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엄익태,김진철,임풍 中央醫學社 1990 中央醫學 Vol.55 No.12
Binder's syndrome (Maxillonasal dysplasia) which has previously been known as congenital dish face, scaphoid face, and congenital flat nose syndrome is a skeletal malformation characterized by an extremely flat, retruded nose and class III malocclusion. The etiology is discussed in relation to birth trauma, nasal trauma in childhood, underdevelopment of premaxilla and an inhibition of ossification center in the canine region. The treatment of this syndrome usuallyy require correction of 3 defects; (1) the perialar flatness (due to underdevelopment of the piriform margin); (2) the hypoplastic nose, which is flat and short, and has a small columella; (3) the short maxilla, with its malocclusion. In this paper we present two binder's syndrome with extremely flat and retruded nose, which were corrected by Le Fort II osteotomy, calvarial bone graft and iliac bone graft, respectively. So we obtained satistactory results and report these with review of articles.
엄익태,임 풍,김진철 大韓成形外科學會 1988 Archives of Plastic Surgery Vol.15 No.2
Meningoencephalocele is an extracranial protrusion of the menings, cerebrospinal fluid and some portion of the brain, and is usually classified into;anterior, occipital, temporal and basal types according to the location. In most large series of cases of this disease reported in the literature, the frontoethmoidal meningoencephalocele which is called anterior type constitute only a small percentage. In contrast to other type of meningoencephalocele, the incidence of associated congenital abnormalities is relatively low in this disease. Therefore, the longoterm prognosis following surgery is excellent. Recently, we experienced 2 cases of frontoethmoidal meningoencephaloceles which were treated by intraand extracranial approach, and obtained satisfactory results and report these with review of articles.
엄익태,임 풍,김진철 大韓成形外科學會 1988 Archives of Plastic Surgery Vol.15 No.2
Binder's syndrome (Maxillonasal dysplasia) which has previously been known as congenital dish face, scaphoid face, and congenital flat nose syndrome is a skeletal malformation characterized by an extremely flat, retruded nose and class Ⅲ malocclusion. The etiology is discussed in relation to birth trauma, nasal trauma in childhood, underdevelopment of premaxilla and an inhibition of ossification center in the canine region. The treatment of this syndrome usually require correction of 3 defects; (1) the perialar flatness (due to underdevelopment of the piriform margin); (2) the hypoplastic nose, which is flat and short, and has a small columella; (3) the short maxilla, with its malocclusion. In this paper we present two binder's syndrome with extremely flat and retruded nose, which were corrected by Le Fort Ⅱ osteotomy, calvarial bone graft and iliac bone graft, respectively. So we obtained satistactory results and report these with review of articles.
화상시 T세포 및 자연살해 세포면역능의 변화에 관한 실험적 연구
김영진,백무현,엄익태,조문제,임풍 大韓成形外科學會 1989 Archives of Plastic Surgery Vol.16 No.2
Studies of the immune system in both clinicl and experimental burn injury have noted impairment of cellular immunity and this impaired cellular immunity after burn has been demonstrated by prolonged survival allograft, marked depression on delayed hypersensitivety or failure of lymphocyte mitogenesis. However, the exact nature of the mechanisms leading to immunosuppression remains unclear. The aim of this study is to evaluate the level of cellular immunity in burned rats with immune paramenters of T cell subsets and colony forming unit-T lymphocyte (CFTL) as well as natural killer cell mediated cytotoxicity (NKMC). Lymphocytes were isolated sequentially from the blood of control and experimental animals on the 1st, 3rd, 5th, 7th and 14th postburn days (PBD). T-cell subsets were defined with monoclonal anti rat T lymphocyte antibody by the indirect immunofluoroscence method. The proportions of tatal T cell (Tan T), helper T cell (Th) and helper T cel/suppressor T cell ratio decreased from the 1st PND and revealed most significant reduction on the 5th PBD (Pan T)and 7th PBD (Th, Th/Ts ratio)respectively, but they recovered by the 14th PBC. On the contrary, the percentage of suppressor T cell (Ts) in creased after burn injury and was maximum on the 7th PBD, thereafter it decreased to the nearly control level on the 14th PBD, Number of T-lymphocyte colony (TL-colony) decreased after burn injury, and the changing pattern of its number was quite similar to that of Pan T. The cytotoxicity of natural killer cell against Molt 4 target cells depressed rapidly after burn injury and revealed most significant reduction on the 3rd PBD, but it restored completely on the 7th PBD. Sequential change of cortisol level didn't co-ordinate with that of cellular immunity, and it showed that stress after burn injury did not frnction as a major role to decrese cellular immunity. The results of this study give us a speculation that immunosuppression after burn injury was resulted from decreased helper T cell, increased suppressor T cell and depressed NKMC, and that damage or recovery fo natural killer cells after burn injury is of precedence to that of the T cells.