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PIMODS서버에서 분산 비디오스트림의 전송을 위한 상호연결망
임강빈,류문간,신준호,김상중,최경희,정기현 대한전자공학회 1999 電子工學會論文誌, C Vol.c36 No.11
본 논문은 멀티미디어 서버의 부하 편중현상을 해결하기 위한 스위치를 제시하고 그 스위치에서의 트래픽 특성 분석을 위한 간단한 확률적 모델을 제안한다. 스위치는 경로설정 방안으로 우회방안을 이용하므로 스위치 내의 트래픽 부하는 우회확률에 커다란 영향을 미친다 본 논문에서는 제안한 모델에 의거하여 스위치의 트래픽 부하에 따르는 우회확률을 추적하였다. 그리고 그 결과를 실험적 결과와 비교함으로써 확률적 모델의 타당성을 검증하였다. 확률적 모델에 의하여 스위치 안으로 유입되는 패킷의 양에 따라 발생하는 우회와 그에 따르는 스위치의 트래픽 포화지점을 예측할 수 있다. This paper presents an interconnection network for load balancing on a multimedia server and proposes a simple probabilistic model of the interconnection network for analysing the traffic characteristics. Because the switch uses deflection algorithm for routing, the traffic load on the switch seriously affects deflection probability. In this paper, we trace the deflection probability as a function of the traffic load according to the model. By comparing the result with the empirical result, we prove that the model is useful for estimating the deflection probability and traffic saturation point against the amount of packets getting into the switch.
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정유채,홍승진,김영호,김성자,강석진,최상욱,류문간 대한의학회 2008 Journal of Korean medical science Vol.23 No.6
Transitional-CpG methylation between unmethylated promoters and nearby methylated retroelements plays a role in the establishment of tissue-specific transcription. This study examined whether chromosomal losses reducing the active genes in cancers can change transitional-CpG methylation and the transcription activity in a cancer-type-dependent manner. The transitional-CpG sites at the CpG-island margins of nine genes and the non-island-CpG sites round the transcription start sites of six genes lacking CpG islands were examined by methylation-specific polymerase chain reaction (PCR) analysis. The number of active genes in normal and cancerous tissues of the stomach, colon, breast, and nasopharynx were analyzed using the public data in silico. The CpG-island margins and non-island CpG sites tended to be hypermethylated and hypomethylated in all cancer types, respectively. The CpG-island margins were hypermethylated and a low number of genes were active in the normal stomach compared with other normal tissues. In gastric cancers, the CpG-island margins and non-island-CpG sites were hypomethylated in association with high-level chromosomal losses, and the number of active genes increased. Colon, breast, and nasopharyngeal cancers showed no significant association between the chromosomal losses and methylation changes. These findings suggest that chromosomal losses in gastric cancers are associated with the hypomethylation of the gene-control regions and the increased number of active genes.
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