http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
양양연어축제의 참가동기가 만족 및 재방문에 미치는 영향
안경모,김주연 대한관광경영학회 2009 觀光硏究 Vol.24 No.2
본 연구는 양양연어축제 참가자들을 대상으로 참가동기요인들이 어떻게 구분되는지 알아보고 이 동기요인들이 참가자의 만족과 재방문에 어떠한 영향을 미치는지 파악해 보았다. 또한, 참가자들의 일반적 특성이나 축제관련 경험특성에 따른 동기의 차이를 살펴보아 지속적인 축제개최를 위해 충족시켜야 하는 참가자의 욕구를 알아보았다. 실증분석 결과, 양양연어축제 방문객들의 참가동기는 ‘사교ㆍ엔터테인먼트성’, ‘일상탈출성’, ‘가족친화성’, ‘축제신기성’의 네 가지 요인으로 구분되었으며, 이 네 가지 요인들과 만족 간의 영향관계 분석결과, 모든 요인이 만족에 유의한 영향을 미치는 것으로 나타났다. 이 중 ‘축제신기성’요인이 가장 높은 영향력을 가지는 것으로 분석되었으며, ‘사교ㆍ엔터테인먼트성’, ‘일상탈출성’, ‘가족친화성’요인 순으로 영향을 미치는 것으로 나타났다. 또, 축제참가자 만족은 재방문에 미치는 높은 수준의 영향력을 갖는 것으로 파악되었다. 축제참가 동기요인과 재방문 간의 영향관계에 있어서는 네 가지 동기요인 중 ‘신기성’요인만이 재방문에 유의한 영향력을 갖는 것으로 분석되었다. 축제참가자의 특성에 따른 동기의 차이를 살펴본 결과, 연령, 결혼여부, 축제방문경험에 따라 유의한 차이를 보였다. 결혼여부에 따른 동기차이를 살펴본 결과, 가족친화성과 축제신기성의 두 가지 동기요인의 차이를 보였으며, 축제참가자의 참가경험여부에 따라서 사교ㆍ엔터테인먼트성요인만이 유의한 차이를 가지는 것으로 분석되었다. Motivation of festival attendance has been used as a valuable information for promoting satisfaction and revisit. This study aimed to explore motivation of "Yang yang Salmon Festival" attendants and find the relations among motivation, satisfaction and revisit. The differences of motivation according to characteristic and satisfaction are also discussed. The results are followings : (1) Derived factors of the festival attendance motivation were 'socialization-entertainment', 'escape', 'family togetherness', and 'festival novelty'. Among these four motivation factors, 'festival novelty' had the highest influence on satisfaction. 'socialization-entertainment', 'escape', 'family also had an impact on satisfaction. While family factor was only significant independent variable to revisit. Satisfaction had an significant impact on revisit. The study also revealed that the motivation differed according to age, marriage, experience of the festival. The 30's and 40's had higher motivation of 'family togetherness' and 'festival novelty'. The married showed higher motivation than singles in 'family togetherness, ‘festival novelty'. First visitors had lower 'socialization-entertainment' motivation than experienced visitors.
사립체매개성세포사멸 억제를 통한 prostaglandin E2의 7-ketocholesterol 유발 세포독성 보호효과
안경모,이승연,한정호,김두응,이정수 대한신경과학회 2009 대한신경과학회지 Vol.27 No.3
Background: It has been shown that defects in mitochondrial function are involved in the induction of neuronal cell injury. Prostanoids such as prostaglandin E2 (PGE2) are thought to play an important role in inflammation and neurologic disorders. However, the effect of PGE2 on cholesterol-oxidation-product-induced neuronal cell injury remains uncertain. Methods: The effect of PGE2 on toxicity of 7-ketocholesterol (7-KCS) was assessed in PC12 cells that were differentiated following treatment with nerve growth factor. The mitochondria-mediated apoptotic process was evaluated by examining the inhibitory effect of PGE2 on 7-KCS-induced toxicity. Results: 7-KCS induced BID cleavage, increased the production of proapoptotic Bax protein, decreased antiapoptotic Bcl-2, increased p53, and promoted cytochrome c release in the cytosolic fraction, which subsequently elicited the activation of caspase-3, DNA fragmentation, and cell death. Treatment with PGE2 inhibited this 7-KCS-induced apoptotic process and cell death. Conclusions: The results show that PGE2 inhibits 7-KCS-induced toxicity in differentiated PC12 cells by suppressing the mitochondria-mediated apoptotic process. PGE2 may protect against cholesterol-oxidation-product-induced neuronal cell injury. Background: It has been shown that defects in mitochondrial function are involved in the induction of neuronal cell injury. Prostanoids such as prostaglandin E2 (PGE2) are thought to play an important role in inflammation and neurologic disorders. However, the effect of PGE2 on cholesterol-oxidation-product-induced neuronal cell injury remains uncertain. Methods: The effect of PGE2 on toxicity of 7-ketocholesterol (7-KCS) was assessed in PC12 cells that were differentiated following treatment with nerve growth factor. The mitochondria-mediated apoptotic process was evaluated by examining the inhibitory effect of PGE2 on 7-KCS-induced toxicity. Results: 7-KCS induced BID cleavage, increased the production of proapoptotic Bax protein, decreased antiapoptotic Bcl-2, increased p53, and promoted cytochrome c release in the cytosolic fraction, which subsequently elicited the activation of caspase-3, DNA fragmentation, and cell death. Treatment with PGE2 inhibited this 7-KCS-induced apoptotic process and cell death. Conclusions: The results show that PGE2 inhibits 7-KCS-induced toxicity in differentiated PC12 cells by suppressing the mitochondria-mediated apoptotic process. PGE2 may protect against cholesterol-oxidation-product-induced neuronal cell injury.