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Structure and energy storage properties of Re0.02 Sr0.98TiO3 (Re = La, Pr, Sm) ceramics
Wen-Qin Luo,Zong-Yang Shen 한양대학교 세라믹연구소 2016 Journal of Ceramic Processing Research Vol.17 No.4
The Re0.02Sr0.98TiO3 (ReST, Re = La, Pr, Sm) ceramics with single cubic perovskite structures were prepared by a solid statereaction method. The structure and dielectric properties of the ReST ceramics were investigated for energy storage capacitorapplications. Strongly improved dielectric constant (εr > 2000 @1 kHz) with a good bias electric field stability was achieved forthe ReST ceramics. Together with relatively low dielectric loss (tanδ ≤ 3% @1 kHz) and normally high breakdown strength(Eb > 15 kV/mm), the ReST ceramics should be very promising for energy storage capacitors in pulsed power electronics.
Gan Yong,Yang Zong-qin,He Lei,Du Chao 보안공학연구지원센터 2016 International Journal of Security and Its Applicat Vol.10 No.10
As it is known, the researches on ownership transfer issues of RFID tag have focused on a single owner. However in practice, RFID tag may have multiple owners who occupy different weights. To solve the problem, the ownership transfer protocol of multi-owner with different weights based on Lagrange has been proposed. Assuming the key is divided into n parts in this paper, and the owner gets the corresponding sub secret key according to the weight. Then, If and only if the sum of the weights of the owners involved in the restoration of the secret key is equal to or greater than t, the secret key can be obtained, or not. Therefore, security of ownership transition and flexibility in the practical application are enhanced.
Sixu Chen,Daocheng Liu,Sihao He,Lei Yang,Quanwei Bao,Hao Qin,Huayu Liu,Yufeng Zhao,Zhaowen Zong 생화학분자생물학회 2018 Experimental and molecular medicine Vol.50 No.-
Type 1 diabetes mellitus (T1DM) is a pathological condition associated with osteopenia. WNT/β-catenin signaling is implicated in this process. Trabecular and cortical bone respond differently to WNT/β-catenin signaling in healthy mice. We investigated whether this signaling has different effects on trabecular and cortical bone in T1DM. We first established a streptozotocin-induced T1DM mouse model and then constitutively activated β-catenin in osteoblasts in the setting of T1DM (T1-CA). The extent of bone loss was greater in trabecular bone than that in cortical bone in T1DM mice, and this difference was consistent with the reduction in the expression of β-catenin signaling in the two bone compartments. Further experiments demonstrated that in T1DM mice, trabecular bone showed lower levels of insulin-like growth factor-1 receptor (IGF-1R) than the levels in cortical bone, leading to lower WNT/β-catenin signaling activity through the inhibition of the IGF-1R/Akt/glycogen synthase kinase 3β (GSK3β) pathway. After β-catenin was activated in T1-CA mice, the bone mass and bone strength increased to substantially greater extents in trabecular bone than those in cortical bone. In addition, the cortical bone of the T1-CA mice displayed an unexpected increase in bone porosity, with increased bone resorption. The downregulated expression of WNT16 might be responsible for these cortical bone changes. In conclusion, we found that although the activation of WNT/ β-catenin signaling increased the trabecular bone mass and bone strength in T1DM mice, it also increased the cortical bone porosity, impairing the bone strength. These findings should be considered in the future treatment of T1DM-related osteopenia.