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당뇨병 환자에 있어서의 혈청 보체 C3 , C4 에 관한 연구
김용성(Yong Seong Kim),서양관(Yang Kwan Seo),오연상(Yeon Sang Oh),신순현(Soon Hyun Shin) 대한내과학회 1989 대한내과학회지 Vol.36 No.2
N/A Complement is required for the final step of the humoral immune process that leads to pancreatic islet cell damage. Recently low serum C4 levels were reported in insulin-dependent diabetes and this might be due to either complement consumption or phenotypic variation. To study the differential levels of the complement system in patients with diabetes mellitus, we measured the serum C3 and C4 levels in 5 patients with insulin dependent diabetes (Type 1), 14 patients with non-obese non-insulin dependent diabetes (Type IIa) and 14 patients with obese non-insulin dependent diabetes (Type IIb). The results were as follows: 1) The serum C3 and C4 levels in the control group were 85.4±8.16 mg/dl and 44.1±8.31 mg/dl respectively. 2) The serum C3 levels in Type I, Type Ila and Type IIb diabetics were not significantly different from that in the control group (p<0,1). 3) The serum C4 levels in Type I diabetics and Type IIa diabetics were 26.9±8.72 mg/dl and 32.2±10.9 mg/ dl, respectively, which were significantly lower than that in the control group (p<0.01, p<0.05, resepectively). However, in Type IIb diahetics, it was not significantly lower than that in the control group (p<0.1). 4) The serum C4 levels were not different between groups with or without diabetic complications, but sereum C3 levels in the groups with diabetic complications were higher than that in the group without diabetic complications (p<0.01). These results showed that there was a significant decrease in serum C4 levels in Type I and Type IIa diabetes mellitus. We suggested that pathogenic similarities were presented between Type I and Type IIa diabetes mellitus in the process of complement systems but further study will he required.
차영주,류왕성,김용성,서양관,현창훈,권기익,유언호 대한내과학회 1988 대한내과학회지 Vol.35 No.3
A study of a family with a hereditary deficiency of antithrombin III (AT III), as measured by an immunoassay, is reported. The propositus was a 37-year-old male with a 3-year history of recurrent attacks of deep vein thrombosis of the lower legs. A study of his family revealed a disposition to thromboembolism on the parental side and an autosomal dominant pattern of inheritance, involving five of ten siblings. They have suffered from frequent episodes of venous thrombosis of the lower legs.