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Jiao Yifan,Zhang Sihui,Yang Jing,Lai Xin-He,Dong Kui,Cheng Yanpeng,Xu Mingchao,Zhu Wentao,Lu Shan,Jin Dong,Pu Ji,Huang Ying,Liu Liyun,Wang Suping,Xu Jianguo 한국미생물학회 2022 The journal of microbiology Vol.60 No.2
Two facultatively anaerobic, short rod-shaped, non-motile, Gram-stain-positive, unknown bacterial strains (JY-X040T and JY-X174) were isolated from fluvial sediments of Tongtian River in Yushu Tibetan Autonomous Prefecture, Qinghai province, China. Cells formed translucent, gray, round and convex colonies, with a diameter of less than 0.5 mm after 5 days of incubation at 30°C on brain heart infusion-5% sheep blood agar. The 16S rRNA gene sequence similarity between strain JY-X040T and Fudania jinshanensis 313T is 93.87%. In the four phylogenetic trees constructed based on the 16S rRNA gene and 423 core genes, the two isolates form an independent branch, phylogenetically closest to F. jinshanensis 313T, but could not be classified as a member of the genus Fudania or any other genus of the family Arcanobacteriaceae. The DNA G + C content of strain JY-X040T was 57.8%. Calculation results of average nucleotide identity, digital DNADNA hybridization value and amino acid identity between strain JY-X040T and F. jinshanensis 313T are 69.9%, 22.9%, and 64.1%. The major cellular fatty acids were C16:0 (23%) and C18:1ω9c (22%). The cell-wall peptidoglycan type was A5α (L-Lys-L-Ala-L-Lys-D-Glu). The polar lipids comprised diphosphatidylglycerol, phosphatidylglycerol, phosphatidylinositol, phosphatidylinositol mannoside and four unidentified components. The whole-cell sugars contained rhamnose and ribose. MK-10(H4) was the sole respiratory quinone. The minimum inhibitory concentration of streptomycin was 32 μg/ml. All physiological, biochemical, chemotaxonomic and genomic characteristics support that strains JY-X040T and JY-X174 represent members of a novel species in a new genus, Changpingibacter yushuensis gen. nov., sp. nov. The type strain is JY-X040T (GDMCC 1.1996T = KCTC 49514T).
Study on the Heterogeneous Reaction of SO₂ and NO₂ on Coal Ash
Ru-Jiao Song,Zi-Xiang Xu,Joo-Chang Park,Hueon Namkung,Li-Hua Xu,Hyung-Taek Kim 한국열환경공학회 2019 한국열환경공학회 학술대회지 Vol.2019 No.춘계
Harmful substances such as sulfur dioxide produced by coal combustion are the main source of air pollution. The heterogeneous reaction of polluting gases and coal ash particles has an important impact on atmospheric pollution. The sulphate formed by the oxidation of SO2 from coal combustion is combined with the dust generated by coal combustion, resulting in a large amount of adsorbed water on the surface becoming agglomerated nucleus, which forms a dense fog, affects the visibility of the atmosphere, and endangers human health. The existing research results show that serious damage to personnel is not only polluted gases such as SO2 and primary particulate matter directly discharged from coal combustion, but mainly secondary particles such as sulfate and nitrate formed by chemical reaction of SO2 and NO2 on the surface of particulate matter. Studying the formation process of secondary particulate matter on the surface of coal ash is crucial to improve the atmospheric environment around the power plant and other institutions. The heterogeneous reaction of mixed gas SO2, NO2 and air on the surface of the coal ash particles was investigated based on the laboratory-built aerosol reactor under ambient temperature and pressure conditions. Experimental studies have found that the relative humidity, light conditions and gas concentration will affect the process of SO2 and NO2 gas in the heterogeneous transformation of coal ash into secondary particles.
Multiple-shot People Re-identify based on Feature Selection with Sparsity
Dongping Zhang,Yanjie Li,Jiao Xu,Ye Shen 보안공학연구지원센터 2015 International Journal of Hybrid Information Techno Vol.8 No.1
In a video surveillance network, it is always required to track and recognize people when they move through the environment. This paper presents a novel re-identification method for multiple-people using feature selection with sparsity. By using the multiple-shot approach, each of appearance models is created in this method. The human body is divided into five parts form which the features of color, height, gradient were extracted respectively. Our appearance model is represented by linear regression method. Experimental results show that our appearance model is robust and attain a high precision rate and processing performance.
Shi Si,Gu Huijie,Xu Jinyuan,Sun Wan,Liu Caiyin,Zhu Tong,Wang Juan,Gao Furong,Zhang Jieping,Ou Qingjian,Jin Caixia,Xu Jingying,Chen Hao,Li Jiao,Xu Guotong,Tian Haibin,Lu Lixia 생화학분자생물학회 2023 Experimental and molecular medicine Vol.55 No.-
Excessive osteoclast activation, which depends on dramatic changes in actin dynamics, causes osteoporosis (OP). The molecular mechanism of osteoclast activation in OP related to type 1 diabetes (T1D) remains unclear. Glia maturation factor beta (GMFB) is considered a growth and differentiation factor for both glia and neurons. Here, we demonstrated that Gmfb deficiency effectively ameliorated the phenotype of T1D-OP in rats by inhibiting osteoclast hyperactivity. In vitro assays showed that GMFB participated in osteoclast activation rather than proliferation. Gmfb deficiency did not affect osteoclast sealing zone (SZ) formation but effectively decreased the SZ area by decreasing actin depolymerization. When GMFB was overexpressed in Gmfb-deficient osteoclasts, the size of the SZ area was enlarged in a dose-dependent manner. Moreover, decreased actin depolymerization led to a decrease in nuclear G-actin, which activated MKL1/SRF-dependent gene transcription. We found that pro-osteoclastogenic factors (Mmp9 and Mmp14) were downregulated, while anti-osteoclastogenic factors (Cftr and Fhl2) were upregulated in Gmfb KO osteoclasts. A GMFB inhibitor, DS-30, targeting the binding site of GMFB and Arp2/3, was obtained. Biocore analysis revealed a high affinity between DS-30 and GMFB in a dose-dependent manner. As expected, DS-30 strongly suppressed osteoclast hyperactivity in vivo and in vitro. In conclusion, our work identified a new therapeutic strategy for T1D-OP treatment. The discovery of GMFB inhibitors will contribute to translational research on T1D-OP.