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Ways of Telicization in Chinese Resultative Compounds
Chenchen Song,Ke Wu 한국생성문법학회 2022 생성문법연구 Vol.32 No.3
In this paper, we build on Song (2018) and undertake a comparative study of the syntactic behavior of verb-resultative (V-R) compounds in three Chinese varieties: Standard Mandarin, Dongying Mandarin, and Wenzhou Wu. They systematically differ from each other in whether they require an obligatory post-V-R morpheme and, if they do, whether the choice of that morpheme is a purely grammatical issue. We find that while both Dongying Mandarin and Wenzhou Wu require a post-V-R morpheme (whereas Standard Mandarin does not), they sharply differ in the choice of said morpheme. We attribute this cross-dialectal variation to the way Chinese varieties telicize their resultative compounds and propose an analysis based on root syntax. After laying out our analysis, which is an extension of the analysis in Song (2018), we further discuss the parameterization of the three-way variation, arguing for a parameter hierarchy-based model over a flat model.
( Chen Chen ),( Ming Zhong Sun ),( Shu Qing Liu ),( Dong Mei Yeh ),( Li Jun Yu ),( Yang Song ),( Lin Lin Gong ),( Li Hong Hao ),( Jun Hu ),( Shu Juan Shao ) 생화학분자생물학회 (구 한국생화학분자생물학회) 2010 BMB Reports Vol.43 No.8
Smad4 is involved in cancer progression and metastasis. Using a pair of human syngeneic epithelial ovarian cancer cells with low (HO-8910) and high (HO-8910PM) metastatic abilities, we aimed to reveal the role of Smad4 in ovarian cancer metastasis in vitro. Smad4 was down-regulated in HO-8910PM cell line relative to HO-8910 by implicating Smad4 was probably a potential tumor suppressor gene for ovarian cancer. Re-expression of Smad4 decreased the migration ability and inhibited the invasion capacity of HO-8910PM, while promoted the cell adhesion capacity for HO-8910PM. The stable expression of Smad4 increased the expression of E-cadherin, reduced the expression of plasminogen activator inhibitor-1 (PAI-1) and slightly down-regulated the expression of VEGF. Smad4 suppresses human ovarian cancer cell metastasis potential through its effect on the expressions of PAI-1, E-cadherin and VEGF. Results from current work implicate Smad4 might suppress the invasion and metastasis of human ovarian tumor cells through a TGF-β/Smad-mediated pathway. [BMB reports 2010; 43(8): 554-560]