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Pamella Marie Sendon,박종범,서학수,박순기,송종태 한국응용생명화학회 2012 Applied Biological Chemistry (Appl Biol Chem) Vol.55 No.6
Expression patterns of AtBSMT1 encoding an S-adenosyl-L-methionine: salicylic acid carboxyl methyltransferase from Arabidopsis thaliana were analyzed in AtBSMT1 promoter::β-glucuronidase (GUS) transgenic Arabidopsis. AtBSMT1 was expressed in flowers and siliques and induced under specific biotic/abiotic stress conditions. The results indicated that the induction of AtBSMT1 is regulated by plant disease response and jasmonic acid signaling.
Sendon, Pamella Marie,Park, Jong-Beum,Seo, Hak Soo,Park, Soon-Ki,Song, Jong Tae The Korean Society for Applied Biological Chemistr 2012 Applied Biological Chemistry (Appl Biol Chem) Vol.55 No.6
Expression patterns of AtBSMT1 encoding an S-adenosyl-L-methionine: salicylic acid carboxyl methyltransferase from Arabidopsis thaliana were analyzed in AtBSMT1 promoter::${\beta}$-glucuronidase (GUS) transgenic Arabidopsis. AtBSMT1 was expressed in flowers and siliques and induced under specific biotic/abiotic stress conditions. The results indicated that the induction of AtBSMT1 is regulated by plant disease response and jasmonic acid signaling.
Activation of C2H2-type Zinc Finger Genes Induces Dwarfism in Arabidopsis thaliana
Sendon, Pamella Marie,Oo, Moe Moe,Park, Jong-Beum,Lee, Byung Ha,Kim, Jeong Hoe,Seo, Hak Soo,Park, Soon-Ki,Song, Jong Tae The Korean Society for Applied Biological Chemistr 2014 Applied Biological Chemistry (Appl Biol Chem) Vol.57 No.1
Zinc finger proteins compose one of the largest transcription factor families in plants, but only a handful have been functionally specified in plant growth. In this study, a semi-dominant mutant, Arabidopsis thaliana dwarf1 (Atdwa1), was discovered from activation tagging screening using the SKI015 T-DNA vector. The Atdwa1 mutant displayed severe dwarfism and loss of apical dominance, as well as other pleiotropic defects, such as earlier flowering, fewer leaves, and shorter sliliques than those of the wild-type plant. Thermal asymmetric interlaced-polymerase chain reaction analysis revealed that activated T-DNA was inserted into the At5g54330 gene, disrupting its coding sequence. We also demonstrated that At5g54330 expression was nullified by the insertion, whereas expression of its downstream genes, At5g54340, At5g54350, and At5g54360, were activated. A database search revealed that all three downstream genes encode proteins related to C2H2-type zinc finger proteins. Overexpression of each gene resulted in dwarfism similar to that of Atdwa1. These findings indicate that Atdwa1 mutant was caused by synergistic manifestation of concomitant activation of At5g54340 to At5g54360, and that the zinc finger proteins may play a role in regulation of plant growth and development.
Salicylic Acid Signaling: Biosynthesis, Metabolism, and Crosstalk with Jasmonic Acid
Sendon, Pamella Marie,Seo, Hak-Soo,Song, Jong-Tae The Korean Society for Applied Biological Chemistr 2011 Applied Biological Chemistry (Appl Biol Chem) Vol.54 No.4
Salicylic acid (SA) signaling plays an important role in local and systemic acquired resistance. Expression and activity of pathogenesis-related proteins are stimulated by the accumulation of SA, conferring resistance to pathogens. SA can be synthesized via the phenylpropanoid route or the isochorismate pathway and metabolized to form SA-glucoside and SA glucose-ester through glucosylation, and methyl salicylate through methylation. This summary focuses on genes involved in SA biosynthesis, metabolism, and signaling. SA and jasmonic acid (JA) crosstalk has an important role in regulating induced defense against pathogens by exerting antagonistic effects. Therefore, results on crosstalk between SA and JA are also shortly reviewed. Further investigation on the molecular aspect of SA and JA antagonism, elucidating how these pathways are linked to each other, and how they resolve the complexity of host-pathogen interaction will provide a better understanding on SA signaling and plant defense.
Review : Salicylic Acid Signaling: Biosynthesis, Metabolism, and Crosstalk with Jasmonic Acid
( Pamella Marie Sendon ),( Hak Soo Seo ),( Jong Tae Song ) 한국응용생명화학회(구 한국농화학회) 2011 Applied Biological Chemistry (Appl Biol Chem) Vol.54 No.4
Salicylic acid (SA) signaling plays an important role in local and systemic acquired resistance. Expression and activity of pathogenesis-related proteins are stimulated by the accumulation of SA, conferring resistance to pathogens. SA can be synthesized via the phenylpropanoid route or the isochorismate pathway and metabolized to form SA-glucoside and SA glucose-ester through glucosylation, and methyl salicylate through methylation. This summary focuses on genes involved in SA biosynthesis, metabolism, and signaling. SA and jasmonic acid (JA) crosstalk has an important role in regulating induced defense against pathogens by exerting antagonistic effects. Therefore, results on crosstalk between SA and JA are also shortly reviewed. Further investigation on the molecular aspect of SA and JA antagonism, elucidating how these pathways are linked to each other, and how they resolve the complexity of host-pathogen interaction will provide a better understanding on SA signaling and plant defense.
