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A method for preventing online games hacking using memory monitoring
Lee, Chang Seon,Kim, Huy Kang,Won, Hey Rin,Kim, Kyounggon Electronics and Telecommunications Research Instit 2021 ETRI Journal Vol.43 No.1
Several methods exist for detecting hacking programs operating within online games. However, a significant amount of computational power is required to detect the illegal access of a hacking program in game clients. In this study, we propose a novel detection method that analyzes the protected memory area and the hacking program's process in real time. Our proposed method is composed of a three-step process: the collection of information from each PC, separation of the collected information according to OS and version, and analysis of the separated memory information. As a result, we successfully detect malicious injected dynamic link libraries in the normal memory space.
Cdo Interacts with APPL1 and Activates AKT in Myoblast Differentiation
Bae, Gyu-Un,Lee, Jae-Rin,Kim, Bok-Geon,Han, Ji-Won,Leem, Young-Eun,Lee, Hey-Jin,Ho, Seok-Man,Hahn, Myong-Joon,Kang, Jong-Sun The American Society for Cell Biology 2010 Molecular biology of the cell Vol.21 No.14
<P>Cell–cell interactions between muscle precursors are required for myogenic differentiation; however, underlying mechanisms are largely unknown. Promyogenic cell surface protein Cdo functions as a component of multiprotein complexes containing other cell adhesion molecules, Boc, Neogenin and N-cadherin, and mediates some of signals triggered by cell–cell interactions between muscle precursors. Cdo activates p38MAPK via interaction with two scaffold proteins JLP and Bnip-2 to promote myogenesis. p38MAPK and Akt signaling are required for myogenic differentiation and activation of both signaling pathways is crucial for efficient myogenic differentiation. We report here that APPL1, an interacting partner of Akt, forms complexes with Cdo and Boc in differentiating myoblasts. Both Cdo and APPL1 are required for efficient Akt activation during myoblast differentiation. The defective differentiation of Cdo-depleted cells is fully rescued by overexpression of a constitutively active form of Akt, whereas overexpression of APPL1 fails to do so. Taken together, Cdo activates Akt through association with APPL1 during myoblast differentiation, and this complex likely mediates some of the promyogenic effect of cell–cell interaction. The promyogenic function of Cdo involves a coordinated activation of p38MAPK and Akt via association with scaffold proteins, JLP and Bnip-2 for p38MAPK and APPL1 for Akt.</P>
Modified Delphi 법을 이용한 의료 커뮤니케이션 교재 범위 설정
김대현 ( Dae Hyen Kim ),노혜린 ( Hey Rin Roh ),신좌섭 ( Jwa Seop Shin ),박훈기 ( Hoon Ki Park ),박일환 ( Eal Whan Park ),성낙진 ( Nak Jin Sung ),임인석 ( Inseok Lim ),이건호 ( Geon Ho Lee ) 대한의료커뮤니케이션학회 2011 의료커뮤니케이션 Vol.6 No.1
To determine the contents and range of the textbook of communication in healthcare, using Modified Delphi method, experts committee group was launched by Korean Association of Medical College and Korean Academy on Communication in Healthcare. Basic contents was composed after review of 4 international guidelines (SEGUE, Kalamazoo Consensus, Calgary-Cambridge Guides, Macy guide) and 2 domestic guidelines. The basic contents was revised according to professors who is in charge of medical communication in 41 Korean medical schools. 15 chapters (4 introduction chapters, 2 practice process chapters, 9 challenging situations chapters) was decided by 4 committee group meetings and 2 review questionnaires. Revision and update need to be done according to the medico-social consensus. And more specific guideline need to be formulated by many healthcare personnel.
Development of traditional wine using biomass from alcoholic fermentation by Phellinus sp. mycelium
Hak-Seob Lim,Gang-Young Lee,Eun-Jung Jung,Won-Su Lee,Young-Kwang Ju,Min-Jeong Seo,Ji-Eun Kim,Tae-Yeon Kim,Hey-Rin Lee,Kyung Tae Chung,Byung Tae Choi,Young Hee Kim,Yong Kee Jeong 한국생명과학회 2006 한국생명과학회 심포지움 Vol.47 No.-
Herbal formulation MIT ameliorates high-fat diet-induced non-alcoholic fatty liver disease
Sang-hyun Ahn,Eun-Sun Yang,Hey-Rin Cho,Syng-Ook Lee,Ki-Tae Ha,Kibong Kim 한국한의학연구원 2020 Integrative Medicine Research Vol.9 No.4
Background: Non-alcoholic fatty liver disease (NAFLD) is one of the most common liver diseases and is caused by obesity, diabetes, high blood pressure, and insulin resistance. Many studies have explored novel candidates to treat NAFLD using herbal medicines owing to their fewer side effects. In this study, we examined the effect of MIT, an herbal formula comprising Ephedra sinica, Panax ginseng, and Alisma orientale, on the murine model of NAFLD. Methods: To evaluate the effect of MIT on NAFLD, we used the high-fat diet (HFD)-induced NAFLD mice model. The mice were divided into four groups: control, HFD, HFD with metformin administration, and HFD with MIT administration. Freeze-dried MIT was dissolved in phosphate buffered saline and orally administered for 8 weeks to MIT-treated mice (60 mg/kg) after feeding them with HFD for 16 weeks. Results: MIT treatment significantly attenuated fat accumulation, serum glucose levels, and excessive cholesterol. It also reduced the activation of NF-κB, JNK, ERK, mammalian target of rapamycin, and peroxisome proliferator-activated receptor γ in the HFD-induced NAFLD mice. The expression level of enzymes involved in the synthesis and oxidation of fatty acids, acetyl-coA carboxylase and CYP2E1, were clearly reduced by MIT treatment. Reactive oxygen species (ROS) production and subsequent liver damage were effectively reduced by MIT treatment. Conclusion: We suggest that MIT is a potent herbal formula that can be used for the prevention and treatment of obesity-related NAFLD via regulating the levels of serum glucose and free fatty acids, inflammation, lipid accumulation, and ROS-mediated liver damage.