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속명탕(續命湯)의 출처(出處), 종류(種類) 및 조성(造成)에 대한 고찰(考察)
나호정,권동렬,Na Ho-Jeong,Kwon Dong-Yeul 대한한의학방제학회 2003 大韓韓醫學方劑學會誌 Vol.11 No.2
Sokmyeungtang was the representative prescription for Apoplexy under the rule of Tang and Song dynasty of which the cultures were thriving in the history of China. However, the clinical use of Sokmyeungtang has been gradually reduced since Geumwon dynasty of China because it was misunderstood that the dryness heat drugs of pungent in flavor and warm in property such as Ephedra, Pubescent Angelica Root, Chinese Cassia Tree-Bark, Divaricate Saposhnikovia Root, Prepared Aconite Root, Fresh Ginger, and Wildginger Herb included in the presciption for Apoplexy supplemented heat as damaging Yin flood. In fact, the drugs pungent in flavor and warm in property activate exterior and interior circulation, circulate channels and collaterals, promote blood circulation, and remove blood stasis with the side effect of relieving exterior syndrome with drugs warming channels. When treating Apoplexy with Sokmyeungtang, the cold drugs such as Gypsum, Baikai Skullcap Root, and Pueraria Root are prescribed to suppress fire of pungent dryness and to control excessive heat of people with Apoplexy as reducing the effects of hot drugs causing impairment of Yin. For treatment of Apoplexy, the above drugs accelerate blood and Qi circulation in channels and collaterals and then in necrotic tissue of human body as removing blood stasis. Consequently, these drugs improve disorders of capillary tube circulation. If Sokmyeungtang, an old prescription, is properly understood, it will be substantially helpful to all kinds of treatments in clinical cases
Paeonol inhibits anaphylactic reaction by regulating histamine and TNE-α
Kim, Sung Hoon,Kim, Seung-Ae,Park, Mi-Kyung,Kim, Seung-Hyung,Park, Young-Doo,Na, Ho-Jeong,Kim, Hyung-Min,Shin, Min-Kyu,Ahn, Kyoo-Seok WHO COLLABORATING CENTRE FOR TRADITIONAL MEDICINE 2004 東西醫學硏究所 論文集 Vol.2004 No.-
Paconol, a major phenolic component of Moutan Cortex, was known to have antiaggregatory, antioxidant and antiinflammatory activities. In the present study, we tried to elucidate thc effects of paconol on anaphylactic reaction and its mode of action. Paconol significantly inhibited histamine release from the rat peritoneal mast cells (RPMCs) treated with compound 48/80, a mast cell degranulator. The release of tumor necrosis factor (TNF)-α mast cell activating cytokine was significantly suppressed in RBL-2H3 mast cells pretreated with anti-dinitrophenyl (DNP) immunoglobulin E (lgE) in a dose-dependent manner. Paconol significantly inhibited lgE production in B cells activated by anti-CD40 mAb, recombinant interlcukin-4 (rIL-4) and recombinant histamine releasing factor (rHRF). Paconol effectively downregulated the expression of IL-4 in the activated B cells by reverse transcription polymerase chain reaction (RT- PCR). We also confirmed that paconol effectivcly inhibited anaphylactic shock in mice by 90% at a dose of 0.5 mg/mouse versus PBS treated control 2 h after the i.p. injection of compound 48/80. These results suggest that paconol has antianaphylatic activity by regulating histamine and TNF-α.