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( Shuji Taketomi ),( Hiroshi Inui ),( Takaki Sanada ),( Kensuke Nakamura ),( Ryota Yamagami ),( Hironari Masuda ),( Sakae Tanaka ),( Takumi Nakagawa ) 대한슬관절학회 2014 대한슬관절학회지 Vol.26 No.3
Introduction: Recently, remnant-preserving anterior cruciate ligament (ACL) reconstruction has been increasingly performed to achieve revascularization, cell proliferation, and recovery of high-quality proprioception. However, poor arthroscopic visualization makes accurate socket placement during remnant-preserving ACL reconstruction difficult. This study describes a surgical technique used to create an anatomical femoral socket with a three-dimensional (3D) fluoroscopy based navigation system during technically demanding remnant-preserving ACL reconstruction. Surgical Technique: After a reference frame was attached to the femur, an intraoperative image of the distal femur was obtained, transferred to the navigation system and reconstructed into a 3D image. A navigation computer helped the surgeon visualize the entire lateral wall of the femoral notch and lateral intercondylar ridge, even when the remnant of the ruptured ACL impeded arthroscopic visualization of the bone surface. When a guide was placed, the virtual femoral tunnel overlapped the reconstructed 3D image in real time; therefore, only minimal soft tissue debridement was required. Materials and Methods: We treated 47 patients with remnant-preserving ACL reconstruction using this system. The center of the femoral socket aperture was calculated according to the quadrant technique using 3D computed tomography imaging.Results: The femoral socket locations were considered to be an anatomical footprint in accordance with previous cadaveric studies.Conclusions: The 3D fluoroscopy-based navigation can assist surgeons in creating anatomical femoral sockets during remnant-preserving ACL reconstruction.
Makoto Kikukawa,Kikuko Taketomi,Motofumi Yoshida 한국의학교육학회 2021 Korean journal of medical education Vol.33 No.2
Purpose: Although many studies have assessed the impact of interventions to increase medical students’ empathy by improving their understanding of patients’ perspectives and feelings, the impact of hospitalization experiences remains unclear. Methods: Fifth-year medical students at Kyushu University from 2009-2013 participated in a 2-day/1-night course to provide a hospitalization experience. After the course, participants answered an online, anonymous, open-ended questionnaire created by the authors. Results: Of 488 participants, 462 provided responses (95% response rate), which were evaluated by thematic analysis. Students understood inpatients not only through their own hospitalization experience, but also through observations of and conversations with the inpatients they encountered, from a shared perspective of both. Students experienced the realities of hospital life, stress and psychological states of being an inpatient, and psychological pressure from physicians. In addition, students observed the distress of other inpatients and dedication of medical staff. Furthermore, through communication with these inpatients, students understood other inpatients’ anxiety about illness and empathy as one of the requirements of health care providers from the patients’ perspective. Conclusion: This qualitative study investigated the effectiveness of a course on the hospitalization experience. Results showed that medical students understood the perspectives, distress, and anxiety of being an inpatient, not only from their own experiences but also from observation and communication with other inpatients they encountered during their hospitalization. This experience appeared to be an effective teaching strategy for enhancing medical students’ empathy by improving their understanding of patients’ perspectives and feelings.
Snapping Pes Syndrome after Unicompartmental Knee Arthroplasty
( Hiroshi Inui ),( Shuji Taketomi ),( Ryota Yamagami ),( Keitaro Tahara ),( Sakae Tanaka ) 대한슬관절학회 2016 대한슬관절학회지 Vol.28 No.2
Snapping pes syndrome is defined as a snapping sensation in the medial knee caused by pes anserinus and rarely occurs. Snapping pes syndrome after unicompartmental knee arthroplasty (UKA) has not been reported yet. We experienced two cases with this syndrome after UKA. Conservative treatment was effective in one case, while surgical excision of the gracilis tendon was necessary to relieve painful snapping in the other case. The main cause of the first case might be posteromedial overhang of the tibial tray that reached up to 5 mm. The probable cause of the second case was posteromedial overhang of the mobile bearing.
