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윤제원(Je-Won Yoon),김영찬(Young-Chan Kim),김금모(Keum-Mo Kim),장강석(Kang-Seok Jang),구본성(Bon-Sung Ku),엄주용(Joo-Yong Eom) 한국소음진동공학회 2011 한국소음진동공학회 학술대회논문집 Vol.2011 No.10
The purpose of this study is to develop an air-passing soundproofing panel with more improved structure to reduce the CO2 emission and installation cost. To reduce the emission of CO2 ; it is suggested to choose low CO2 emission material relative to the aluminum and to reduce the materials by developing a specially designed air-passing soundproofing panel structure. First of all, we performed the flow analysis to predict the wind pressure according to the open angle of the air-passing soundproofing panel and the noise level analysis at the receiver point. To verify the simulation, a prototype of the soundproofing panel was made. The flow test in the wind tunnel and load test were performed. The economic evaluation for the installation of the air-passing soundproofing panel was performed and specifications of the installation was prepared. As the results of this research, it was verified that the wind load was reduced about 40% to that of the conventional one at 25m/s wind speed in the wind tunnel test. By applying the 4m span soundproofing wall with air-passing soundproofing panel and under the cost of 250 thousand won/m2 instead of the conventional 2m span panel, the installation cost will always be lowered than the conventional one in the combination of (60:40~50:50) conventional to air-passing soundproofing panel from the economic evaluation. The 20% reduction of CO2 was found by changing the 50% of aluminum soundproof panel to air-passing soundproofing panel.
Case Reports : Disseminated Mycobacterium intracellulare Infection in an Immunocompetent Host
( Won Young Kim ),( Sun Joo Jang ),( Tae Jin Ok ),( Gwang Un Kim ),( Han Seung Park ),( Jae Chan Leem ),( Bo Hyoung Kang ),( Se Jeong Park ),( Dong Kyu Oh ),( Byung Ju Kang ),( Bo Young Lee ),( Won Ju 대한결핵 및 호흡기학회 2012 Tuberculosis and Respiratory Diseases Vol.72 No.5
Disseminated Mycobacterium avium complex (MAC) infection can occur in immunocompromised patients, and rarely in immunocompetent subjects. Due to the extensive distribution of the disease, clinical presentation of disseminated MAC may mimic malignancies, and thorough examinations are required in order to make accurate diagnosis. We report a case of disseminated Mycobacterium intracellulare disease in an immunocompetent patient, which involved the lung, lymph nodes, spleen, and multiple bones. F-18 fluorodeoxyglucose positron-emission tomography imaging showed multiple hypermetabolic lesions, which are suggestive of typical hematogenous metastasis. However, there was no evidence of malignancy in serial biopsies, and M. intracellulare was repeatedly cultured from respiratory specimens and bones. Herein, we should know that disseminated infection can occur in the immunocompetent subjects, and it can mimic malignancies.
