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Hui-Chen Tsai,Julia Yu-Fong Chang,Chia-Chun Tu,Chung-Chen Jane Yao 대한치과교정학회 2023 대한치과교정학회지 Vol.53 No.2
Before progress was recently made in the application of temporary anchorage devices (TADs) in bio-mechanical design, orthodontists were rarely able to intrude molars to reduce upper posterior dental height (UPDH). However, TADs are now widely used to intrude molars to flatten the occlusal plane or induce counterclockwise rotation of the mandible. Previous studies involving clinical or animal histological evaluation on changes in periodontal conditions after molar intrusion have been reported, however, studies involving human histology are scarce. This case was a Class I malocclusion with a high mandibular plane angle. Upper molar intrusion with TADs was performed to reduce UPDH, which led to counterclockwise rotation of the mandible. After 5 months of upper molar intrusion, shortened clinical crowns were noticed, which caused difficulties in oral hygiene and hindered orthodontic tooth movement. The mid-treatment cone-beam computed tomography revealed redundant bone physically interfering with buccal attachment and osseous resective surgeries were followed. During the surgeries, bilateral mini screws were removed and bulging alveolar bone and gingiva were harvested for biopsy. Histological examination revealed bacterial colonies at the bottom of the sulcus. Infiltration of chronic inflammatory cells underneath the non-keratinized sulcular epithelium was noted, with abundant capillaries being filled with red blood cells. Proximal alveolar bone facing the bottom of the gingival sulcus exhibited active bone remodeling and woven bone formation with plump osteocytes in the lacunae. On the other hand, buccal alveolar bone exhibited lamination, indicating slow bone turnover in the lateral region.
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Hui-Yu Chen 한국중국학회 2020 中國學報 Vol.91 No.-
The characteristic of Korean confucian Ki Jung-jin’s (Lu-Sha, 1798 ~ 1879) theory is that he emphasizes very much on the dominance of principle, and in order to maintain the highest status of Li(理), he does not allow Chi’s(氣) affect. He argues that if the concept of Chi is connected to the concept of Li (Principle), then this is seriously damaging Li’s status. He argues that the “evil” is when “good cannot pervade well". That means when Chi is dominated by Li, it may be limited because it cannot follow Li's rule completely. Therefore, we can say that evil is not caused by other but based on good, and could not express good appropriately. Although “Chi due to some of its components cannot follow the Principle very well” it will not affect Li (Principle) and Li still controls Chi absolutely . But if this argument is true, can we still say that the dominant role of Li is "absolute and inevitable"? More important, if we admit that evil comes from the component of Chi itself, then how do we blame the person who is doing bad? This paper is trying to solve the problem through Kant’s theory of radical evil. If we compare Kant to Lu-sha, we can discover that both of them are emphasize that there is a principle which has absolute dominance, and claim that evil is not caused by other foundation but based on good. That is to say that evil and good are there at the same time. Generally, though both of their theories have many similar points, they are still very different. The key point is that Kant emphasizes that human beings have "volition”, but in Lu-sha’s theory this concept is not described sufficiently. Based on this point, the purpose of this paper is to demonstrate that because Lu-sha’s theory lacks the concept of volition, he cannot explain the theory of evil very well. 韓儒奇正鎮(蘆沙,1798~1879年)的理論特性,在於他不遺餘力地強調理之主宰性是絕對的,並且為了維持性理的至尊無對之地位,他不容有絲毫「氣質」的概念滲入。他主張,若以為必然得關聯著氣質才能完整性理之概念,則這是嚴重地在損害性理的尊貴性。在這一論述下,他以「善之不直遂」來說不「不善」一概念。這指的是,氣質在遵循性理之命令而行時,亦可能有受限於自身材性限制,而導致這一遵循有不順遂之狀態。因此,不善並非是根源於一外於性理的其它根據,而是在以善為根源下而不能恰如其分地、無過無不及地表現之,才產生的。所以,指出一表現可能因氣禀材質本身問題,而有過有不及之狀況,並不影響理對於氣有絕對主宰之義。 只是這一說法若成立,則是否還能穩當地說性理的主宰作用是「絕對的、必然的」?更重要的是,在將「不善」歸咎於「食色的氣禀材性」時,則人之咎責性應如何說明?本文將透過與康德對「根本惡」的說明,來進一步思考此一問題。蘆沙與康德皆十分強調性理(或道德之理)的絕對主宰義,也同時地在說明「不善」之概念時,主張不善與善應是共存,而不是由二個不同的根源而發生的。基本上,二人對於「不善」的論述是有一定程度上的相似。但關鍵的是,康德還強調人有「自由決意」,此一預設便為蘆沙理論中較缺乏了。本文目的即在透過對比康德與蘆沙,在因著強調「自由決意與否」的不同下,二者對「不善」的論述之異同,進而思考蘆沙論惡之合理性,並突顯蘆沙之理論特色。
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( Hui Yu Chen ),( Sung Min Park ),( Jong Hak Lee ),( Xian Hui Meng ),( Dong Wook Shin ),( Ji Beom Yoo ) 대한금속재료학회 ( 구 대한금속학회 ) 2008 ELECTRONIC MATERIALS LETTERS Vol.4 No.4
This paper reports the large scale production of CuO nanoplatelets with smooth surface using a simple hydrothermal method in a water and ethanol solution without any templates or additives. The micro-structure and morphology of the CuO nanoplatelets were examined by X-ray diffraction, Raman spectra, field emission scanning electron microscopy and transmission electron microscopy. The CuO nanoplatelets were monoclinic, and their length and width ranged from 100 to 300 nm and 80 to 120 nm, respectively. A possible growth mechanism for the formation of CuO nanoplatelets was proposed. The volume ratio of ethanol in the solution was found to be a critical effect on the CuO morphology during the hydrothermal stage. The band gap of the CuO nanoplatelets was estimated to be 2.13 eV from the UV-vis spectra.
