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재생 불량성 빈혈 환자에서 B 형 및 C 형 간염 바이러스 간염에 대한 분석 평가
김남진(Nam Jin Kim),임우현(Woo Hyun Lim),유상두(Sang Doo You),김명성(Myeong Seong Kim),서순팔(Soon Pal Suh),이현철(Hyun Chul Lee),황태주(Tae Joo Hwang),김세종(Sei Jong Kim) 대한내과학회 1998 대한내과학회지 Vol.55 No.3
Objective: Aplastic anemia is a rare but serious complication of viral hepatitis. Both aplastic anemia and viral hepatitis are more common in Korea than in the Western countries. It is necessary to study about the relationship between them. Methods: Twenty-three patients with aplastic anemia visiting Chonnam University Hospital from 1995 to 1996 were studied for positivity of hepatitis B virus (HBV) and hepatitis C virus (HCV) to investigate the association of hepatitis virus infection with aplastic anemia. The surface antigen of HBV (HBsAg) and anti-HCV in sera were tested by EIA(enzyme immunoassay), and the presence of HBV-DNA and HCV-RNA in both sera and bone marrow cells was examined by the polymerase chain reaction (PCR). Results: The positivities of HBsAg and anti-HCV in 23 patients with aplastic anemia were 4.3% (1 patient) and 8.7% (2 patients), respectively. The positivity of HBsAg is similar to that of HBsAg in general population of Korea. The positivity of anti-HCV is higher than that of anti-HCV in general population of Korea. One patient had HBV DNA and 3 patients had HCV HNA in their sera. All of the 3 hepatitis C viremic patients received 11 to 15 units of blood products in the past. None of the patients showed the evidence of recent viral hepatitis infection. HBV DNA and HCV RNA were not detected by the PCR in bone marrow cells in any of the patients. Conclusion: This study suggests that the HBV or HCV might not be a causative agent of aplastic anemia. The higher positivity of anti-HCV in the patients might be due to passive transmission of EICV after transfusion of blood products.
궤양성 대장염 환자에서 혈청 Interleukin-10과 Interleukin-12 측정에 의한 Th1/Th2 반응의 평가
최성규,김태연,김현수,주영은,김세종,유종선,서강석,민상운,유상두 대한소화기학회 2000 대한소화기학회지 Vol.35 No.1
Background/Aims: Previous studies showed that ulcerative colitis (UC) was a T-helper 2 (Th2) response dominant disease while Crohn's disease was a T-helper 1 (Th1) response dominant disease. In this study, we evaluated the Th1 and Th2 reponses by measuring the serum levels of interleukin (IL)-10 and IL-12 in patients with UC and normal controls. Methods: Serum concentrations of IL-10 and IL-12 were measured using an enzyme linked immunosorbent assay in 24 patients (15 with mild to moderatie disease, 9 with severe disease) with UC and 11 healthy controls. Results: The serum concentrations of IL-10 in patients with UC were significantly higher than those of controls (P$lt;0.05), while the concentrations of IL-12 in the patients were significantly lower than those of controls (P$lt;0.05). Additionally, the serum concentrations of IL-10 and IL-12 were not closely related to the severity of the disease. Conclusions: The above results suggest that UC may be resulted from dysregulation of Th1 and Th2 immune response. However, there was no definite correlation between the severity of disease and the level of IL-10 or IL-12.