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공명선,조준상,이학민,김경호 경북대학교 센서기술연구소 1991 센서技術學術大會論文集 Vol.2 No.1
고분자 습도센서의 감습재료로 이용하고자 4-vinylpyridine (4-VP) 과 methylmethacrylate(MMA) 의 공중함체를 전극에 도포한 후 α, ω-dihaloalkane으로 4 차 암모늄화시킨 후 습도변화에 따른 전기저항을 측정한 결과 소수성 모노머인 MMA의 양이증가할 수록 전기저항이 증가하였으며, 또한 α, ω-dihaloalkane에 의한 4차 암모늄화 반응시간에 따라서도 전기저항의 크기 및 감습막의 안정성에 차이가 있음을 알 수 있었다. In order to for a humidity sensitive material of polymeric humidity sensor, a copolymer of 4-vinylpyridine (4-VP) and methylmethacrylate (IBA) is coated on the electrode and then 4 quartermization by α, ω-dihaloalkane, and thereafter measured the electrical resistance due to humidity variation. As a result, the more content of MMA which is a hydrophobic monomer is increased, the more property of electrical resistance is increased. Also, according to the reaction time in 4 quarternization by α, ω-dihaloalkane we can see the diferences of the magnitude of electrical resistance and the stability of humidity -sensitive membrane.
A Case Report of Fast Growing Mediastinal Desmoid Tumor
( Joon Hyung Lee ),( Jae Seok Jeong ),( So Ri Kim ),( Seung Yong Park ),( Yeong Hun Choe ),( Heung Bum Lee ),( Seoung Ju Park ),( Gong Yong Jin ),( Myoung Ja Chung ),( Ja Hong Kuh ),( Yong Chul Lee ) 대한결핵 및 호흡기학회 2015 대한결핵 및 호흡기학회 추계학술대회 초록집 Vol.120 No.-
Desmoid tumors (DTs) are rare and benign soft tissue tumors that result from monoclonal proliferation of well-differentiated fibroblasts. Since DTs tend to infiltrate and compress adjacent structures, the location of DTs is one of the most crucial factors in determining the severity of the disease. Furthermore, DTs can further complicate the clinical course of the patients whenthe growth is remarkably rapid, especially for DTs occurring in anatomically critical compartments including the thoracic cavity. We report a case of a 71-year-old man with a known mediastinal mass incidentally detected 4 months ago, presenting dyspnea with right-sided atelectasis and massive pleural effusion. Imaging studies revealed a fibrous, 16.4 cm×9.4 cm-sized anterior mediastinal mass with high glucose metabolism. The mass infiltrated into the chest wall and also shifted the mediastinum contralaterally. Interestingly, the tumor had an extremely rapid doubling time of 31.3 days. En bloc resection of the tumor was performed as a curative as well as a diagnostic measure. Histopathological examination showed spindle cells with low cellularity and high collagen deposit in the stroma. Immunohistochemical staining showed the positivity for nuclear β -catenin. Based on these pathologic findings, the mass was diagnosed as DT. After the surgery, there has been no evidence of recurrence of disease in this patient.
Change in gene expression of mouse embryonic stem cells derived from parthenogenetic activation
Gong, Seung Pyo,Kim, Heebal,Lee, Eun Ju,Lee, Seung Tae,Moon, Sunjin,Lee, Ho-Joon,Lim, Jeong Mook Oxford University Press 2009 Human reproduction Vol.24 No.4
<P>BACKGROUND: We previously established parthenogenetic mouse embryonic stem cells (ESCs) and this study was subsequently conducted for elucidating the influence of oocyte parthenogenesis on gene expression profile of ESCs. METHODS: Gene expression of parthenogenetic ESC (pESC)-1 or pESC-2 was separately compared with that of two normally fertilized ESC (nfESC) lines (B6D2F1 and R1 strains), and quantification of mRNA expression was conducted for validating microarray data. RESULTS: In two sets of comparison, reaction of 11 347 and 15 454 gene probes were altered by parthenogenesis, while strain difference changed the expression of 15 750 and 14 944 probes. Level of correlation coefficient was higher in the comparisons between normal fertilization and parthenogenesis (0.974-0.985) than in the comparisons between strains of nfESCs (0.97-0.971). Overall, the expression of 3276-3329 genes was changed after parthenogenesis, and 