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Dispersion of carbon nanotubes in aluminum improves radiation resistance
So, Kang Pyo,Chen, Di,Kushima, Akihiro,Li, Mingda,Kim, Sangtae,Yang, Yang,Wang, Ziqiang,Park, Jong Gil,Lee, Young Hee,Gonzalez, Rafael I.,Kiwi, Miguel,Bringa, Eduardo M.,Shao, Lin,Li, Ju unknown 2016 Nano energy Vol.22 No.-
<P><B>Abstract</B></P> <P>We can mass-produce metal/carbon nanotube (CNT) composites that show improved radiation tolerance. The 0.5wt% Al+CNT composite showed improved tensile strength without reduction of tensile ductility before radiation, and reduced void/pore generation and radiation embrittlement at high displacements per atom (DPA). Under helium ion irradiation up to 72DPA, the 1D carbon nanostructures survive, while sp<SUP>2</SUP> bonded graphene transforms to sp<SUP>3</SUP> tetrahedral amorphous carbon. Self-ion (Al) irradiation converts CNTs to a metastable form of Al<SUB>4</SUB>C<SUB>3</SUB>, but still as slender 1D nanorods with prolific internal interfaces that catalyze recombination of radiation defects, reducing radiation hardening and porosity generation. The 1D fillers may also form percolating paths of “nano-chimneys” that outgas the accumulated helium and other fission gases, providing an essential solution to the gas accumulation problem.</P> <P><B>Highlights</B></P> <P> <UL> <LI> Dispersion of CNTs showed improved tensile strength without reduction of ductility. </LI> <LI> CNTs in Al reduced void/pore generation and radiation embrittlement at high DPA. </LI> <LI> Under He ion irradiation, 1D CNTs survive, while sp<SUP>2</SUP> bond transform to sp<SUP>3</SUP> carbon. </LI> <LI> Formation of metastable 1D Al<SUB>4</SUB>C<SUB>3</SUB> from CNTs still recombine radiation defects. </LI> <LI> This nanocomposite can help improve bulk properties for nuclear applications. </LI> </UL> </P> <P><B>Graphical abstract</B></P> <P>[DISPLAY OMISSION]</P>
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서호진,최으뜸,송장훈,마경복,조예슬,I-Ju Chen,양상진 한국국제농업개발학회 2020 韓國國際農業開發學會誌 Vol.32 No.4
1. 감은 대만의 경제 과수 작물 중 하나로 세계 8위의 생산국이다. 1990년 재배면적은 1,650 ha 정도 였으나, 이후 빠르게 증가하여 2010년에는 5.348 ha로 약 20년 동안 3.2배 이상 늘었다. 2. 현재 떫은감의 재배면적은 약 1,000 ha 정도이고, 단감의 재배면적은 약 4,400 ha를 차지하고 있다. 생산량은 2000년까지는 평균 1.5~2.5만 톤 수준이었으나 2010년대 이후에는 평 균 6.7만 톤으로 3배 이상 증가하였다. 3. 최대 주산지는 타이중(Taizhong, 3,413 ha)으로 전체 생산 량의 약 67%를 차지하며, 기타 주산 지역은 자이(Chiayi, 469 ha), 미아오리(Miaoli, 473 ha), 신주(Hsinchu, 274 ha) 등으로 해발 800~1,100 m 이상의 산간 지역에서 재배되고 있다. 4. 대만 감의 출하 시기는 9월~12월 사이이나, 9~10월에 집 중하여 출하되고 있다. 9~10월에는 ‘Syh Jou’, ‘Bovine Heart’ 등 떫은감, 11~12월에는 ‘부유’, ‘차랑’, ‘화어소’ 등 단감 품종이 출하되고 있다. 5. 2010년 이후 감 생산량 증가에 따라 수입량은 줄어들고 있으며, 수출량은 늘어나고 있다. 주요 수입국은 일본, 뉴질랜드이며, 수출은 중국 홍콩 위주의 아시아지역을 대상으로 하고 있다. 6. 대만에서 감의 연구는 초기에는 재배 적지 선정과 떫은 감 품종의 탈삽을 위한 연구가 수행되었으며, 최근에는 조생종 품종 육성을 위한 연구가 수행되고 있다. Persimmon is one of Taiwan's economic fruit crops. Taiwan is the 8th largest persimmon producer in the world, and its cultivation area markedly increased from about 1,650 ha in 1990 to 5,348 ha in 2010, 3.2 times increase over 20 years, but no significant change during the last 10 years. Astringent persimmons occupy about 1,000 ha of the acreage and sweet persimmons about 4,400 ha. Its tonnage averaged 1.5 to 2.5 metric tons until 2000, but since then increased to about 6.7 metric tons. The major producing district is Taichung (3,413 ha) which comprises 67% of total persimmon cultivation areas. The other main producing districts, mountainous areas of 800~1100 meters or more above sea level, include Chiayi (469 ha), Miaoli (473 ha), and Hsinchu (274 ha). Of the persimmon fruits, astringent varieties such as ‘Syh Jou’ in and ‘Bovine Heart’ are circulated mainly in September to October, and the sweet ones such as ‘Fuyu’, ‘Jiro’ and ‘Hanagosho’ in November-December. Since 2010, the increase in the domestic persimmon production has led to its decreased import volumes and the expanded export volumes. The fruits, mainly, are imported from Japan and New Zealand, and the exported to Asian countries including China and Hong Kong. Research on Taiwanese persimmons was initially focused on selecting a suitable cultivation site and removing astringency after harvest for astringent persimmon varieties, and recently breeding early-growing varieties for earlier harvest and encouraging exports.
