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노승익(Seung Ik Roh),김명선(Myeong Seon Kim),김은실(Eun Sil Kim),서두선(Du Seon Seo),정내인(Nae In Jeong),이명숙(Myeong Suk Lee),한승수(Seung Soo Han),김광희(Kwang Hoi Kim),김윤권(Yoon Kweon Kim) 대한내과학회 1994 대한내과학회지 Vol.47 No.1
N/A Background: Enalapril is an angiotensin-converting enzyme inhibitor that has been widely used in treating hypertension, congestive heart failure, DM and various renal diseases. Cough has been recognized as a side effect of angiotensin-converting enzyme inhibitors. Therefore we studied the incidence and the chracteritics of Enalapril induced cough in Korea. Method: The sixty eight patients with Enalapril who visited out-patients department of internal medicine, Han-Il General Hospital during the period from april 1992 to october 1992 were included in this study. In all patients careful history taking and review of chart was done. Patients with respiratory disease were excluded. The criteria of angiotensin converting enzyme inhibitor induced cough was that cough developed after administration of Enalapril and improved after discontinuing drug and readministration of drug induce resume of symptom. Resnlts: Subjects include 34 males, 34 females and their average age was 53.3 years, Diseases of theirs were hypertension (57 cases}, congestive heart failure (5 eases), various renal disease (28 cases) and 9 patients of them were smokers. Average drug dosage was 11.7 mg a day and average administration duration was 182 days. Of the 68 patients, 23 patients (33.8%) had chronic cough due to enalapril, which had 12 male and 11 female patients. The cough disappearing time after withdrawal of the angiotensin-converting enzyme inhibitors was average 7.8 days. In 16 patients (two thirds of them), cough disappeared within 4 days after drug withdrawal. 9 patients had to stop taking drug because of severe cough. Generally the characters of cough induced by angiotensin-converting enzyme inhibitors were dry, persistent, tickling and tended to develope especially at night, Conclusion: Cough is not uncommon side effect in angiotensin-converting enzyme inhibitor users. There fore clinician should consider angiotensin-converting enzyme inhibitor induced cough initially when cough developed after angiotensin-converting enzyme inhibitor administration.
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갑상선암 환자에 대한 방사선옥소 치료시 물리적 선량 측정
고창순(Chang Soon Koh),이명철(Myung Chul Lee),김종순(Chong Soon Kim),김희근(Hee Geun Kim),강덕원(Duck Won Kang),송명재(Myung Jae Song),김명선(Myung Seon Kim),정내인(Nae In Jeong),이재용(Jai Yong Lee),김종호(Chong Ho Kim) 대한핵의학회 1994 핵의학 분자영상 Vol.28 No.1
N/A Radioactive iodine has been widely used in patients with thyroid cancer combined with surgical treatment. However, due to individual variations in absorption and excretion and uptake by tumor tissue of radioactive iodine, there are differences in therapeutic effect and adverse effects even if the same doses are administrated. So this study compared the therapeutic effect and radiation hazard by measuring internal radiation dose. Of total 27 patients with well differentiated thyroid cancer who had been thyroidectomized, we administered radioactive iodine 100 mCi, 150 mCi, 200 mCi. According to BEL DOSIMETRY PROTOCOL, beta and gamma ray dose were estimated from a polt of the logarithrn of the percent of dose per liter of whole blood versus day, and percent dose retained versus day using semilogarithmic paper, respectivel y. 1) Physical dose to whole blood averaged 56.54+13.02 rad in 100 mCi administered group, 76.83+ 19.97 rad in 150 mCi administered group, 95.08 +- 25.51 rad in 200 mCi administered group and there has been a significant correlation among the groups. 2) Mean percent dose retained 48 hours later was 26.34%. 3) There was no significant correlation of physical dose between absence and presence of metastasis. 4) l7 of 19 patients who has been followed up with TSH and serum throglobulin, Thallium scan were successfully ablated by radioactive iodine. 5) Leukocyte, lymphocyte, neutrophil, platelet counts all declined in 4-6 weeks and most of all were restored 3 months 1ater. 6) There was no significant correlation between physical dosimetry and biologic dosimetry. Generally administered doses of radioactive iodine (100-200 mCi) to patients with thyroid cancer postoperatively had developed transient bone marrow suppression and minimal chromosomal aberration, but they were within safety dose to blood (200 rad). And there has been no significant differences in residual dose 48 hours later between Korean and western people.
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김종순(Chong Soon Kim),김은실(Eun Sil Kim),한승수(Seung Soo Han),김희근(Hee Geun Kim),강덕원(Duck Won Kang),송명재(Myung Jae Song),이재용(Jae Yong Lee),정내인(Nae In Jeong),이명선(Myung Soon Kim),서두선(Doo Seon Seo),노승익(Seung Ik 대한핵의학회 1994 핵의학 분자영상 Vol.28 No.1
N/A Recently, there are many considerations and studies on biological effects of radiations in radiation workera, as well as in accidentally or therapeutically irradiated persons. The most practical and reliable method of dosimetry for radiation accidents is the scoring of gross chromosomal aberrations in human lymphocytes (Ydr) as a biological dosimetry. By the way, although usual doses of 131I administered therapeutically for thyroid cancer are ranging from 100 mCi to 200mCi, there are differences of absarbed doses and Ydr, ranging from 0.004 to 0.04, on equally administered 131I due to variations in metabolic characteristics, stage of tumors and physical status of subjects. In this study, We exert to obtain the dose-response relationships of 131I, as a good guide to evaluating acute effects of accidental irradiations and radiation induced leukemia or solid tumor, by in vitro induction of chromosomal aberrations. we studied the relationship between radiation dose <D) and the frequency of chromosomal aberrations (Ydr) obserbed in peripheral lymphocytes that were irradiated in vitro with 'I at doses ranging from 0.05 to 6.00 Gy. By scoring cells with unstable chromosomal aberrations (dicentric chromosomes and ring chromosomes) we obtained this linearquadratic dose response equation Ydr=0.064351 D 0.13143 D+0.045684 This dose-response relationship may be useful for evaluating acute and chronic I induced biological effects.
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