Structural Health Monitoring Based on Electrical Impedance of a Carbon Nanotube Neuron
Kang, In Pil,Lee, Jong Won,Choi, Gyeong Rak,Jung, Joo Yung,Hwang, Sung Ho,Choi, Yeon Sun,Yoon, Kwang Joon,Schulz, Mark J. Trans Tech Publications, Ltd. 2006 Key Engineering Materials Vol.321 No.-
<P>This paper introduces a new sensor design based on a carbon nanotube structural neuron for structural health monitoring applications. The carbon nanotube neuron is a thin and narrow polymer film sensor that is bonded or deposited onto a structure. The electrochemical impedance (resistance and capacitance) of the neuron changes due to deterioration of the structure where the neuron is located. A network of the long carbon nanotube neurons can form a structural neural system to provide large area coverage and an assurance of the operational health of a structure without the need for actuators and complex wave propagation analyses that are used with other SHM methods. The neural system can also reduce the cost of health monitoring by using biomimetic signal processing to minimize the number of channels of data acquisition needed to detect damage. The carbon nanotube neuron is lightweight and easily applied to the structural surface, and there is no stress concentration, no piezoelectrics, no amplifier, and no storage of high frequency waveforms. The carbon nanotube neuron is expected to find applications in detecting damage and corrosion in large complex structures including composite and metallic aircraft and rotorcraft, bridges, and almost any type of structure with almost no penalty to the structure.</P>
Joo, Seong-Soo,Kang, Hee-Chul,Won, Tae-Joon,Lee, Do-ik The Pharmaceutical Society of Korea 2003 Archives of Pharmacal Research Vol.26 No.12
Ursodeoxycholic acid (UDCA) is a non-toxic, hydrophilic bile acid in widespread clinical use mainly for acute and chronic liver disease. Recently, treatment with UDCA in hepatic graft-versus-host disease has been given in immunosuppressive therapy for improvement of the biochemical markers of cholestasis. Moreover, it has been reported that UDCA possesses immunomodulatory effects by the suppression of cytokine production. In the present study, we hypothesized that UDCA may inhibit the production of the pro-inflammatory cytokine, IL-1$\beta$, and nitric oxide (NO) in microglia. In the study, we found that 100 $\mu$ g/mL UDCA effectively inhibited these two pro-inflammatory factors at 24 hand 48 h, compared to the $A\beta$42-pretreated groups. These results were compared with the LPS+UDCA group to confirm the UDCA effect. As microglia can be activated by several stimulants, such as $A\beta$42, in Alzheimers brain and can release those inflammatory factors, the ability to inhibit or at least decrease the production of IL-1$\beta$ and NO in Alzheimers disease (AD) is essential. Using RT-PCR, ELISA and the Griess Reagent System, we therefore found that UDCA in $A\beta$42 pre-treated cultures played a significant role in suppressing the expression or the production of IL-1$\beta$ and NO. Similarly, lipopolysaccharide (LPS) did not activate microglia in the presence of UDCA. Moreover, we found that UDCA exhibits a prolonged effect on microglial cells (up to 48 h), which suggests that UDCA may play an important role in chronic cell damage due to this long effect. These results further imply that UDCA could be an important cue in suppressing the microglial activation stimulated by massive AD peptides in the AD progressing brain.
Kang, Dong Hoon,Lee, Doo Jae,Kim, Jiran,Lee, Joo Young,Kim, Hyun-Woo,Kwon, Kihwan,Taylor, W. Robert,Jo, Hanjoong,Kang, Sang Won American Heart Association, Inc. 2013 CIRCULATION - Vol.128 No.8
<P><B>Background—</B></P><P>Typical 2-Cys peroxiredoxin (Prx) is inactivated by overoxidation of the peroxidatic cysteine residue under oxidative stress. However, the significance in the context of vascular disease is unknown.</P><P><B>Methods and Results—</B></P><P>Immunohistochemical analyses revealed that 2-Cys Prxs, particularly Prx type II, are heavily overoxidized in balloon-injured rodent carotid vessels and in human atherosclerotic lesions. Consistent with this observation, the selective depletion of Prx II exacerbated neointimal hyperplasia in injured carotid vessels. We also found that the epipolythiodioxopiperazine class of fungal metabolites exhibited an enzyme-like activity mimicking 2-Cys Prx peroxidase and manifestly eliminated the intracellular H<SUB>2</SUB>O<SUB>2</SUB> in the vascular cells. Functionally, the epipolythiodioxopiperazines reciprocally regulated the platelet-derived growth factor receptor-β– and vascular endothelial growth factor receptor–mediated signaling in these vascular cells by replacing Prx II. As a consequence, the epipolythiodioxopiperazines inhibited the proliferative and migratory activities of smooth muscle cells but promoted those of endothelial cells in vitro. Moreover, administration of the epipolythiodioxopiperazines to the injured carotid vessels resulted in a successful recovery by inhibiting neointimal hyperplasia without causing cytotoxicity and simultaneously inducing reendothelialization.</P><P><B>Conclusions—</B></P><P>This study reveals for the first time the involvement of the 2-Cys Prx overoxidation and thus the therapeutic use of their activity mimetic in vascular injuries like stenting.</P>