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Kuo-Feng Hua,A-Ching Chao,Ting-Yu Lin,Wan-Tze Chen,Yu-Chieh Lee,Wan-Han Hsu,Sheau-Long Lee,Hsin-Min Wang,Ding-I. Yang,Tz-Chuen Ju 고려인삼학회 2022 Journal of Ginseng Research Vol.46 No.4
Background: Huntington's disease (HD) is a neurodegenerative disorder caused by the expansion oftrinucleotide CAG repeat in the Huntingtin (Htt) gene. The major pathogenic pathways underlying HDinvolve the impairment of cellular energy homeostasis and DNA damage in the brain. The protein kinaseataxia-telangiectasia mutated (ATM) is an important regulator of the DNA damage response. ATM isinvolved in the phosphorylation of AMP-activated protein kinase (AMPK), suggesting that AMPK plays acritical role in response to DNA damage. Herein, we demonstrated that expression of polyQ-expandedmutant Htt (mHtt) enhanced the phosphorylation of ATM. Ginsenoside is the main and most effectivecomponent of Panax ginseng. However, the protective effect of a ginsenoside (compound K, CK) in HDremains unclear and warrants further investigation. Methods: This study used the R6/2 transgenic mouse model of HD and performed behavioral tests,survival rate, histological analyses, and immunoblot assays. Results: The systematic administration of CK into R6/2 mice suppressed the activation of ATM/AMPK andreduced neuronal toxicity and mHTT aggregation. Most importantly, CK increased neuronal density andlifespan and improved motor dysfunction in R6/2 mice. Conversely, CK enhanced the expression of Bcl2protected striatal cells from the toxicity induced by the overactivation of mHtt and AMPK. Conclusions: Thus, the oral administration of CK reduced the disease progression and markedlyenhanced lifespan in the transgenic mouse model (R6/2) of HD.
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Lim, Byung-Kwan,Xiong, Dingding,Dorner, Andrea,Youn, Tae-Jin,Yung, Aaron,Liu, Taylor I.,Gu, Yusu,Dalton, Nancy D.,Wright, Adam T.,Evans, Sylvia M.,Chen, Ju,Peterson, Kirk L.,McCulloch, Andrew D.,Yajim American Society for Clinical Investigation 2008 The Journal of clinical investigation Vol.118 No.8
<P>The coxsackievirus and adenovirus receptor (CAR) is a transmembrane protein that belongs to the family of adhesion molecules. In the postnatal heart, it is localized predominantly at the intercalated disc, where its function is not known. Here, we demonstrate that a first degree or complete block of atrioventricular (AV) conduction developed in the absence of CAR in the adult mouse heart and that prolongation of AV conduction occurred in the embryonic heart of the global CAR-KO mouse. In the cardiac-specific CAR-KO (CAR-cKO) mouse, we observed the loss of connexin 45 localization to the cell-cell junctions of the AV node but preservation of connexin 40 and 43 in contracting myocardial cells and connexin 30.2 in the AV node. There was also a marked decrease in beta-catenin and zonula occludens-1 (ZO-1) localization to the intercalated discs of CAR-cKO mouse hearts at 8 weeks before the mice developed cardiomyopathy at 21 weeks of age. We also found that CAR formed a complex with connexin 45 via its PSD-95/DigA/ZO-1-binding (PDZ-binding) motifs. We conclude that CAR expression is required for normal AV-node conduction and cardiac function. Furthermore, localization of connexin 45 at the AV-node cell-cell junction and of beta-catenin and ZO-1 at the ventricular intercalated disc are dependent on CAR.</P>
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