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Improved AQPSO Algorithm for Solving the Model of the Skin Effect Electric Heating System
Ding Li,Ding Xinghua,Ren Weina,Lin Aiguo 대한전기학회 2022 Journal of Electrical Engineering & Technology Vol.17 No.5
In view of the importance of the skin-eff ect electric heat tracing system of the submarine oil pipeline in heating and condensation reduction, it is necessary to start from the skin electric heating mechanism and construct its complete mathematical model to realize the effi cient operation of the system. Aiming at the problem of unknown parameters in the model of the skin-eff ect electric heat tracing system, an improved adaptive quantum behaviour particle swarm optimization algorithm (AQPSO) based on the quantum behaviour particle swarm optimization algorithm is proposed, in order to eff ectively identify the model parameters. In the algorithm design, in view of the high dependence of the basic QPSO algorithm on the shrinkage and expansion coeffi cients, the particle aggregation factor is introduced, and the shrinkage and expansion coeffi cients are redesigned. In view of the diff erent proportions of the fi tness value of the individual optimal position, the weight coeffi cient is introduced to construct the best position of the average weight to realize the improvement of the basic QPSO algorithm. The simulation results show that the AQPSO algorithm has better performance than QPSO in terms of convergence accuracy and robustness. This algorithm not only overcomes the inherent premature defects of particle swarms, but also improves the accuracy of the algorithm and obtains accurate model parameters. It can be seen that the improved adaptive quantum behaviour particle swarm algorithm has certain feasibility and eff ectiveness in the parameter identifi cation of Hammerstein model.
Empirical study on the impact of PM<sub>10</sub> on mortality rate in China
( Ding Lin ),( Akira Hibiki ) 한국환경경제학회·한국자원경제학회(구 한국환경경제학회) 2018 한국환경경제학회 학술발표논문집 Vol.2018 No.하계
This paper investigates the impact of PM<sub>10</sub> on mortality throughout 31 provinces in China by using dynamic panel data model. The results show PM<sub>10</sub> has significant negative impact on mortality rate in the long term. Higher income reduces the mortality rate, as well as reduces the impact of PM<sub>10</sub> on mortality rate. Provinces with higher increasing rate in PM<sub>10</sub> bring out more increasing in mortality rate. With increasing in PM<sub>10</sub> by 0.01 mg/m<sup>3</sup> (6.9%, 24.61%), death toll will be increased by at most 0.2188 per thousand people. If the government adopt measures to reduce PM<sub>10</sub> concentration, the death toll is reduced by (285679.09, 370132.46) for reaching Chinese national standard and (1218762.97, 1549211.57) for WHO’s standard.
Ding, Rui,Lin, Chunnan,Wei, ShanShan,Zhang, Naichong,Tang, Liangang,Lin, Yumao,Chen, Zhijun,Xie, Teng,Chen, XiaoWei,Feng, Yu,Wu, LiHua Korean Society for Molecular and Cellular Biology 2017 Molecules and cells Vol.40 No.2
Previous studies have shown that bone marrow mesenchymal stromal cell (MSC) transplantation significantly improves the recovery of neurological function in a rat model of intracerebral hemorrhage. Potential repair mechanisms involve anti-inflammation, anti-apoptosis and angiogenesis. However, few studies have focused on the effects of MSCs on inducible nitric oxide synthase (iNOS) expression and subsequent peroxynitrite formation after hypertensive intracerebral hemorrhage (HICH). In this study, MSCs were transplanted intracerebrally into rats 6 hours after HICH. The modified neurological severity score and the modified limb placing test were used to measure behavioral outcomes. Blood-brain barrier disruption and neuronal loss were measured by zonula occludens-1 (ZO-1) and neuronal nucleus (NeuN) expression, respectively. Concomitant edema formation was evaluated by H&E staining and brain water content. The effect of MSCs treatment on neuroinflammation was analyzed by immunohistochemical analysis or polymerase chain reaction of CD68, Iba1, iNOS expression and subsequent peroxynitrite formation, and by an enzyme-linked immunosorbent assay of pro-inflammatory factors (IL-$1{\beta}$ and TNF-${\alpha}$). The MSCs-treated HICH group showed better performance on behavioral scores and lower brain water content compared to controls. Moreover, the MSC injection increased NeuN and ZO-1 expression measured by immunochemistry/immunofluorescence. Furthermore, MSCs reduced not only levels of CD68, Iba1 and pro-inflammatory factors, but it also inhibited iNOS expression and peroxynitrite formation in perihematomal regions. The results suggest that intracerebral administration of MSCs accelerates neurological function recovery in HICH rats. This may result from the ability of MSCs to suppress inflammation, at least in part, by inhibiting iNOS expression and subsequent peroxynitrite formation.