Salicylic Acid Signaling: Biosynthesis, Metabolism,and Crosstalk with Jasmonic Acid
Pamella Marie Sendon,서학수,송종태 한국응용생명화학회 2011 Applied Biological Chemistry (Appl Biol Chem) Vol.54 No.4
Salicylic acid (SA) signaling plays an important role in local and systemic acquired resistance. Expression and activity of pathogenesis-related proteins are stimulated by the accumulation of SA, conferring resistance to pathogens. SA can be synthesized via the phenylpropanoid route or the isochorismate pathway and metabolized to form SA-glucoside and SA glucose-ester through glucosylation, and methyl salicylate through methylation. This summary focuses on genes involved in SA biosynthesis, metabolism, and signaling. SA and jasmonic acid (JA) crosstalk has an important role in regulating induced defense against pathogens by exerting antagonistic effects. Therefore, results on crosstalk between SA and JA are also shortly reviewed. Further investigation on the molecular aspect of SA and JA antagonism, elucidating how these pathways are linked to each other, and how they resolve the complexity of host-pathogen interaction will provide a better understanding on SA signaling and plant defense.
Sendon, Pamella Marie,Park, Jong-Beum,Park, Soon-Ki,Song, Jong Tae Institute of Agricultural Science and Technology 2013 慶北大農學誌 Vol.31 No.2
AtSAGT1 encodes a salicylic acid (SA) glucosyltransferase enzyme that catalyzes the formation of SA glucoside and SA glucose ester. Here, the AtSAGT1 gene expression patterns were determined in AtSAGT1 promoter::GUS transgenic Arabidopsis plants. As a result, the factors regulating the induction of AtSAGT1 were identified as pathogen defense response, wound response, exogenous application of SA, and jasmonic acid treatment.
Activation of C2H2-type Zinc Finger Genes Induces Dwarfism in Arabidopsis thaliana
Pamella Marie Sendon,Moe Moe Oo,박종범,이병하,김정회,서학수,박순기,송종태 한국응용생명화학회 2014 Applied Biological Chemistry (Appl Biol Chem) Vol.57 No.1
Zinc finger proteins compose one of the largesttranscription factor families in plants, but only a handful havebeen functionally specified in plant growth. In this study, a semidominantmutant, Arabidopsis thaliana dwarf1 (Atdwa1), wasdiscovered from activation tagging screening using the SKI015 TDNAvector. The Atdwa1 mutant displayed severe dwarfism andloss of apical dominance, as well as other pleiotropic defects, suchas earlier flowering, fewer leaves, and shorter sliliques than thoseof the wild-type plant. Thermal asymmetric interlaced- polymerasechain reaction analysis revealed that activated T-DNA wasinserted into the At5g54330 gene, disrupting its coding sequence. We also demonstrated that At5g54330 expression was nullified bythe insertion, whereas expression of its downstream genes,At5g54340, At5g54350, and At5g54360, were activated. Adatabase search revealed that all three downstream genes encodeproteins related to C2H2-type zinc finger proteins. Overexpressionof each gene resulted in dwarfism similar to that of Atdwa1. Thesefindings indicate that Atdwa1 mutant was caused by synergisticmanifestation of concomitant activation of At5g54340 toAt5g54360, and that the zinc finger proteins may play a role inregulation of plant growth and development.
Pamella Marie Sendon,Jong-Beum Park,Soon-Ki Park,Jong Tae Song 경북대학교 농업생명과학대학 2013 Current Research on Agriculture and Life Sciences Vol.31 No.2
AtSAGT1 encodes a salicylic acid (SA) glucosyltransferase enzyme that catalyzes the formation of SA glucoside and SA glucose ester. Here, the AtSAGT1 gene expression patterns were determined in AtSAGT1 promoter::GUS transgenic Arabidopsis plants. As a result, the factors regulating the induction of AtSAGT1 were identified as pathogen defense response, wound response, exogenous application of SA, and jasmonic acid treatment.
박종범,Pamella Marie Sendon,So Hyun Kwon,서학수,박순기,김정회,송종태 한국식물학회 2012 Journal of Plant Biology Vol.55 No.5
We have previously shown that overexpression of BrERF4 (Brassica rapa ETHYLENE-RESPONSIVE FACTOR4)increases salt and drought tolerance in Arabidopsis plants,and also retarded organ growth. In the present study, we investigated in detail the leaf growth retardation phenotype at the cellular level. Results showed that BrERF4-overexpressing Arabidopsis plants developed small leaves by reducing their cell size but not the cell number. Detailed kinematic analysis revealed that changes in cell size appeared from the very early stages of leaf development, directly affecting the size of leaf organs. RT-PCR analysis showed that expression of expansin genes was reduced in the overexpressors, whereas expression of the cell cycle gene, CYCB1;1, was not altered at all. In addition, overexpression of AtMYB44, another stress-related transcription factor gene, reduced leaf growth,which also resulted from reduction in cell size but not in cell number. These results suggest that overexpression of those transcription factors negatively affects cell expansion during leaf growth without altering cell number. We discuss about the advantages that the BrERF4- or AtMYB44-induced cell expansion retardation confers on plants under natural environmental adversity.