http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Yong-Lee의 익명 핑거프린팅 프로토콜의 안전성 취약점 및 개선 방안
손기욱(Kiwook Sohn),이윤호(Yunho Lee),원동호(Dongho Won) 한국정보보호학회 2006 정보보호학회논문지 Vol.16 No.6
2005년, Yong과 Lee는 buyer-seller 핑거프린팅 프로토콜을 제안하면서, 대칭(symmetric) 암호계와 가환 (commutative) 암호계를 이용하기 때문에 속도가 빠르고 익명성을 갖는다고 주장한 바 있다. 그러나 이 방식은 공격자가 man-in-the-middle 방법을 이용하여 공격할 경우 정당한 사용자의 핑거프린트가 포함된 콘텐츠를 얻을 수 있는 문제가 있다. 본 논문에서는 Yong-Lee 방식의 안전성 취약점을 살펴보고 이를 막을 수 있는 방안을 제시한다. In 2005, Yong and Lee proposed a buyer-seller fingerprinting protocol using symmetric and commutative encryptions. They claimed that their protocol was practical and anonymous since they used symmetric and commutative encryptions. However, an attacker can get the content embedded with one or more honest buyers' fingerprints using man-in-the-middle attack. In this letter, we point out the weakness and propose methods for improving to their protocol.
Lee, Da Hye,Shin, Ji-Sun,Kang, Shin-Young,Lee, Seung-Bin,Lee, Jin Su,Ryu, Seung Mok,Lee, Kyung Tae,Lee, Dongho,Jang, Dae Sik American Chemical Society and American Society of 2018 Journal of natural products Vol.81 No.6
<P>An activity-guided fractionation procedure of the 70% aqueous EtOH extract from the roots of <I>Patrinia scabra</I> led to the isolation and characterization of five new iridoids, patriscabrins A-E (<B>1</B>-<B>5</B>), along with 13 known compounds. The structures of <B>1</B>-<B>5</B> were determined by interpretation of spectroscopic data, particularly by 1D and 2D NMR, ECD, and VCD studies. Thereafter, isolates were evaluated for their inhibitory effects on lipopolysaccharide-induced nitric oxide production in RAW 264.7 cells. Of these, the new iridoids <B>2</B> and <B>5</B> and the known lignan patrineolignan B (<B>6</B>) exhibited IC<SUB>50</SUB> values of 14.7 to 17.8 μM.</P> [FIG OMISSION]</BR>
Phenolic compounds from the leaves of Cornus controversa
Lee, Dongho,Kang, Shin-Jung,Lee, Seung-Ho,Ro, Jaiseup,Lee, Kyongsoon,Kinghorn, A.Douglas 영남대학교 약품개발연구소 2000 영남대학교 약품개발연구소 연구업적집 Vol.10 No.-
Two novel phenolie compounds from the leaves of Cornus controversa(Cornaceae) were characterized as (-)-2.3-digalloyl-4-(E)-caffeoyl-L-threonic acid and (-)-2-galloyl-4-(E)-caffeoyl-L-threonic acid, using spectroscopic methods. ⓒ 2000 Elsevier Science Ltd. All rights reserved.
Lee, Jeong-Hyung,Jung, Haeng Sun,Giang, Phan Minh,Jin, Xuejun,Lee, Sangku,Son, Phan Tong,Lee, Dongho,Hong, Young-Soo,Lee, Kyeong,Lee, Jung Joon Williams Wilkins 2006 The Journal of pharmacology and experimental thera Vol.316 No.1
<P>Nuclear factor-kappaB (NF-kappaB) and the signaling pathways that regulate its activity have become a focal point for intense drug discovery and development efforts. NF-kappaB regulates the transcription of a large number of genes, particularly those involved in immune, inflammatory, and antiapoptotic responses. In our search for NF-kappaB inhibitors from natural resources, we identified cardamomin, 2',4'-dihydroxy-6'-methoxychalcone, as an inhibitor of NF-kappaB activation from Alpinia conchigera Griff (Zingiberaceae). In present study, we demonstrated the effect of cardamomin on NF-kappaB activation in lipopolysaccharide (LPS)-stimulated RAW264.7 cells and LPS-induced mortality. This compound significantly inhibited the induced expression of NF-kappaB reporter gene by LPS or tumor necrosis factor (TNF)-alpha in a dose-dependent manner. LPS-induced production of TNF-alpha and NO as well as expression of inducible nitric-oxide synthase and cyclooxygenase-2 was significantly suppressed by the treatment of cardamomin in RAW264.7 cells. Also, cardamomin inhibited not only LPS-induced degradation and phosphorylation of inhibitor kappaBalpha (IkappaBalpha) but also activation of inhibitor kappaB (IkappaB) kinases and nuclear translocation of NF-kappaB. Further analyses revealed that cardamomin did not directly inhibit IkappaB kinases, but it significantly suppressed LPS-induced activation of Akt. Moreover, cardamomin suppressed transcriptional activity and phosphorylation of Ser536 of RelA/p65 subunit of NF-kappaB. However, this compound did not inhibit LPS-induced activation of extracellular signal-regulated kinase and stress-activated protein kinase/c-Jun NH(2)-terminal kinase, but significantly impaired activation of p38 mitogen-activated protein kinase. We also demonstrated that pretreatment of cardamomin rescued C57BL/6 mice from LPS-induced mortality in conjunction with decreased serum level of TNF-alpha. Together, cardamomin could be valuable candidate for the intervention of NF-kappaB-dependent pathological condition such as inflammation.</P>