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Wang, Yu-Qian,Zhang, Hai-Hong,Liu, Chen-Lu,Xia, Qiu,Wu, Hui,Yu, Xiang-Hui,Kong, Wei Asian Pacific Journal of Cancer Prevention 2012 Asian Pacific journal of cancer prevention Vol.13 No.11
Aim: The aim of this study was to investigate the frequency and correlation between auto-antibodies to survivin and MUC1 variable number tandem repeats (VNTR) in colorectal cancer (CRC), which can provide valuable information for the design of immunotherapeutic vaccines for this disease. Methods: Enzyme-linked immunosorbent assays (ELISA) were used to examine the level of auto-antibodies against survivin and MUC1 VNTR in the serum of 135 CRC patients and 95 healthy volunteers. Results: Using mean absorbance + 2 standard deviations (SD) of the healthy samples as a cut-off value, the positive rates of survivin and MUC1 VNTR auto-antibodies in CRC were 31.1% and 18.5%, respectively. Altogether, the survivin and MUC1 VNTR positive samples accounted for 36.3% of the CRC patients, and 7.4% were positive for both. Conclusion: A significant positive correlation was found between levels of specific antibodies against survivin and MUC1 VNTR in the serum of CRC patients (r = 0.3652, P < 0.0001), suggesting that vaccines against both targets would elicit immune responses more effectively.
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陳繪宇 ( Chen Hui-yu ) 한국중국학회 2020 中國學報 Vol.91 No.-
韓儒奇正鎭(號蘆沙, 1798~1879)的理論特性, 在於他不遺餘力地强調理之主宰性是絶對的, 幷且爲了維持性理的至尊無對之地位, 他不容有絲毫「氣質」的槪念滲入。他主張, 若以爲必然得關聯著氣質才能完整性理之槪念, 將嚴重地損害性理的尊貴性。在這一論述下, 他以「善之不直遂」來說明「不善」一槪念。這指的是, 氣質在遵循性理之命令而行時, 亦可能有受限於自身材性限制, 而導致這一遵循有不順遂之狀態。因此, 不善幷非是根源於一外於性理的其它根據, 而是在以善爲根源下而不能恰如其分地、無過無不及地表現之, 才産生的。所以, 蘆沙認爲, 一表現可能因氣稟材質本身問題, 而有過有不及之狀況, 這幷不會影響「理對於氣有絶對主宰」之義。 只是這一說法若成立, 則是否還能穩當地說性理的主宰作用是「絶對的、必然的」?更重要的是, 在將「不善」歸咎於「食色的氣稟材性」時, 則人之咎責性應如何說明?本文將透過與康德對「根本惡」的說明, 來進一步思考此一問題。蘆沙與康德皆十分强調性理(或道德之理)的絶對主宰義, 也同時地在說明「不善」之槪念時, 主張不善與善應是共存, 而不是由二個不同的根源而發生的。基本上, 二人對於「不善」的論述是有一定程度上的相似。但關鍵的是, 康德還强調人有「自由決意」, 此一預設則爲蘆沙理論中較缺乏了。本文目的卽在透過對比康德與蘆沙, 在因著强調「自由決意與否」的不同下, 二者對「不善」的論述之異同, 進而思考蘆沙論惡之合理性, 幷突顯蘆沙之理論特色。 The characteristic of Korean confucian Ki Jung-jin’s (Lu-Sha, 1798 ~ 1879) theory is that he emphasizes very much on the dominance of principle, and in order to maintain the highest status of Li(理), he does not allow Chi’s(氣) affect. He argues that if the concept of Chi is connected to the concept of Li (Principle), then this is seriously damaging Li’s status. He argues that the “evil” is when “good cannot pervade well”. That means when Chi is dominated by Li, it may be limited because it cannot follow Li's rule completely. Therefore, we can say that evil is not caused by other but based on good, and could not express good appropriately. Although “Chi due to some of its components cannot follow the Principle very well” it will not affect Li (Principle) and Li still controls Chi absolutely. But if this argument is true, can we still say that the dominant role of Li is “absolute and inevitable”? More important, if we admit that evil comes from the component of Chi itself, then how do we blame the person who is doing bad? This paper is trying to solve the problem through Kant’s theory of radical evil. If we compare Kant to Lu-sha, we can discover that both of them are emphasize that there is a principle which has absolute dominance, and claim that evil is not caused by other foundation but based on good. That is to say that evil and good are there at the same time. Generally, though both of their theories have many similar points, they are still very different. The key point is that Kant emphasizes that human beings have “volition”, but in Lu-sha’s theory this concept is not described sufficiently. Based on this point, the purpose of this paper is to demonstrate that because Lu-sha’s theory lacks the concept of volition, he cannot explain the theory of evil very well.
