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소화성 궤양환자의 흡연여부에 따른 혈청 Pepsinogen 1 치의 변화
오재선(Jae Sun Oh),김평남(Pyung Nam Kim),나한식(Han Sik Na),김중남(Jung Nam Kim),문철웅(Chul Oong Moon),김만우(Man Woo Kim),양성훈(Sung Hun Yang) 대한내과학회 1990 대한내과학회지 Vol.39 No.4
N/A Serum group I Pepsinogen (sPG I) levels were measured by radioimmunoassay in 159 patients undergoing endoscopy, then all patients were classified by endoscopic diagnosis, gastric mucosal histology, and smoking habits. The results were as follows: 1) Both nonulcer dyspepsia and duodenal ulcer smokers had significantly higher sPG I levels than their nonsmoking controls (p<0.005). 2) The frequency distvibution of superficial gastritis in the nonulcer dyspcpsia group with smoking habits revealed significantly higher levels than that of the nonsmoking controls (p<0.0025). 3) Serum PG Iin nonulcer dyspepsia patients revealed significantly higher levels in patients with superficial gastritis but there were not related to cigarette smoking. 4) The relationship between the mean level of serum PG I and fundic mucosal histology in duodenal ulcer patients was found to be statistically insignificant, but the mean level of sPG I in the smoking group showed a higher value than that of the nonsmoking controis (p< 0. 05). 5) The frequency distribution of high serum PG I level (> 80 ng/ml) in duodenal ulcer smokers was higher than that of the nonsmoking control group (chi square test: p<0.). From the above findings, it is concluded that the smoking induced increase in sPG I in duodenal ulcer is proposed to release an augmented pepsin secretory capacity, which can have aetiologic significance in association between smoking and duodenal ulcer.
위장관 ( 胃腸管 ) : 정상인에서 호기내 수소농도 측정법을 이용한 유당불내성률
김만우(Man Woo Kim),김평남(Pyoung Nam Kim),나한식(Han Sik Na),오재선(Jae Sun Oh),이승일(Seung Il Lee),박찬국(Chan Guk Park) 대한소화기학회 1990 대한소화기학회지 Vol.22 No.3
N/A In order to detect lactose intolerance in study groups, we have performed the lactose breath test, lactulose breath test, and oral lactose tolerance test. The 50 cases (28 men, 22 women) of this study were selected from healty adults without known disease and free from gastrointestinal symptoms. 1) In 50 subjects, there were 32 subjects who had the intolerant symptoms such as abdominal pain, flatulence and diarrhea. 2) All subjects with intolerant symptoms were more than 20 ppm in breath hydrogen. 3) In two subjects among 11 cases with less than 20 ppm in breath hydrogen by lactose breath test have not increased by lactulose breath test (<10ppm). 4) The prevalence of lactose intolerance in healthy adults was 82 percent. From the above results, it is concluded that lactose breath test offers an attractive, noninvasive way of diagnosing lactose intolerance.