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Hee Jin Jeong,Laurent Eude,Manoharan Gowtham,Bernd Marquardt,Sung Hun Lim,Shaïma Enouz,Costel Sorin Cojocaru,Kyung Ah Park,이영희,디디에르프리밧 성균관대학교(자연과학캠퍼스) 성균나노과학기술원 2008 NANO Vol.3 No.3
The effects of an atomic hydrogen (Hat) pretreatment of the catalyst layer on the low temperature growth of single-walled carbon nanotubes (SWCNTs) have been investigated using a modified catalytic chemical vapor deposition system. Well-defined and isolated individual Fe nanoparticles as a catalyst are successfully formed on the defects with high trapping energy which are created on the Al2O3 surface by Hat pretreatment, yielding highly dense SWCNTs. The pretreatment mechanism of Hat, compared to H2, is also discussed. It was also found that the quality of SWCNTs can be enhanced when Hat is flowed with CH4 during nanotubes growth at low temperature. In this case, the undesired carbon products and defects on catalyst seeds and nanotubes walls can be selectively removed by Hat. Therefore, it is essential to use Hat in pretreatment stage for increasing catalytic activity and keep the size of nanoparticles in the nm range. Hat can also be employed in growth stage for enhancing SWCNTs quality and density at low temperature.
Effects of Rad51 on Survival of A549 Cells
Yu, Sha-Sha,Tu, Yi,Xu, Lin-Lin,Tao, Xue-Qin,Xu, Shan,Wang, Shan-Shan,Xiong, Yi-Feng,Mei, Jin-Hong Asian Pacific Journal of Cancer Prevention 2015 Asian Pacific journal of cancer prevention Vol.16 No.1
Rad51, a key factor in the homologous recombination pathway for the DNA double-strand break repair, plays a vital role in genesis of non-small-cell lung cancer (NSCLC). In recent years, more and more studies indicate that high expression of Rad51 is of great relevance to resistance of NSCLC to chemotherapeutic agents and ionizing radiation. However, the underlying molecular mechanisms are poorly understood. In this study, we investigated the role of single Rad51 on cell viability in vitro. Our results show that depletion of endogenous Rad51 is sufficient to inhibit the growth of the A549 lung cancer cell line, by accumulating cells in G1 phase and inducing cell death. We conclude that independent Rad51 expression is critical to the survival of A549 cells and can be an independent prognostic factor in NSCLC patients.