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Yang Hui-Hui,Jiang Hui-Ling,Tao Jia-Hao,Zhang Chen-Yu,Xiong Jian-Bing,Yang Jin-Tong,Liu Yu-Biao,Zhong Wen-Jing,Guan Xin-Xin,Duan Jia-Xi,Zhang Yan-Feng,Liu Shao-Kun,Jiang Jian-Xin,Zhou Yong,Guan Cha-Xi 생화학분자생물학회 2022 Experimental and molecular medicine Vol.54 No.-
Necroptosis is the major cause of death in alveolar epithelial cells (AECs) during acute lung injury (ALI). Here, we report a previously unrecognized mechanism for necroptosis. We found an accumulation of mitochondrial citrate (citratemt) in lipopolysaccharide (LPS)-treated AECs because of the downregulation of Idh3α and citrate carrier (CIC, also known as Slc25a1). shRNA- or inhibitor–mediated inhibition of Idh3α and Slc25a1 induced citratemt accumulation and necroptosis in vitro. Mice with AEC-specific Idh3α and Slc25a1 deficiency exhibited exacerbated lung injury and AEC necroptosis. Interestingly, the overexpression of Idh3α and Slc25a1 decreased citratemt levels and rescued AECs from necroptosis. Mechanistically, citratemt accumulation induced mitochondrial fission and excessive mitophagy in AECs. Furthermore, citratemt directly interacted with FUN14 domain-containing protein 1 (FUNDC1) and promoted the interaction of FUNDC1 with dynamin-related protein 1 (DRP1), leading to excessive mitophagy-mediated necroptosis and thereby initiating and promoting ALI. Importantly, necroptosis induced by citratemt accumulation was inhibited in FUNDC1-knockout AECs. We show that citratemt accumulation is a novel target for protection against ALI involving necroptosis.
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Family History of Cancer and Head and Neck Cancer Risk in a Chinese Population
Huang, Yu-Hui Jenny,Lee, Yuan-Chin Amy,Li, Qian,Chen, Chien-Jen,Hsu, Wan-Lun,Lou, Pen-Jen,Zhu, Cairong,Pan, Jian,Shen, Hongbing,Ma, Hongxia,Cai, Lin,He, Baochang,Wang, Yu,Zhou, Xiaoyan,Ji, Qinghai,Zho Asian Pacific Journal of Cancer Prevention 2015 Asian Pacific journal of cancer prevention Vol.16 No.17
Background: The aim of this study was to investigate whether family history of cancer is associated with head and neck cancer risk in a Chinese population. Materials and Methods: This case-control study included 921 cases and 806 controls. Recruitment was from December 2010 to January 2015 in eight centers in East Asia. Controls were matched to cases with reference to sex, 5-year age group, ethnicity, and residence area at each of the centers. Results: We observed an increased risk of head and neck cancer due to first degree family history of head and neck cancer, but after adjustment for tobacco smoking, alcohol drinking and betel quid chewing the association was no longer apparent. The adjusted OR were 1.10 (95% CI=0.80-1.50) for family history of tobacco-related cancer and 0.96 (95%CI=0.75-1.24) for family history of any cancer with adjustment for tobacco, betel quid and alcohol habits. The ORs for having a first-degree relative with HNC were higher in all tobacco/alcohol subgroups. Conclusions: We did not observe a strong association between family history of head and neck cancer and head and neck cancer risk after taking into account lifestyle factors. Our study suggests that an increased risk due to family history of head and neck cancer may be due to shared risk factors. Further studies may be needed to assess the lifestyle factors of the relatives.
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