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Sodium Nitroprusside(SNP)에 의한 유도저혈압이 폐흉일동맥혈 산소분압차 및 폐션트율에 미치는 영향
권무일,정윤국 대한마취과학회 1985 Korean Journal of Anesthesiology Vol.18 No.4
The effect of induced hypotension with sodium nitroprusside(SNP) on alveolar-arterial oxygen tension difference and pulmonary shunting in 10 surgical patients whith normal pulmonary function was studied under general anesthesia. Heart rate, mean arterial pressure, central venous pressure, blood gases and hemoglobin were measured before, during and after SNP infusion. Using the above data, alveolar-arterial oxygen tension difference and pulmonary shunting were calculated. The results were as follows. 1) Mean arterial pressure decreased significantly from 98±9.1 mmHg before SNP infusion to 61±3.0 mmHg during SNP infusion(p<0.001), and increased to 96±6.7 mmHg again after SNP infusion. 2) Heart rate increased significantly from 78±10.5 beats/min. to 101±11.9 beats/min. (p<0.001), and decreased to 84±11.5 beats/min. again. 3) Srterial oxygen tension decreased from 489±32.5 mmHg to 480±25.0 mmHg, but was not statistically significant.(p>0.05). 4) Alveolar-arterial oxygen tension difference increased from 188±29.0 mmHg to 196±25.0 mmHg, but was not statistically significant(p>0.05). 5) Pulmonary shunting increased from 9.0±1.83% to 9.2±1.50%, but was not statistically significant.(p>0.05) The above findings suggest that pulmonary shunting in patients with normal lung function will not affected during induced hypotension with SNP.
Sodium Nitroprusside(SNP)에 의한 유도저혈압이 동맥혈산소분압에 미치는 영향
김동수,이원용,최영규,권무일,정윤국 대한마취과학회 1983 Korean Journal of Anesthesiology Vol.16 No.4
The effect of sodium nitroprusside(SNP) on arterial oxygen tension in 20 neurosurgical patients with normal lung function was studied under general anesthesia. Blood gas, heart rate, mean arterial pressure and central venous pressure were measured before, during and after SNP infusion. The results were as follows: 1) Arterial oxygen tension significantly decreased from 177±40.6 mmHg before SNP to 138±50.1 mmHg during SNP(p$lt;0.005), and increased again to 168±44.4 mmHg after SNP. 2) Mixed venous oxygen tension decreased 47±8.2 mmHg to 40±7.3 mmHg(p$lt;0.005), and increased again to 44±10.3 mmHg. 3) Heart rate significantly increased from 92±24.6 beats/min to 118±27.4 beats/min(p$lt;0.005), and decreased again to 94±18.6 beats/min. The above findings have shown a marked reduction in PaO2 when SNP was administered during general anesthesia. After SNP, PaO2, returned to the previous values. It is suggested that the reduction in PaO2, is the result of an increased scatter of ventilation/perfusion relationships in and inhibition of hypoxic pulmonary vasoconstriction by SNP. These results provide evidence that SNP induced hypotension may cause significant impairment in pulmonary gas exchange in patients with normal lung function.