, and material property. Unit knowledge base is devised to model a process unit. It consists of unit behavior model and unit function model. In unit knowledge base, a process unit is modeled in different terms of variable and function. Variables describing unit are divided into inlet, internal, and outlet variables explicitly, by interconnecting behavior model and function model through these variables. This model represents physical hazards. In structure model, process is separated into several streams which represent different connections. In material knowledge base, chemical properties are considered according to NFPA code. A hazard analysis system, AHA, was developed on G2. The system was applied to feed section
Five-year Clinical follow-up after Revascularization for Chronic Total Coronary Artery Occlusion
Woo Seok Park,Myung Ho Jeong,Eun Suk Shin,Ju Hyup Yum,Seung Hyun Lee,Young Joon Hong,Ock Young Park,Ju Han Kim,Weon Kim,Young Keun Ahn,Jeong Gwan Cho,Jong Chun Park,Jung Chaee Kang Korean Society of Critical Care Medicine 2005 Acute and Critical Care Vol.20 No.1
BACKGROUND: Chronic total occlusion (CTO) has been considered as an unsuitable lesion for percutaneous coronary intervention (PCI) because of technical difficulty and low success rate. Owing to technical advances and increased operator's experience, PCI has been attempted in a large number of patients with CTO in recent years, but there are few long-term follow-up reports for PCI to CTO. METHODS: We analyzed 83 patients (59.7+/-9.2 years, 28 female) with CTO on diagnostic coronary angiogram at the Heart Center and Coronary Care Unit of Chonnam National Hospital from January 1996 to July 1997. The patients were divided into two groups according to revascularization by PCI or CABG (coronary artery bypass graft): the revascularized group (received PCI or CABG, Group I) and non-revascularized group (Group II). RESULTS: PCI was tried in 46 patients and successful in 33 patients (71.7% of the success rate). Eleven patients (13.3%) were treated with the coronary artery bypass graft (CABG) and 31 (37.3%) patients were medically treated. During 5-year clinical follow-up 11 patients died [13.1%; cardiac death 6 (7.1%), non-cardiac death 5 (6.0%)] and the major adverse cardiac events occurred to 24 (28.6%) patients. Cardiac death occurred to one patient of the Group I and 5 patients of Group II (p=0.06). The mean survival time was significantly different (57.8+/-9.2 months in Group I and 50.9+/-19.5 months in Group II, p=0.038). CONCLUSIONS: Revascularization for CTO prolonged the mean survival time of the patients on long- term clinical follow-up.
Woo, Sung Woong,Kim, Jae Hoon,Kang, Hee In,Kim, Deok Ryeong,Moon, Byung Gwan,Kim, Joo Seung The Korean Neurosurgical Society 2015 Journal of Korean neurosurgical society Vol.58 No.4
Objective : The goal of this study was to assess the effect of high-dose simvastatin on cerebral vasospasm and its clinical outcome after aneurysmal subarachnoid hemorrhage (SAH) in Korean patients. Methods : This study was designed as a prospective observational cohort study. Its subjects were aneurysmal SAH patients who had undergone aneurysm clipping or coiling. They were assigned to 1 of 3 groups : the 20 mg, 40 mg, and 80 mg simvastatin groups. The primary end-point was the occurrence of symptomatic vasospasm. The clinical outcome was assessed with the modified Rankin Scale (mRS) score after 1 month and 3 months. The risk factors of the development of vasospasm were assessed by logistic regression analysis. Results : Ninety nine patients with aneurysmal SAH were treated and screened. They were sequentially assigned to the 20 mg (n=22), 40 mg (n=34), and 80 mg (n=31) simvastatin groups. Symptomatic vasospasm occurred in 36.4% of the 20 mg group, 8.8% of the 40 mg group, and 3.2% of the 80 mg group (p=0.003). The multiple logistic regression analysis showed that poor Hunt-Hess grades (OR=5.4 and 95% CI=1.09-26.62) and high-dose (80 mg) simvastatin (OR=0.09 and 95% CI=0.1-0.85) were independent factors of symptomatic vasospasm. The clinical outcomes did not show a significant difference among the three groups. Conclusion : This study demonstrated that 80 mg simvastatin treatment was effective in preventing cerebral vasospasm after aneurysmal SAH, but did not improve the clinical outcome in Korean patients.
Kang, Jiho,Boonanantanasarn, Kanitsak,Baek, Kyunghwa,Woo, Kyung Mi,Ryoo, Hyun-Mo,Baek, Jeong-Hwa,Kim, Gwan-Shik Korean Academy of Periodontology 2015 Journal of Periodontal & Implant Science Vol.45 No.3
Purpose: Sclerostin, an inhibitor of Wnt/${\beta}$-catenin signaling, exerts negative effects on bone formation and contributes to periodontitis-induced alveolar bone loss. Recent studies have demonstrated that serum sclerostin levels are increased in diabetic patients and that sclerostin expression in alveolar bone is enhanced in a diabetic periodontitis model. However, the molecular mechanism of how sclerostin expression is enhanced in diabetic patients remains elusive. Therefore, in this study, the effect of hyperglycemia on the expression of sclerostin in osteoblast lineage cells was examined. Methods: C2C12 and MLO-Y4 cells were used in this study. In order to examine the effect of hyperglycemia, the glucose concentration in the culture medium was adjusted to a range of levels between 40 and 100 mM. Gene expression levels were examined by quantitative reverse transcription-polymerase chain reaction and Western blot assays. Top-Flash reporter was used to examine the transcriptional activity of the ${\beta}$-catenin/lymphoid enhanced factor/T-cell factor complex. Tumor necrosis factor-alpha ($TNF{\alpha}$) protein levels were examined with the enzyme-linked immunosorbent assay. The effect of reactive oxygen species on sclerostin expression was examined by treating cells with 1 mM $H_2O_2$ or 20 mM N-acetylcysteine. Results: The high glucose treatment increased the mRNA and protein levels of sclerostin. High glucose suppressed Wnt3a-induced Top-Flash reporter activity and the expression levels of osteoblast marker genes. High glucose increased reactive oxygen species production and $TNF{\alpha}$ expression levels. Treatment of cells with $H_2O_2$ also enhanced the expression levels of $TNF{\alpha}$ and sclerostin. In addition, N-acetylcysteine treatment or knockdown of $TNF{\alpha}$ attenuated high glucose-induced sclerostin expression. Conclusions: These results suggest that hyperglycemia increases sclerostin expression via the enhanced production of reactive oxygen species and $TNF{\alpha}$.