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- 저자 : ( Gu Qingqing ) (검색결과 4 건)
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Gu Cuihua,Hong Sidan,Wang Jie,Shang Linxue,Zhang Guozhe,Zhao Yu,Ma Qingqing,Ma Dandan 한국원예학회 2024 Horticulture, Environment, and Biotechnology Vol.65 No.1
Lagerstroemia indica is a summer ornamental fl owering tree, belonging to the Lythraceae family. Its fl ower color is a key ornamental characteristic. Recent research indicates that WRKY and bZIP transcription factors play important role in f l ower color formation. Based on the transcriptome database of L. indica , this study identifi ed 85 LibZIPs and 61 LiWRKYs from the L. indica transcriptome database and analyzed their expression profi les using qRT-PCR. Phylogenetic analysis divided LibZIPs into 11 subfamilies and LiWRKYs into six subfamilies. Nine LibZIPs and four LiWRKYs were found to have higher expression in colored samples compared to the white sample. LibZIP31 , LibZIP65 , LibZIP34 , LiWRKY16 , LiWRKY25 , LiWRKY40 , and LiWRKY67 may play important roles in fl ower color formation in L. indica . These results provide valuable information for further functional analysis of the bZIP and WRKY genes families in L. indica and help clarify their roles in fl ower color formation.
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( Pan Su ),( Zhou Weiwei ),( Gu Qingqing ),( Ye Qiang ) 한국인터넷정보학회 2015 KSII Transactions on Internet and Information Syst Vol.9 No.1
Future wireless network aims to integrate different radio access networks (RANs) to provide a seamless access and service continuity. In this paper, a new resource denotation method is proposed in the WLAN and LTE heterogeneous networks based on a concept of spectral bandwidth mapping. This method simplifies the denotation of system resources and makes it possible to calculate system residual capacity, upon which an economic model-based network selection algorithm is designed in both under-loaded and over-loaded scenarios in the heterogeneous networks. The simulation results show that this algorithm achieves better performance than the utility function-based access selection (UFAS) method proposed in [12] in increasing system capacity and system revenue, achieving load balancing and reducing the new call blocking probability in the heterogeneous networks.
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Zhiqi Huang,Xingyu Li,Tian Xie,Changjiang Gu,Kan Ni,Qingqing Yin,Xiaolei Cao,Chunhui Zhang 대한암학회 2020 Cancer Research and Treatment Vol.52 No.4
Purpose RIOK1 has been proved to play an important role in cancer cell proliferation and migration in various types of cancers—such as colorectal and gastric cancers. However, the expression of RIOK1 in breast cancer (BC) and the relationship between RIOK1 expression and the development of BC are not well characterized. In this study, we assessed the expression of RIOK1 in BC and evaluated the mechanisms underlying its biological function in this disease context. Materials and Methods We used immunohistochemistry, western blot and quantitative real-time polymerase chain reaction to evaluate the expression of RIOK1 in BC patients. Then, knockdown or overexpression of RIOK1 were used to evaluate the effect on BC cells in vitro and in vivo. Finally, we predicted miR-204-5p could be a potential regulator of RIOK1. Results We found that the expression levels of RIOK1 were significantly higher in hormone receptor (HR)–negative BC patients and was associated with tumor grades (p=0.010) and p53 expression (p=0.008) and survival duration (p=0.011). Kaplan-Meier analysis suggested a tendency for the poor prognosis. In vitro, knockdown of RIOK1 could inhibit proliferation, invasion, and induced apoptosis in HR-negative BC cells and inhibited tumorigenesis in vivo, while overexpression of RIOK1 promoted HR-positive tumor progression. MiR-204-5p could regulate RIOK1 expression and be involved in BC progression. Conclusion These findings indicate that RIOK1 expression could be a biomarker of HR-negative BC, and it may serve as an effective prognostic indicator and promote BC progression.
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Songyi Zhou,Yizhao Pan,Yan Zhang,Lijun Gu,Leikai Ma,Qingqing Xu,Weijian Wang,Jiehao Sun 대한통증학회 2023 The Korean Journal of Pain Vol.36 No.3
Background: Spinal N-methyl-D-aspartate (NMDA) receptor activation is attributed to remifentanil-induced hyperalgesia (RIH). However, the specific mechanism and subsequent treatment is still unknown. Previous studies have shown that the dynamin-related protein 1 (DRP1)-mitochondria-reactive oxygen species (ROS) pathway plays an important role in neuropathic pain. This study examined whether antisense oligodeoxynucleotides against DRP1 (AS-DRP1) could reverse RIH. Methods: The authors first measured changes in paw withdrawal mechanical threshold (PWMT) and paw withdrawal thermal latency (PWTL) at 24 hours before remifentanil infusion and 4, 8, 24, and 48 hours after infusion. The expression levels of DRP1 and NR2B were measured after behavioral testing using Western blotting. In addition, DRP1 expression was knocked down by intrathecal administration of AS-DRP1 to investigate the effects of DRP1 on RIH. The behavioral testing, the expression levels of spinal DRP1 and NR2B, and dorsal mitochondrial superoxide were measured. Changes in mitochondrial morphology were assessed using electron microscopy. Results: After remifentanil exposure, upregulation of spinal DRP1 and NR2B was observed along with a reduction in PWMT and PWTL. In addition, AS-DRP1 improved RIH-induced PWTL and PWMT (P < 0.001 and P < 0.001) and reduced remifentanil-mediated enhancement of spinal DRP1 and NR2B expression (P = 0.020 and P = 0.022). More importantly, AS-DRP1 reversed RIH-induced mitochondrial fission (P = 0.020) and mitochondrial superoxide upregulation (P = 0.031). Conclusions: These results indicate that AS-DRP1 could modulate NMDA receptor expression to prevent RIH through the DRP1-mitochondria-ROS pathway.
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