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- 검색키워드
- 저자 : Yijing Chen (검색결과 3 건)
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Zhao Yijing,Shang Wenwen,Chen Nan,Jeong, GangHoan 한국문화산업학회 2016 문화산업연구 Vol.16 No.3
The rapid growth in international tourism has motivated many countries to promote their cities as tourist destinations. The host country or region of Olympic Games, World cup and EXPO got attention from the world by fastest speed and usually be chosen as the popular tourism destination. Since Pyeongchang was declared the host region for 2018 Olympic Winter Games, Korea has been preparing in many ways to improve Pyeongchang as a global city. This study research 2018 PyeongChang Winter Olympic official website information service from the perspective of destination marketing system(DMS). Then the study uses impact-range performance analysis and asymmetry analysis, which are improved versions of importance-performance analysis, to examine how visitors evaluate the offcial website information service. Results of a questionnaire survey on Chinese respondents reveal the performance of different website information service, their different levels of impact on visitors satisfaction, and the probability that they will generate satisfaction or dissatisfaction. The theoretical and practical implications of these findings are discussed. The rapid growth in international tourism has motivated many countries to promote their cities as tourist destinations. The host country or region of Olympic Games, World cup and EXPO got attention from the world by fastest speed and usually be chosen as the popular tourism destination. Since Pyeongchang was declared the host region for 2018 Olympic Winter Games, Korea has been preparing in many ways to improve Pyeongchang as a global city. This study research 2018 PyeongChang Winter Olympic official website information service from the perspective of destination marketing system(DMS). Then the study uses impact-range performance analysis and asymmetry analysis, which are improved versions of importance-performance analysis, to examine how visitors evaluate the offcial website information service. Results of a questionnaire survey on Chinese respondents reveal the performance of different website information service, their different levels of impact on visitors satisfaction, and the probability that they will generate satisfaction or dissatisfaction. The theoretical and practical implications of these findings are discussed.
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The Relaxant Effect of Propofol on Isolated Rat Intrapulmonary Arteries
Guangyan Zhang,Jianxiu Cui,Yijing Chen,Jue Ma 대한약리학회 2014 The Korean Journal of Physiology & Pharmacology Vol.18 No.5
Propofol is a widely used anesthetic. Many studies have shown that propofol has direct effects on bloodvessels, but the precise mechanism is not fully understood. Secondary intrapulmonary artery ringsfrom male rats were prepared and mounted in a Multi Myograph System. The following constrictorswere used to induce contractions in isolated artery rings: high K+ solution (60 mmol/L); U46619 solution(100 nmol/L); 5-hydroxytryptamine (5-HT; 3 μmol/L); or phenylephrine (Phe; 1 μmol/L). The relaxationeffects of propofol were tested on high K+ or U46619 precontracted rings. Propofol also was addedto induce relaxation of rings preconstricted by U46619 after pretreatment with the nitric oxide synthaseinhibitor NG-nitro-L-arginine methyl ester (L-NAME). The effects of propofol on Ca2+ influx via theL-type Ca2+ channels were evaluated by examining contraction-dependent responses to CaCl2 in theabsence or presence of propofol (10 to 300 μmol/L). High K+ solution and U46619 induced remarkablecontractions of the rings, whereas contractions induced by 5-HT and Phe were weak. Propofol induceddose-dependent relaxation of artery rings precontracted by the high K+ solution. Propofol also inducedrelaxation of rings precontracted by U46619 in an endothelium-independent way. Propofol at differentconcentrations significantly inhibited the Ca2+-induced contractions of pulmonary rings exposed to highK+-containing and Ca2+-free solution in a dose-dependent manner. Propofol relaxed vessels precontractedby the high K+ solution and U46619 in an endothelium-independent way. The mechanism forthis effect may involve inhibition of calcium influx through voltage-operated calcium channels (VOCCs)and receptor-operated calcium channels (ROCCs).
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The Relaxant Effect of Propofol on Isolated Rat Intrapulmonary Arteries
Zhang, Guangyan,Cui, Jianxiu,Chen, Yijing,Ma, Jue The Korean Society of Pharmacology 2014 The Korean Journal of Physiology & Pharmacology Vol.18 No.5
Propofol is a widely used anesthetic. Many studies have shown that propofol has direct effects on blood vessels, but the precise mechanism is not fully understood. Secondary intrapulmonary artery rings from male rats were prepared and mounted in a Multi Myograph System. The following constrictors were used to induce contractions in isolated artery rings: high $K^+$ solution (60 mmol/L); U46619 solution (100 nmol/L); 5-hydroxytryptamine (5-HT; $3{\mu}mol/L$); or phenylephrine (Phe; $1{\mu}mol/L$). The relaxation effects of propofol were tested on high $K^+$ or U46619 precontracted rings. Propofol also was added to induce relaxation of rings preconstricted by U46619 after pretreatment with the nitric oxide synthase inhibitor $N^G$-nitro-L-arginine methyl ester (L-NAME). The effects of propofol on $Ca^{2+}$ influx via the L-type $Ca^{2+}$ channels were evaluated by examining contraction-dependent responses to $CaCl_2$ in the absence or presence of propofol (10 to $300{\mu}mol/L$). High $K^+$ solution and U46619 induced remarkable contractions of the rings, whereas contractions induced by 5-HT and Phe were weak. Propofol induced dose-dependent relaxation of artery rings precontracted by the high $K^+$ solution. Propofol also induced relaxation of rings precontracted by U46619 in an endothelium-independent way. Propofol at different concentrations significantly inhibited the $Ca^{2+}$-induced contractions of pulmonary rings exposed to high $K^+$-containing and $Ca^{2+}$-free solution in a dose-dependent manner. Propofol relaxed vessels precontracted by the high $K^+$ solution and U46619 in an endothelium-independent way. The mechanism for this effect may involve inhibition of calcium influx through voltage-operated calcium channels (VOCCs) and receptor-operated calcium channels (ROCCs).
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