http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
나로도 주변 해역에서 어업자원의 계절별 어획물의 종특성과 변동
민은비(Eunbi MIN),윤주원(Juwon YUN),윤은아(Eun-a YOON),황두진(Doojin HWANG) 전남대학교 어업기술연구소 2017 어업기술연구소보고지 Vol.10 No.1
Seasonal variation in catch-per-unit-effort (CPUE), species composition and size frequency distribution of major fish were analyzed by gill net, trap net, and long line in the Korean coastal area of Go-heung in 2016. The dominant fish species were Portunus trituberculatus in gill nets, Charybdis japonica in trap nets and Muraenesox cinereus in long lines. Seasonal catch was the most in July and was the least in March. Catch by fishing gear was the highest in gill net and was the lowest in long line. The dominant species are similar to seasonal biological properties. Species compositions did not vary greatly by season.
Delayed diagnosis of imperforate hymen with huge hematocolpometra: a case report
( Ji Young Hwang ),( Eunbi Jang ),( Kyeong A So ) 대한산부인과학회 2020 대한산부인과학회 학술대회 Vol.106 No.-
Imperforate hymen is a rare obstructive anomaly of the female reproductive tract. It is associated with complications include cyclical abdominal pain, urinary retention, and pelvic mass. Case: A 13-year-old girl has repeatedly visited the emergency room with lower abdominal pain for the last a year. Each time she received conservative treatment such as pain control. Because of the worsening pain, she visited to gynecological clinic and was finally diagnosed with an imperforate hymen and huge hematocolpos. A hymenectomy was performed and she had cyclic menstruation in good condition. Summary and Conclusion: Imperforate hymen should be considered when periodic abdominal pain in premenarcheal adolescent girl.
Cui, Zhen Yang,Jo, Eunbi,Jang, Hyun Jin,Hwang, In-Hu,Lee, Kyung-Bok,Yoo, Hwa-Seung,Park, Soo Jung,Jung, Mi-Kyung,Lee, Yeon Wol,Jang, Ik-Soon World Scientific Publishing Company 2018 The American journal of Chinese medicine Vol.46 No.7
<P>The cytokine C-X-C motif chemokine ligand 8 (CXCL8) is produced in the tumor microenvironment and has an important role in cancer pathogenesis. CXCL8 activates the nuclear factor (NF)-<TEX>$ \kappa $</TEX>B signaling. However, the role of NF-<TEX>$ \kappa $</TEX>B inactivation in apoptosis induced by negative regulation of CXCL8 remains unclear. Here, we assessed the effects of MRGX on the transcriptional activity of NF-<TEX>$ \kappa $</TEX>B and the expression of tumor necrosis factor (TNF)-<TEX>$ \alpha $</TEX>-stimulated target genes in liver cancer cells. Furthermore, we found that modified regular ginseng extract (MRGX)-mediated inhibition of NF-<TEX>$ \kappa $</TEX>B signaling induced apoptosis. Importantly, MRGX exerted strong activity, inhibiting TNF-<TEX>$ \alpha $</TEX>-induced expression of Akt and NF-<TEX>$ \kappa $</TEX>B in a concentration-dependent manner. Furthermore, MRGX inhibited the TNF-<TEX>$ \alpha $</TEX>-induced expression of genes encoding CXCL8, CXCL1, inducible nitric oxide synthase and intercellular adhesion molecule 1. MRGX also dowregulated Akt activation, and there was a significant decrease in Akt activation in HepG2 cells treated with CXCL8 siRNA. Conversely, CXCL8 overexpression increased Akt activation in MRGX-treated HepG2 cells. When Akt was silenced, MRGX treatment of HepG2 cells overexpressing CXCL8 decreased nuclear translocation of NF-<TEX>$ \kappa $</TEX>B, whereas Akt overexpression increased nuclear translocation of NF-<TEX>$ \kappa $</TEX>B in MRGX-treated HepG2 cells. Moreover, MRGX negatively regulated the TNF-<TEX>$ \alpha $</TEX>-mediated I<TEX>$ \kappa $</TEX>B/NF-<TEX>$ \kappa $</TEX>B pathway to promote Bax activation, resulting in caspase-3 activation and apoptosis. Taken together, these results indicated that MRGX inhibited CXCL8-mediated Akt/NF-<TEX>$ \kappa $</TEX>B signaling, which upregulated Bax activation and consequently induced apoptosis in HepG2 cells.</P>
Ik-Soon Jang,Eunbi Jo,Soo Jung Park,Su Jeong Baek,In-Hu Hwang,Hyun Mi Kang,Je-Ho Lee,권요셉,Junik Son,HO JEONG KWON,최종순 고려인삼학회 2020 Journal of Ginseng Research Vol.44 No.1
Background: The cellular senescence of primary cultured cells is an irreversible process characterized bygrowth arrest. Restoration of senescence by ginsenosides has not been explored so far. Rg3(S) treatmentmarkedly decreased senescence-associated b-galactosidase activity and intracellular reactive oxygenspecies levels in senescent human dermal fibroblasts (HDFs). However, the underlying mechanism of thiseffect of Rg3(S) on the senescent HDFs remains unknown. Methods: We performed a label-free quantitative proteomics to identify the altered proteins in Rg3(S)-treated senescent HDFs. Upregulated proteins induced by Rg3(S) were validated by real-time polymerasechain reaction and immunoblot analyses. Results: Finally, 157 human proteins were identified, and variable peroxiredoxin (PRDX) isotypes werehighly implicated by network analyses. Among them, the mitochondrial PRDX3 was transcriptionally andtranslationally increased in response to Rg3(S) treatment in senescent HDFs in a time-dependent manner. Conclusion: Our proteomic approach provides insights into the partial reversing effect of Rg3 on senescentHDFs through induction of antioxidant enzymes, particularly PRDX3.
Jang, Ik-Soon,Jo, Eunbi,Park, Soo Jung,Baek, Su Jeong,Hwang, In-Hu,Kang, Hyun Mi,Lee, Je-Ho,Kwon, Joseph,Son, Junik,Kwon, Ho Jeong,Choi, Jong-Soon The Korean Society of Ginseng 2020 Journal of Ginseng Research Vol.44 No.1
Background: The cellular senescence of primary cultured cells is an irreversible process characterized by growth arrest. Restoration of senescence by ginsenosides has not been explored so far. Rg3(S) treatment markedly decreased senescence-associated β-galactosidase activity and intracellular reactive oxygen species levels in senescent human dermal fibroblasts (HDFs). However, the underlying mechanism of this effect of Rg3(S) on the senescent HDFs remains unknown. Methods: We performed a label-free quantitative proteomics to identify the altered proteins in Rg3(S)-treated senescent HDFs. Upregulated proteins induced by Rg3(S) were validated by real-time polymerase chain reaction and immunoblot analyses. Results: Finally, 157 human proteins were identified, and variable peroxiredoxin (PRDX) isotypes were highly implicated by network analyses. Among them, the mitochondrial PRDX3 was transcriptionally and translationally increased in response to Rg3(S) treatment in senescent HDFs in a time-dependent manner. Conclusion: Our proteomic approach provides insights into the partial reversing effect of Rg3 on senescent HDFs through induction of antioxidant enzymes, particularly PRDX3.