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Analysis of Operation Characteristics of a Diesel Generator Set with Constant‑Power Pulsed Load
Meng Shi,Puyu Wang,Jinquan Wang,Haichao Li,Ye Xu,Kefeng Huang,Jianke Li 대한전기학회 2019 Journal of Electrical Engineering & Technology Vol.14 No.6
To improve the power quality of a diesel generator set with a pulsed load in a microgrid, this paper studied the operation characteristics of the system and proposed pulsed load conditions for an actual project based on test data. For a low average pulse power and a high peak power, a new mathematical model of the pulsed load in diferent operation modes was proposed. Then, a simulation model of the microgrid was established in MATLAB/Simulink, and the operation of the system was studied considering changes in various pulsed load parameters (the peak power Pk, output duty cycle D and switching period Ts). It was verifed by tests that the simulation model is efective and can refect the operation of the system. Based on the simulation model, the operation characteristics of the system with a bivariate pulsed load were studied, and qualifcation conditions for the diesel generator set in an actual project were proposed.
The Hyponormal Toeplitz operators on the vector valued Bergman space
Yufeng Lu,Puyu Cui,Yanyue Shi 대한수학회 2014 대한수학회보 Vol.51 No.1
In this paper, we give a necessary and sufficient condition for the hyponormality of the block Toeplitz operators TΦ, where Φ = F +G∗, F(z), G(z) are some matrix valued polynomials on the vector valued Bergman space L2 a(D, Cn). We also show some necessary conditions for the hyponormality of TF+G* with F + G∗ 2 h∞ ⓧ Mn×n on L2 a(D, Cn). In this paper, we give a necessary and sufficient condition for the hyponormality of the block Toeplitz operators TΦ, where Φ = F +G∗, F(z), G(z) are some matrix valued polynomials on the vector valued Bergman space L2 a(D, Cn). We also show some necessary conditions for the hyponormality of TF+G* with F + G∗ ∈ h∞ ⓧ Mn×n on L2 a(D, Cn).
THE HYPONORMAL TOEPLITZ OPERATORS ON THE VECTOR VALUED BERGMAN SPACE
Lu, Yufeng,Cui, Puyu,Shi, Yanyue Korean Mathematical Society 2014 대한수학회보 Vol.51 No.1
In this paper, we give a necessary and sufficient condition for the hyponormality of the block Toeplitz operators $T_{\Phi}$, where ${\Phi}$ = $F+G^*$, F(z), G(z) are some matrix valued polynomials on the vector valued Bergman space $L^2_a(\mathbb{D},\mathbb{C}^n)$. We also show some necessary conditions for the hyponormality of $T_{F+G^*}$ with $F+G^*{\in}h^{\infty}{\otimes}M_{n{\times}n}$ on $L^2_a(\mathbb{D},\mathbb{C}^n)$.
Yang, Tian,Li, Hong,Chen, Tianjun,Ren, Hui,Shi, Puyu,Chen, Mingwei Korean Society for Molecular and Cellular Biology 2019 Molecules and cells Vol.42 No.3
This study was aimed to explore if lncRNA MALAT1 would modify chemo-resistance of non-small cell lung cancer (NSCLC) cells by regulating miR-197-3p and p120 catenin (p120-ctn). Within this investigation, we totally recruited 326 lung cancer patients, and purchased 4 NSCLC cell lines of A549, H1299, SPC-A-1 and H460. Moreover, cisplatin, adriamycin, gefitinib and paclitaxel were arranged as chemotherapies, and half maximal inhibitory concentration (IC50) values were calculated to evaluate the chemo-resistance of the cells. Furthermore, mice models of NSCLC were also established to assess the impacts of MALAT1, miR-197-3p and p120-ctn on tumor growth. Our results indicated that MALAT1 and miR-197-3p were both over-expressed within NSCLC tissues and cells, when compared with normal tissues and cells (P < 0.05). The A549, H460, SPC-A-1 and SPC-A-1 displayed maximum resistances to cisplatin ($IC50=15.70{\mu}g/ml$), adriamycin ($IC50=5.58{\mu}g/ml$), gefitinib ($96.82{\mu}mol/L$) and paclitaxel (141.97 nmol/L). Over-expression of MALAT1 and miR-197-3p, or under-expression of p120-ctn were associated with promoted viability and growth of the cancer cells (P < 0.05), and they could significantly strengthen the chemo-resistance of cancer cells (P < 0.05). MALAT1 Wt or p120-ctn Wt co-transfected with miR-197-3p mimic was observed with significantly reduced luciferase activity within NSCLC cells (P < 0.05). Finally, the NSCLC mice models were observed with larger tumor size and weight under circumstances of over-expressed MALAT1 and miR-197-3p, or under-expressed p120-ctn (P < 0.05). In conclusion, MALAT1 could alter chemo-resistance of NSCLC cells by targeting miR-197-3p and regulating p120-ctn expression, which might assist in improvement of chemo-therapies for NSCLC.
Tian Yang,Hong Li,Tianjun Chen,Hui Ren,Puyu Shi,Mingwei Chen 한국분자세포생물학회 2019 Molecules and cells Vol.42 No.3
This study was aimed to explore if lncRNA MALAT1 would modify chemo-resistance of non-small cell lung cancer (NSCLC) cells by regulating miR-197-3p and p120 catenin (p120-ctn). Within this investigation, we totally recruited 326 lung cancer patients, and purchased 4 NSCLC cell lines of A549, H1299, SPC-A-1 and H460. Moreover, cisplatin, adriamycin, gefitinib and paclitaxel were arranged as chemotherapies, and half maximal inhibitory concentration (IC50) values were calculated to evaluate the chemo-resistance of the cells. Furthermore, mice models of NSCLC were also established to assess the impacts of MALAT1, miR-197-3p and p120-ctn on tumor growth. Our results indicated that MALAT1 and miR-197-3p were both over-expressed within NSCLC tissues and cells, when compared with normal tissues and cells (P < 0.05). The A549, H460, SPC-A-1 and SPC-A-1 displayed maximum resistances to cisplatin (IC50 = 15.70 μg/ml), adriamycin (IC50 = 5.58 μg/ml), gefitinib (96.82 μmol/L) and paclitaxel (141.97 nmol/L). Over-expression of MALAT1 and miR-197-3p, or under-expression of p120-ctn were associated with promoted viability and growth of the cancer cells (P < 0.05), and they could significantly strengthen the chemo-resistance of cancer cells (P < 0.05). MALAT1 Wt or p120-ctn Wt co-transfected with miR-197-3p mimic was observed with significantly reduced luciferase activity within NSCLC cells (P < 0.05). Finally, the NSCLC mice models were observed with larger tumor size and weight under circumstances of over-expressed MALAT1 and miR-197-3p, or under-expressed p120-ctn (P < 0.05). In conclusion, MALAT1 could alter chemo-resistance of NSCLC cells by targeting miR-197-3p and regulating p120-ctn expression, which might assist in improvement of chemo-therapies for NSCLC.