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당뇨병 환자에 있어서의 혈청 보체 C3 , C4 에 관한 연구
김용성(Yong Seong Kim),서양관(Yang Kwan Seo),오연상(Yeon Sang Oh),신순현(Soon Hyun Shin) 대한내과학회 1989 대한내과학회지 Vol.36 No.2
N/A Complement is required for the final step of the humoral immune process that leads to pancreatic islet cell damage. Recently low serum C4 levels were reported in insulin-dependent diabetes and this might be due to either complement consumption or phenotypic variation. To study the differential levels of the complement system in patients with diabetes mellitus, we measured the serum C3 and C4 levels in 5 patients with insulin dependent diabetes (Type 1), 14 patients with non-obese non-insulin dependent diabetes (Type IIa) and 14 patients with obese non-insulin dependent diabetes (Type IIb). The results were as follows: 1) The serum C3 and C4 levels in the control group were 85.4±8.16 mg/dl and 44.1±8.31 mg/dl respectively. 2) The serum C3 levels in Type I, Type Ila and Type IIb diabetics were not significantly different from that in the control group (p<0,1). 3) The serum C4 levels in Type I diabetics and Type IIa diabetics were 26.9±8.72 mg/dl and 32.2±10.9 mg/ dl, respectively, which were significantly lower than that in the control group (p<0.01, p<0.05, resepectively). However, in Type IIb diahetics, it was not significantly lower than that in the control group (p<0.1). 4) The serum C4 levels were not different between groups with or without diabetic complications, but sereum C3 levels in the groups with diabetic complications were higher than that in the group without diabetic complications (p<0.01). These results showed that there was a significant decrease in serum C4 levels in Type I and Type IIa diabetes mellitus. We suggested that pathogenic similarities were presented between Type I and Type IIa diabetes mellitus in the process of complement systems but further study will he required.
정영호(Young Ho Jeong),김광철(Kwang Chul Kim),박무영(Mu Yeong Park),문현웅(Hyun Woong Moon),최창환(Chang Whan Choi),이성걸(Seong Geol Lee),서양관(Yang Kwan Seo),김종숙(Chong Sook Kim) 대한소화기학회 1994 대한소화기학회지 Vol.26 No.2
Since Abrikossoff first reported five cases of granular celll tumor of the tongue in 1926, this tumor has been reported in many different locations throughout the body. The purpose of this paper is to discuss the clinical and pathologic features in a case of gran- ular cell tomor of the esophagus. A case of granular cell tumor of the esophagus is reported. This 36 year old female patient was admitted to the hospital because of vomiting. She was re- vealed esophageal rnass on gastrofibroscopy and demonstrated granular cell tumor in histo- pathologic examination of esophageal biopsy. (Korean J Gastroenterol 1994; 26: 327-331)
차영주,류왕성,김용성,서양관,현창훈,권기익,유언호 대한내과학회 1988 대한내과학회지 Vol.35 No.3
A study of a family with a hereditary deficiency of antithrombin III (AT III), as measured by an immunoassay, is reported. The propositus was a 37-year-old male with a 3-year history of recurrent attacks of deep vein thrombosis of the lower legs. A study of his family revealed a disposition to thromboembolism on the parental side and an autosomal dominant pattern of inheritance, involving five of ten siblings. They have suffered from frequent episodes of venous thrombosis of the lower legs.
갑상선 기능 저하증에 동반된 대퇴골두의 무혈성 괴사 1 예
정영호,김종숙,김광철,문현웅,서양관,박무영 대한내과학회 1995 대한내과학회지 Vol.49 No.3
There has been no report of avascular necrosis of femoral head with hypothyroidism. Avascular necrosis of femoral head develop secondary to femur neck fracture or traumatic dislocation of hip joint But the pathogenesis of idiopathic necrosis of femoral head is not clear as that seen in trauma, Several causes linked to avascular necrosis are as follows: administration of corticosteroid, Diahetes Mellitus, alcohol drinking, sickle cell disease, gout, connective tissue disease, coagulopathy, fat embolism, iron overolad, etc. Recently we experienced a 42-year old woman complained of left hip joint pain. Radiologic finding of left hip joint showed decreased mottled density on left femoral haad. we could not find any etiologic factors described above. Only T3 and T4 were decreased and TSH was increased markedly. We started thyroid hormone replacement. 1 month later after treatment, left hip joint pain was subcided. 6 months later, previous radiologic finding was normalized on follow up study. We can not conclude that hypothyroidism caused avascular necrosis of femoral had, but we infer that hypothyroidism might influence on femoral head lesion at least.