The Expression of Genes Related to Egg Production in the Liver of Taiwan Country Chickens
Ding, S.T.,Ko, Y.H.,Ou, B.R.,Wang, P.H.,Chen, C.L.,Huang, M.C.,Lee, Y.P.,Lin, E.C.,Chen, C.F.,Lin, H.W.,Cheng, Winston Teng Kuei Asian Australasian Association of Animal Productio 2008 Animal Bioscience Vol.21 No.1
The purpose of this study was to detect expression of genes related to egg production in Taiwan Country chickens by suppression subtractive hybridization. Liver samples of mRNA extraction from two Taiwan Country chicken strains (L2 and B), originated from the same population but with very distinct egg production rates after long-term selection for egg and meat production respectively. Two-way subtraction was performed. The hepatic cDNA from the low egg production chickens (B) was subtracted from the hepatic cDNA from the high egg production strain (L2). The reversed subtraction (L2 from B) was also performed. The resulting differentially expressed gene fragments were cloned and sequenced. We sequenced 288 clones from the forward subtraction and 96 clones from the reverse subtraction. These genes were subjected to further screening to confirm the differential expression between the two genetic breeds of chickens. The apolipoprotein B (apoB) was expressed to a greater extent in the liver of the L2 than in the B line chickens. The 5-aminoimidazole- 4-carboxamide ribonucleotide formyltransferase/IMP cyclohydrolase (PURH) was expressed to a greater extent in the liver of the B than in the L2 strain chickens. We demonstrated that both apoB and PURH were more highly expressed in the liver than that in other tissues (muscle, ovary, and oviduct) in laying Taiwan Country chickens. Taken together, these data suggest that after the selection for egg production, expression of apoB and PURH genes were also changed. Whether the changed expression of these genes is directly related to egg production is not known, but these two genes may be useful markers for egg laying performance in Taiwan Country chickens.
Lin, Lin,Yang, Jing,Ding, Yan,Wang, Jing,Ting, Liu Asian Pacific Journal of Cancer Prevention 2014 Asian Pacific journal of cancer prevention Vol.15 No.7
Background: The results of previous researches that analyzed the association between genetic polymorphisms of transcription factor-7-like 2 (TCF7L2, rs7903146) and polycystic ovary syndrome (PCOS) were conflicting. Current systematic analysis was conducted to re-explore this association using updated materials. Materials and Methods: The PubMed database was used for data collection and the final search was conducted on January 3, 2014. For TCF7L2 rs7903146, a non-signficiant slight increase in risk of PCOS development was observed under three genetic models (dominant model: OR=1.06, 95%CI: 0.93-1.21, p>0.05; recessive model: OR=1.12, 95%CI: 0.87-1.43, p> 0.05; homozygous model: OR=1.14, 95%CI: 0.87-1.47, p>0.05). In the subgroup analyses in Asian group, allele susceptibility of PCOS was calculated (allele model: OR=1.00, 95%CI: 0.74-1.35, p>0.05; dominant model: OR=0.98, 95%CI: 0.71-1.35, p>0.05; recessive model: OR=1.79, 95%CI: 0.33-9.84, p>0.05; homozygous model: OR=1.78, 95%CI: 0.32-9.80, p>0.05), the differences were again not statistically significant. Conclusions: The findings of this systemic analysis suggest that the polymorphism of TCF7L2 rs7903146 may not be associated with the susceptibility to PCOS.
Correlation between E-Cadherin-Regulated Cell Adhesion and Human Osteosarcoma MG-63 Cell Anoikis
Lin, Ding-Sheng,Cai, Le-Yi,Ding, Jian,Gao, Wei-Yang Asian Pacific Journal of Cancer Prevention 2014 Asian Pacific journal of cancer prevention Vol.15 No.19
Purpose: The aim of this study was to investigate the relationship between cell adhesion and anoikis evasion among human osteosarcoma cells (MG-63), and to further study the molecular mechanisms. Materials and Methods: Human osteosarcoma cells (MG-63) were assessed for apoptosis, and caspase-3, E-cadherin and ${\beta}$-catenin expression in EDTA and control non-EDTA groups. Results: MG-63 cells were predominantly aggregated when in suspension, and the suspended cells were more dispersed in the EDTA group. Following culture in suspension for 24 h, 48 h, or 72 h, the rates of apoptosis were $34.88%{\pm}3.64%$, $59.3%{\pm}7.22%$ and $78.5%{\pm}5.21%$ in the experimental group and $7.34%{\pm}2.13%$, $14.7%{\pm}3.69%$, and $21.4%{\pm}3.60%$ in the control group, respectively. Caspase-3 expression progressively increased and E-cadherin and ${\beta}$-catenin were decreased in the experimental group, whereas there was no change in the control group. Conclusions: MG-63 cells could avoid anoikis through cell adhesion, and E-cadherin might play a role in this process.
Shu Lin-Zhen,Zhang Xian-Lei,Ding Yi-Dan,Lin Hui 생화학분자생물학회 2024 Experimental and molecular medicine Vol.56 No.-
Neutrophils are emerging as an important player in skeletal muscle injury and repair. Neutrophils accumulate in injured tissue, thus releasing inflammatory factors, proteases and neutrophil extracellular traps (NETs) to clear muscle debris and pathogens when skeletal muscle is damaged. During the process of muscle repair, neutrophils can promote self-renewal and angiogenesis in satellite cells. When neutrophils are abnormally overactivated, neutrophils cause collagen deposition, functional impairment of satellite cells, and damage to the skeletal muscle vascular endothelium. Heterotopic ossification (HO) refers to abnormal bone formation in soft tissue. Skeletal muscle injury is one of the main causes of traumatic HO (tHO). Neutrophils play a pivotal role in activating BMPs and TGF-β signals, thus promoting the differentiation of mesenchymal stem cells and progenitor cells into osteoblasts or osteoclasts to facilitate HO. Furthermore, NETs are specifically localized at the site of HO, thereby accelerating the formation of HO. Additionally, the overactivation of neutrophils contributes to the disruption of immune homeostasis to trigger HO. An understanding of the diverse roles of neutrophils will not only provide more information on the pathogenesis of skeletal muscle injury for repair and HO but also provides a foundation for the development of more efficacious treatment modalities for HO.