http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
플라즈마 조건 변화에 따른 ITO 특성 분석 및 유기발광소자의 제작에 관한 연구
김중연,강성종,조재영,김태구,오환술,Kim, Joong-Yeon,Kang, Seong-Jong,Cho, Jae-Young,Kim, Tae-gu,Oh, Hwan-Sool 한국전기전자재료학회 2005 전기전자재료학회논문지 Vol.18 No.10
In this experiment, OLEDs(Organic Light Emitting Diodes) was fabricated to confirm effect of Plasma treatment which increase the hole injection characteristic from anode. Device structure was $ITO/2-TNATA/{\alpha}-NPD/DPVBi/BAlq/Alq_3/Al:Li$. We used DPVBi (4, 4 - Bis (2,2-diphenylethen-1-yls) - Biphenyl) as a blue emitting material. To optimize the process condition of plasma treatment, we used 2 gases of the oxygen and nitrogen gas under 120 mTorr with 100 W, 200 W, and 400 W plasma power. The current efficiency of $N_2$ plasma is more efficient than that of $O_2$ plasma. At $1000 cd/m^2$, we obtained the maximum current efficiency of 6.45 cd/A using $N_2$ gas with 200 W plasma power.
온도와 접촉금속이 Tricyclodecane의 열안정성에 미치는 영향
박선희(Sun Hee Park),김중연(Joong Yeon Kim),전병희(Byung-Hee Chun),권정훈(Cheong Hoon Kwon),강정원(Jeong Won Kang),한정식(Jeong Sik Han),정병훈(Byung Hun Jeong),남궁혁준(Hyuck-Joon Namkoung),김성현(Sung Hyun Kim) 한국추진공학회 2009 한국추진공학회 학술대회논문집 Vol.2009 No.5
온도가 조절되는 회분식 반응기에서 온도와 접촉금속이 Exo-tricyclo[5.2.1.02,6]decane(tricyclodecane, 이하 exo-THDCP로 표기함)의 열안정도에 미치는 영향을 GC/MS로 조사하였다. 그리고 Exo-THDCP와 접촉하는 금속의 특성은 SEM-EDX로 분석하였다. 보관 연료온도 증가 실험에서 exo-THDCP의 분해는 350℃에서 시작되는 것으로 밝혀졌다. 연료접촉 금속의 경우, 스테인리스 스틸 보다 티타늄이 exo-THDCP의 분해에 미치는 영향이 작은 것으로 나타났다. In temperature controlled batch reactor, the effect of temperature and contact metals on the thermal stability of Exo-tricyclo[5.2.1.02,6]decane (tricyclodecane, exo-THDCP) were investigated by use of GC/MS. And the characteristic of metal in contact with tricyclodecane were analyzed by SEM-EDX. In fuel temperature variation test, thermal decomposition of exo-THDCP was occurred at 350℃. In case of fuel contact metals, Titanium was less effective to decomposition of exo-THDCP than stainless steel 304, 316.
김승남,박장상,고용복,최해권,김중연 대한혈관외과학회 1997 Vascular Specialist International Vol.13 No.1
Many studies over the past few years have suggested that cigarette smoking appears to be a risk factor for disease of the cardiovascular system. Smoking has been associated with an increase in arterial thrombosis, ischemic heart disease, myocardial infarction, occlusive peripheral arterial disease, high cholesterol, and hypertension. The increased occurrence of atherosclerotic lesions in autopsied vessels from smokers has also been reported. Recent studies have shown that nicotine has a desquamating effect on endothelial cells and repeated endothelial cell injury has been suggested as an initiating factor in thrombosis and atherosclerosis. This study was conducted to define the effects of chronic oral nicotine consumption on the morphologic characteristics of rat aortic endothelial cells and their distribution. In these experiments, rats of Wistar strain, weighing 250∼300 gm, were divided into: normal control group(n=12), acute nicqtine group(n=12, 5 mg/kg/day nicotine for 1 week), chronic nicotine group(n=12, Smg/kg/day nicotine for 10 weeks), cessation group(n=12). Nicotine group were fed a stock diet and administered daily doses of nicotine(5mg/kg/day) in their drinking water. From previous human and animal studies on smoking and oral nicotine consumption, it was estimated that each rabbit consumed about as much nicotine per as a personal smoking 2∼4 packs of cigaretts per day. The type of pathologic damage included endothelial cell separation, partial loss of endothelial cell, crater, exposed basement membrane, exposed fibrillar collagen. The damaged endothelial cells among the 100 cells were counted in the scanning electron microscope and the extend of damage was expressed on the basis of percent. The results were as follows: 1) In normal control group, well-preserved endothelial lining with cobble-stone appearance was seen. 2) No significant endothelial cell damage was showed in acute nicotine group. 3) Partial erosion of endothelial lining, intercellular separation and exposure of underlying subendothelial components were examed with 56±1.57% of cell damage in chronic group. 4) No significant endothelial disruption or detachment was seen with nomal lining of in control group and acute group with 4.1±0.19% of damaged cell respectively. The aorta segments that were treated with nicotine for 10 weeks showed significantily higher degree of endothelial cell damage then there treated for only 1 week. And cessation of nicotine administration didn't decreased the endothelial cell injury. This study indicates that nicotine administered orally to rats, has a demonstrable in vivo morphologic injury effect on endothelial cells in the aorta.