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Hong, Seokjo,Yoo, Jin,Park, Nojin,Lee, Sang Moon,Park, Je-Geun,Park, Ji Hoon,Son, Seung Uk The Royal Society of Chemistry 2015 Chemical communications Vol.51 No.100
<P>This work shows the new engineering strategy of magnetic adsorbents by the combination of zeolitic imidazolate framework (ZIF) and microporous organic network (MON) chemistry. ZIF-67 nanoparticles containing Co<SUP>2+</SUP> ions were coated with MON. The thermolysis of ZIF-67@MON under argon resulted in hollow carbon materials bearing cobalt nanoparticles which showed promising performance as magnetic adsorbents for aromatic pollutants in water.</P> <P>Graphic Abstract</P><P>ZIF-67 nanoparticles containing Co<SUP>2+</SUP> ions were coated with a microporous organic network (MON). The thermolysis of ZIF-67@MON resulted in the formation of hollow Co@C materials which showed good performance as magnetic adsorbents for aromatic pollutants in water. <IMG SRC='http://pubs.rsc.org/services/images/RSCpubs.ePlatform.Service.FreeContent.ImageService.svc/ImageService/image/GA?id=c5cc06873h'> </P>
Incidence of Wolbachia and Cardinium Endosymbionts in the Osmia Community in Korea
Gilsang Jeong,Kyeongyong Lee,최지영,Seokjo Hwang,박병도,Wontae Kim,최영철,Ingyun Park,Jonggill Kim 한국미생물학회 2009 The journal of microbiology Vol.47 No.1
Sex ratio distorting endosymbionts induce reproductive anomalies in their arthropod hosts. They have recently been paid much attention as firstly texts of evolution of host-symbiont relationships and secondly potential biological control agents to control arthropod pests. Among such organisms, Wolbachia and Cardinium bacteria are well characterized. This study aims at probing such bacteria in the Osmia community to evaluate their potential utilization to control arthropod pests. Among 17 PCR tested species, Osmia cornifrons and a parasitic fly are infected with Wolbachia and a mite species is infected with Cardinium. Phylogenetic tree analyses suggest that horizontal transfer of the bacteria occurred between phylogenetically distant hosts.
Park, Joon Hyun,Ko, Ju Hong,Hong, Seokjo,Shin, Young Jun,Park, Nojin,Kang, Sungah,Lee, Sang Moon,Kim, Hae Jin,Son, Seung Uk American Chemical Society 2015 Chemistry of materials Vol.27 No.17
<P><B>Graphic Abstract</B> <IMG SRC='http://pubs.acs.org/appl/literatum/publisher/achs/journals/content/cmatex/2015/cmatex.2015.27.issue-17/acs.chemmater.5b02385/production/images/medium/cm-2015-023856_0003.gif'></P><P><A href='http://pubs.acs.org/doi/suppl/10.1021/cm5b02385'>ACS Electronic Supporting Info</A></P>
Son, Sung Min,Cha, Moon-Yong,Choi, Heesun,Kang, Seokjo,Choi, Hyunjung,Lee, Myung-Shik,Park, Sun Ah,Mook-Jung, Inhee Taylor and Francis 2016 AUTOPHAGY Vol.12 No.5
<P>The secretion of proteins that lack a signal sequence to the extracellular milieu is regulated by their transition through the unconventional secretory pathway. IDE (insulin-degrading enzyme) is one of the major proteases of amyloid beta peptide (A beta), a presumed causative molecule in Alzheimer disease (AD) pathogenesis. IDE acts in the extracellular space despite having no signal sequence, but the underlying mechanism of IDE secretion extracellularly is still unknown. In this study, we found that IDE levels were reduced in the cerebrospinal fluid (CSF) of patients with AD and in pathology-bearing AD-model mice. Since astrocytes are the main cell types for IDE secretion, astrocytes were treated with A beta. A beta increased the IDE levels in a time- and concentration-dependent manner. Moreover, IDE secretion was associated with an autophagy-based unconventional secretory pathway, and depended on the activity of RAB8A and GORASP (Golgi reassembly stacking protein). Finally, mice with global haploinsufficiency of an essential autophagy gene, showed decreased IDE levels in the CSF in response to an intracerebroventricular (i.c.v.) injection of A beta. These results indicate that IDE is secreted from astrocytes through an autophagy-based unconventional secretory pathway in AD conditions, and that the regulation of autophagy is a potential therapeutic target in addressing A beta pathology.</P>