88% (96/109) of major functional genes differentially (P < 0.01) expressed in one comparison set showed the same change in the other. When we monitored imprinted genes, expression of nine paternal and eight maternal genes were altered after parthenogenesis and 88% (14/16) of these was confirmed by mRNA quantification. CONCLUSIONS: The change in gene expression after parthenogenesis was similar to, or less than, the change induced by a strain difference under a certain genetic background. These results may suggest the clinical feasibility of parthenogenesis-derived, pluripotent cells.</P>
차량 보수도장 보험사기 규명을 위한 수용성 페인트 성분분석
이준배 ( Joon-bae Lee ),이천호 ( Cheon-ho Lee ),유승진 ( Seung-jin Ryu ),공보경 ( Bokyoung Gong ),권오성 ( O-seong Kwon ),김명덕 ( Myung-duck Kim ),김남이 ( Nam Yee Kim ),팽기정 ( Ki Jung Paeng ) 한국공업화학회 2017 공업화학 Vol.28 No.4
최근 차량 증가와 더불어 이에 따른 사고 또한 증가하고 있는 추세로써 대부분의 사고차량에서 도색작업이 반드시 요구되고 있다. 통상적인 차량 도색시 다량의 유기용제가 사용되어 환경문제가 대두되고 있으며 이에 대처하기 위해 최근 수용성 도료가 국내에서도 개발되고 그 사용이 증가하고 있다. 그러나 수용성 도료의 경우 도장 작업비용이 고가이기에 정비공장에서 보험금 과다청구의 수단으로 악용하는 사례 또한 적지 않다. 본 연구에서는 수용성 도료를 사용하였는지 여부를 규명하기 위해 수용성 도료에 사용되는 비이온성 계면활성제 성분인 Surfynol 104 성분을 지표물질로 하여 퀴리포인트를 이용한 열분해-기체크로마토그래피/질량분석법(Py-GC/MS)으로 분석하였다. 분석결과 도색작업이 완료된 실제 차량 페인트에서 동위원소 치환된 fluoranthene-d10을 내부표준물질로 하여 표준물첨가법(standard addition method)으로 약 0.38%의 계면활성제 성분이 검출되었다. With increasing the number of vehicles, the accident rate also goes up and the damaged vehicles should be painted as a final repair process. At the painting stage the solvent-based paint causes environmental problems. To overcome these problems waterborne refinish paint is frequently used recently. However, for waterborne refinish, the costs of insurance coverage are too expensive, and insurance reimbursement costs could be burdensome. Because of the high price of aquatic paint treatment, the service shop might charge the malicious service price. In this study, the surfactant of Surfynol 104, which is the component in the paint, was used as an indicator whether the vehicle was painted with waterborne paint. The specimen was quantitatively analyzed to contain 0.38% of the surfactant through the standard addition method with isotope substituted internal standard (IS) of fluranthene-d10 by curie point pyrolysis-gas chromatography/mass spectrometry (Py-GC/MS).
Lee, Gong-Rak,Jang, Soo Hwa,Kim, Chang Jae,Kim, Ah-Ram,Yoon, Dong-Joon,Park, Neung-Hwa,Han, In-Seob Taylor Francis ; Rapid Science Publishers 2014 Clinical & experimental metastasis Vol.31 No.8
<P>Cholangiocarcinoma is one of the most difficult malignancies to cure. An important prognostic factor is metastasis, which precludes curative surgical resection. Recent evidence shows that capsaicin has an inhibitory effect on cancer cell migration and invasion. Here, we investigated the molecular mechanism of the capsaicin-induced anti-migration and anti-invasion effects on HuCCT1 cholangiocarcinoma cells. Migration and invasion were significantly reduced in response to capsaicin. Capsaicin also inhibited the expression of matrix metalloproteinase-9 (MMP-9). In capsaicin-treated cells, levels of phosphorylated AMPK increased, and this effect was abolished by treatment with the AMPK inhibitor, Compound C. Capsaicin enhanced the expression of SIRT1, which can activate the transcription factor NF-κB by deacetylation. This suggests that NF-κB is activated by capsaicin via the SIRT1 pathway. In addition, capsaicin-activated AMPK induced the phosphorylation of IκBα and nuclear localization of NF-κB p65. Chromatin immunoprecipitation assays demonstrated that capsaicin reduced MMP-9 transcription by inhibiting NF-κB p65 translocation and deacetylation via SIRT1. These findings provide evidence that capsaicin suppresses the migration and invasion of cholangiocarcinoma cells by inhibiting NF-κB p65 via the AMPK-SIRT1 and the AMPK-IκBα signaling pathways, leading to subsequent suppression of MMP-9 expression.</P>