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체외 수정에 의한 임신 중 발견된 과자극 황체 난소 낭종 염전
김봉석 ( Bong Suk Seok ),김윤숙 ( Yoon Sook Kim ),서정호 ( Jeong Ho Seo ),송은석 ( Eun Suk Song ),김종민 ( Jong Min Kim ),이동운 ( Dong Woon Lee ),최홍준 ( Hong Jun Choi ),박혜인 ( Hye In Park ),배동한 ( Dong Han Bae ),양승하 ( Se 대한산부인과학회 2003 Obstetrics & Gynecology Science Vol.46 No.6
Ovarian torsion is the fifth most common condition in gynecologic surgical emergencies, with an incidence of 2.7% occuring mainly in women of reproductive age. It is an uncommon but well recognized complication of ovarian stimulation, especially when ovar
A study on frequency response of two-mass system for gyroscope applications
황영석(Young Suk Hwang),정형균(Hyoung Kyoon Jung),송은석(Eun Seok Song),백창욱(Chang Wook Baek),김용권(Yong Kweon Kim) 대한전기학회 2007 대한전기학회 학술대회 논문집 Vol.2007 No.11
This paper describes frequency response of two-mass system for gyroscope applications. The two-mass system of the proposed device is adapted to the sensing part of the gyroscope in this research. Two-mass system has two resonant peaks and wide flat region between two resonant peaks. The resonant frequency of driving part is in this flat region. Therefore, frequency tuning is not necessary for mode matching. In the proposed device, resonant frequency is designed as 7183 ㎐ in driving part. Mass ratio of two masses in sensing part is 0.1 and device size is 6 ㎜ × 6 ㎜. The device is fabricated by SiOG process. The fabricated spring width is increased from 4 ㎛ to 4.5~4.7 ㎛, and the measured resonant frequency is 8392 ㎐ in driving mode. We operated the sensing part using parallel plate of proof mass to verify the sensing part. It is confirmed the device has a wide flat region in frequency response curve and the resonant frequency of the driving part is in the wide flat region of sensing mode.
백서 대뇌 피질에서 허혈에 의한 [^3H]5-Hydroxytryptamine의 유리
장영문,송은석,노시운,김성수,김재천,김기원 의과학연구소 1995 全北醫大論文集 Vol.19 No.1
In an attempt to elucidate the mechanisms for ischemia-induced release of neurotransmitters in vitro. I examined the ischemia-induced release of [^3H]5-hydroxytryptamine(5-HT) from cerebral cortex of the rat. Ischemia, deprivation of oxygen and glucose, induced significant release of [^3H]5-HT(about 6% of total tissue content) from the cerebral cortex in vitro. This ischemia-induced release of [^3H]5-HT from the cerebral cortex was significantly attenuated by TTX(1μM), Mg^2+(1.2mM), MK-801(10μM), ketamine(10μM), NMDA receptor antagonists, DNQX(30μM), a kainate/AMPA receptor antagonist, or carbetapentane(30μM), an inhibitor of glutamate release. Dantrolene(30μM) and ryanodine(100μM), inhibitors of intracellular Ca^2+ release, flunarizine(5μM) and ω-conotxin(100μM), inihbitors of N-type Ca^2+ channels, signficantly attenuated the ischemia-induced release of [^3H]5-HT, but verapamil(5μM), and inhibitor of L-type Ca^2+ chnnels, did not. Fluoxetine(1μM), a relatively selective 5-HT transporter blocker, significantly inhibited the ischemia-induced release of [^3H]5-HT. These results suggest that ischemia-evoked release of norepinephrine was caused by release of glutamate via activation of NMDA and non-NMDA receptors, and that CA^2+-dependent and -independent release processes are underlying in this phenomenon.
백서 대뇌 피질에서 Norepinephrine 효과에 대한 Opioids의 영향
정남,정회상,송은석,장광철,김성수,조규박 의과학연구소 1995 全北醫大論文集 Vol.19 No.1
It has been reported that there are differences among the species in the opioid receptor types mediating the inhibitory effect of opioids. Previous studies suggested that the inhibition of high K^+-or elecrically-stimulated release of [^3H]norepinephrine ([^3H]NE) is mediated mainly by μ-opioid receptor. We characterized receptors mediating opioid-induced inhibitation of high potassium(20mM)-evoked release of [^3H]NE from slices of rat cortex incubated in vitro by comparison of the pA_2 values of the competitive antagonists, naloxone, D-Pen-Cys-Try-D-Orn-Thr-Pen_The-NH_2(CTOP), nor-binaltophimine(nor-BNI), naltrindole and bremazocine against the agonists, Tyr-D-Al-Gly-(Me)Phe-(Gly-ol) (DAMGO), Tyr-D-Arg-Phe-Sar(TAPS), [D-Ser^2, Leu^5]enkephaliln-Thr(Ds-LET), β-endorphin(1-31), β-END(1-26) and ethylketocyclazocine(EKC). All the opiod agonists listed above inhibited high potassium-evoked release of [^3H]NE in a dose-dependent manner. (5a, 7a, 8b)-(-)-N-Met-N-(7-[1-py-rolidinyl]-1-oxaspiron [4, 5]-Dec-8-yl)benzenacetamide(U-69,593), a k_1 receptor selective agonisy, and [D-Pen^2, D-Pen^5]enkephalin(DPDPE), a selective δ receptor agonist, did not ingibit[^3H]NE release even in 1μM no more concentration, indicating that δ and k_1 receptors are not involved. DAMGO, TAPS and DSLWT were more sensitively antagonized by μ-preferential opioid antagonists, CTOP and naloxone than other antagonists. TAPS and DSLET showed similar sensitivities to all tested antagonists, suggesting that they act through similar receptors. EKC and β-END(1-31) differed from other agonists and from each other in their sensitivities to most possibly of the k_2 for EKC and of the ε for β-END(1-31) These results suggest that there is heterogeneity in the opioid receptors regulating norepinephrine release in rat cortex.