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SEMG-based Upper Trapezius-specific Emotional Assessment System; Design and Implementation
Li, Gang,Chen, Hai-Feng,Gil, Yeong-Joon,Wu, Wan-Qing,Lee, Jung-Tae The Korean Society of Medical and Biological Engin 2009 의공학회지 Vol.30 No.5
Some serious neck diseases are closely related to negative emotion. In order to explain the etiology deeply, we assumed that upper trapezius is innervated by emotional motor system (EMS), a special motor system. And then we developed an upper trapezius-specific surface electromyography acquisition system concerned with its special innervation to prove our assumption. Through a targeted experiment, we found that upper trapezius is indeed innervated by EMS.
김태형(Tae-Hyoung Kim),이동희(Dong-Hee Lee),안진우(Jin-Woo Ahn),하병길(Byung-Gil Ha),강병주(Byung-Joo Gang) 대한전기학회 2008 대한전기학회 학술대회 논문집 Vol.2008 No.10
This paper present in-wheel BLDC motor using parallel winding. The voltage of proposed motor is higher than Y-connection three phase BLDC motor in order to separated connection. When the stator resistance and inductance are stable, maximum phase current and maximum torque is increased by high injected voltage. The proposed motor is verified from experimental result of the 2[㎾] in-wheel motor on electric vehicle drive.
KS F 2862의 단일수치평가량에 의한 건축용 건식 벽체의 차음성능 분류
장길수(Jang Gil-Soo),이태강(Lee Tae-Gang),송민정(Song Min-Jeong),김선우(Kim Sun-Woo) 한국건축친환경설비학회 2008 한국건축친환경설비학회 논문집 Vol.2 No.1
KS F 2808, laboratory measurements of airborne sound insulation of building elements. was revised in 2000 and KS F 2862 was newly established in 2002. Thus Related Korean Industrial Standard such as KS F 4034, KS F 4722, KS F 4724, KS F 4734, KS F 4735, KS F 4736, KS F 4738 which are related with prefabricated walls or panels should be revised according to KS F 2808 and KS F 2862. This study aimed to elicit the single number quantity such as Rw, Rw+C, Rw+Ctr, Lm defined in KS F 2862 instead of exiting rating number; transmission loss of 500㎐. As a result, there was a good correlation between single number quantities and transmission loss of 500㎐, thus it could be possible to change the rating index from existing rating number to new single number quantities properly.
Lee, Ji-Min,Gang, Gil-Tae,Kim, Don-Kyu,Kim, Yong Deuk,Koo, Seung-Hoi,Lee, Chul-Ho,Choi, Hueng-Sik American Society for Biochemistry and Molecular Bi 2014 The Journal of biological chemistry Vol.289 No.2
<P>Small heterodimer partner interacting leucine zipper protein (SMILE) has been identified as a nuclear corepressor of the nuclear receptor (NRs) family. Here, we examined the role of SMILE in the regulation of nuclear receptor liver X receptor (LXR alpha)-mediated sterol regulatory element binding protein-1c (SREBP-1c) gene expression. We found that SMILE inhibited T0901317 (T7)-induced transcriptional activity of LXR alpha, which functions as a major regulator of lipid metabolism by inducing SREBP-1c, fatty acid synthase (FAS), and acetyl-CoA carboxylase (ACC) gene expression. Moreover, we demonstrated that SMILE physically interacts with LXR alpha and represses T7-induced LXR alpha transcriptional activity by competing with coactivator SRC-1. Adenoviral overexpression of SMILE (Ad-SMILE) attenuated fat accumulation and lipogenic gene induction in the liver of T7 administered or of high fat diet (HFD)-fed mice. Furthermore, we investigated the mechanism by which ursodeoxycholic acid (UDCA) inhibits LXR alpha-induced lipogenic gene expression. Interestingly, UDCA treatment significantly increased SMILE promoter activity and gene expression in an adenosine monophosphate-activated kinase-dependent manner. Furthermore, UDCA treatment repressed T7-induced SREBP-1c, FAS, and ACC protein levels, whereas knockdown of endogenous SMILE gene expression by adenovirus SMILE shRNA (Ad-shSMILE) significantly reversed UDCA-mediated repression of SREBP-1c, FAS, and ACC protein levels. Collectively, these results demonstrate that UDCA activates SMILE gene expression through adenosine monophosphate-activated kinase phosphorylation, which leads to repression of LXR alpha-mediated hepatic lipogenic enzyme gene expression.</P>
Kim, Don-Kyu,Gang, Gil-Tae,Ryu, Dongryeol,Koh, Minseob,Kim, Yo-Na,Kim, Su Sung,Park, Jinyoung,Kim, Yong-Hoon,Sim, Taebo,Lee, In-Kyu,Choi, Cheol Soo,Park, Seung Bum,Lee, Chul-Ho,Koo, Seung-Hoi,Choi, Hu American Diabetes Association 2013 Diabetes Vol.62 No.9
<P>Type 2 diabetes mellitus (T2DM) is a progressive metabolic disorder with diverse pathological manifestations and is often associated with abnormal regulation of hepatic glucose production. Many nuclear receptors known to control the hepatic gluconeogenic program are potential targets for the treatment of T2DM and its complications. Nevertheless, the therapeutic potential of the estrogen-related receptor γ (ERRγ) in T2DM remains unknown. In this study, we show that the nuclear receptor ERRγ is a major contributor to hyperglycemia under diabetic conditions by controlling hepatic glucose production. Hepatic ERRγ expression induced by fasting and diabetic conditions resulted in elevated levels of gluconeogenic gene expression and blood glucose in wild-type mice. Conversely, ablation of hepatic ERRγ gene expression reduced the expression of gluconeogenic genes and normalized blood glucose levels in mouse models of T2DM: <I>db</I>/<I>db</I> and diet-induced obesity (DIO) mice. In addition, a hyperinsulinemic-euglycemic clamp study and long-term studies of the antidiabetic effects of GSK5182, the ERRγ-specific inverse agonist, in <I>db</I>/<I>db</I> and DIO mice demonstrated that GSK5182 normalizes hyperglycemia mainly through inhibition of hepatic glucose production. Our findings suggest that the ability of GSK5182 to control hepatic glucose production can be used as a novel therapeutic approach for the treatment of T2DM.</P>
Ahn, Seung Won,Gang, Gil-Tae,Tadi, Surendar,Nedumaran, Balachandar,Kim, Yong Deuk,Park, Ji Hoon,Kweon, Gi Ryang,Koo, Seung-Hoi,Lee, Keesook,Ahn, Ryun-Sup,Yim, Yong-Hyeon,Lee, Chul-Ho,Harris, Robert A. American Society for Biochemistry and Molecular Bi 2012 The Journal of biological chemistry Vol.287 No.50