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최재혁,김재현,신우중,최정식,류경부,이상민,박설현,이주희,임태우,Choi, Jae-Hyuk,Kim, Jae-Hyun,Shin, Woo-Jung,Choi, Jung-Sik,Ryu, Kyoung-Boo,Lee, Sang-Min,Park, Seol-Hyun,Lee, Joo-Hee,Lim, Tae-Woo 해양환경안전학회 2012 해양환경안전학회지 Vol.18 No.5
이 연구에서는 친환경 선박용 재료로 각광받는 탄소나노물질에 대하여 실험적 연구를 수행하였다. 탄소나노물질의 합성을 위한 열원으로서는 대향류 메탄 화염을 이용하였다. 탄소나노물질 합성을 위한 촉매로서는 페로센을 사용하였다. 합성 특성을 파악하기 위한 주요 파라메타로는 대향류 메탄 화염에 수소의 혼합 비율과 샘플링 위치를 변화시켰다. 탄소나노물질의 성향은 SEM과 TEM 이미지를 이용하여 결정되었다. 실험 결과로서는 수소의 혼합 비율이 증가할수록 탄소나노물질의 생성이 잘 이루어졌다. 또한 대향류 메탄 확산화염 내 탄소나노튜브의 생성을 위한 적정 온도로는 1500 K 정도가 적당하다는 것을 알 수 있었다. In this study, experimental studies were performed for the carbon nanomaterial(CNM) which is catching on as a material for eco-ship. The opposed-flow methane flame was used as a heat source for synthesis of CNM. Ferrocene was used as a catalyst for the synthesis of CNM. These major parameters were $H_2$ mixing rate and sampling positions that synthesize CNMs in opposed-flow diffusion flames. The propensities of CNMs were experimentally determined using SEM and TEM images. The experimental result showed that the amount of CNTs was increased with increasing $H_2$ concentration. It can also be found that the optimal temperature in opposed-flow methane flame for synthesis of CNT was about 1500 K.
Ketamine이 돼지 관상동맥의 Ca(2+)Channel과 K(+)Channel에 미치는 영향
김경현,김교상,서정국,유희구,염종훈,신우중,김경현 대한마취과학회 1997 Korean Journal of Anesthesiology Vol.32 No.4
Background: Ketamine produces increasing in heart rate and arterial blood pressure, in vivo. However, the direct effects of ketamine itself on the porcine coronary arteries are not well determined. In this study, the direct effects of ketamine on the porcine coronary artery responses to vasoactive agents that operate through Ca2+ channel, K+ channels and endothelium related mechanisms were investigated, in vitro. Methods: Adult porcine hearts(n=12) were obtained from a slaughter house. Coronary arteries were perfused and dissected with 4oC Krebs solution, and were cut into vessel rings and prepared with and without the endothelium(3∼4mm in length). The ring segments were suspended in tissue bath(5ml) filled with Krebs solution at 37oC and bubbled with 95% O2-5% CO2 gas mixture. The effect of ketamine(5 10 5, 10 4, 2 10 4M) on vascular smooth muscle tone caused by Ca2+[voltage operated channel(VOC), receptor operated channel(ROC)] and K+channels(Ca2+activated K+ currents, ATP-sensitive K+ currents) regulationwere studied with Ca2+ free solution and K+channel blocker. Results: Ketamine induced vasorelaxation of porcine coronary rings that were precontracted by KCl(50 mM) or acetylcholine(3 10 7M). The changes of vascular tone in endothelium intact and removed group did not show statistical significance. In ketamine pretreated group(Ca2+ free solution), after ketamine pretreatment, the last vascular tone was same as that relaxed by ketamine. The other group that without pretreatment of ketamine, the last vascular tone was same as that precontracted with KCl or acetylcholine. In the TEA pretreated group, the porcine coronary artery relaxation was reversed. However, pretreatment with glybenclamide, the porcine coronary artery relaxation was not reversed. Conclusions: Ketamine induced vasorelaxation of the porcine coronary artery as concentration relating manner, in vitro. The vasorelaxation induced by ketamine was not associated with endothelium. Furthermore, an antagonism of Ca2+ channels(VOC, ROC) and activation of Ca2+ activated K+ channels may be responsible for the porcine coronary arterial relaxing effect of ketamine. (Korean J Anesthesiol 1997; 32: 504∼509)