Realizations of some contact metric manifolds as Ricci soliton real hypersurfaces
Cho, Jong Taek,Hashinaga, Takahiro,Kubo, Akira,Taketomi, Yuichiro,Tamaru, Hiroshi Elsevier 2018 Journal of geometry and physics Vol.123 No.-
<P><B>Abstract</B></P> <P>Ricci soliton contact metric manifolds with certain nullity conditions have recently been studied by Ghosh and Sharma. Whereas the gradient case is well-understood, they provided a list of candidates for the nongradient case. These candidates can be realized as Lie groups, but one only knows the structures of the underlying Lie algebras, which are hard to be analyzed apart from the three-dimensional case. In this paper, we study these Lie groups with dimension greater than three, and prove that the connected, simply-connected, and complete ones can be realized as homogeneous real hypersurfaces in noncompact real two-plane Grassmannians. These realizations enable us to prove, in a Lie-theoretic way, that all of them are actually Ricci soliton.</P>
( X Li ),( O Kwon ),( D Y Kim ),( Y Taketomi ),( M Murakami ),( H W Chang ) 영남대학교 약품개발연구소 2016 영남대학교 약품개발연구소 연구업적집 Vol.26 No.-
Background: IgEjAg-stimulated mast cells release various pro-allergic inflamma-tory mediators, including histamine, eicosanoids, and pro-inflammatory cytokines. NecroX-5, a cell permeable necrosis inhibitor, showed cytoprotective effects in both in vitro and in vivo models. However, the anti-allergic effect of NecroX-5 has not yet been investigated. The aims of this study were to evaluate the anti-allergic activity of NecroX-5 in vivo and to investigate the underlying mechanism in vitro. Methods: The anti-allergic activity of NecroX-5 was evaluated in vitro using bone marrow-derived mast cells (BMMCs) and IgE receptor-bearing RBL-2H3 or KU812 cells and in vivo using a mouse model of passive anaphylaxis. The levels of histamine, eicosanoids (PGD2 and LTC4), tumor necrosis factor-α (TNF-α), and interleukin-6 (IL-6) were measured using enzyme immunoassay kits. The mechanism underlying the action of NecroX-5 was investigated using immunoblotting, immunoprecipitation, and gene knockdown techniques. Results: NecroX-5 markedly inhibited mast cell degranulation and the synthesis of eicosanoids, TNF-α, and IL-6 by suppressing the activation of Syk, LAT, phospholipase Cγl, MAP kinases, the Akt/NF-κB pathway, and intracellular Ca2+ mobilization via the activation of phosphatase SHP-I. Oral administration of NecroX-5 effectively suppressed mast cell-dependent passive cutaneous and systemic anaphylactic reactions in a dose-dependent manner. Conclusions: NecroX-5 might be a potential candidate for the development of a novel anti-allergic agent that suppresses IgE-dependent mast cells signaling.
Hwang, Seung-Lark,Li, Xian,Lu, Yue,Jin, Ye,Jeong, Yong-Tae,Kim, Yong Deuk,Lee, In-Kyu,Taketomi, Yoshitaka,Sato, Hiroyasu,Cho, You Sook,Murakami, Makoto,Chang, Hyeun Wook Elsevier 2013 The Journal of allergy and clinical immunology Vol.132 No.3
<P><B>Background</B></P> <P>Aggregation of FcεRI activates a cascade of signaling events leading to mast cell activation, followed by inhibitory signals that turn off the activating signals. However, the overall view of negative signals in mast cells is still incomplete. Although AMP-activated protein kinase (AMPK), which is generally known as a regulator of energy metabolism, is also associated with anti-inflammation, little is known about the role of AMPK in mast cells.</P> <P><B>Objectives</B></P> <P>We investigated the role of AMPK and its regulatory mechanism in mast cells.</P> <P><B>Method</B></P> <P>The roles of AMPK in FcεRI-dependent activation of bone marrow–derived mast cells (BMMCs) were evaluated by using chemical agents, small interfering RNAs (siRNAs), or adenovirus that modulated the activity or expression of AMPK signaling components. In addition, <I>AMPKα2</I> <SUP>−/−</SUP> mice were used to verify the role of AMPK in anaphylactic models.</P> <P><B>Results</B></P> <P>FcεRI signaling and associated effector functions in BMMCs were suppressed by the AMPK activator 5-aminoimidazole-4-carboxamide-1-β-4-ribofuranoside (AICAR) and were conversely augmented by siRNA knockdown of AMPKα2 or liver kinase B1 (LKB1), an upstream kinase of AMPK. Furthermore, <I>AMPKα2</I> deficiency led to increased FcεRI-mediated BMMC activation and anaphylaxis that were insensitive to AICAR, whereas enforced expression of AMPKα2 in <I>AMPKα2</I> <SUP>−/−</SUP> BMMCs reversed the hypersensitive FcεRI signaling to normal levels. Pharmacologic inhibition or siRNA knockdown of Fyn mimicked AMPK activation, suggesting that Fyn counterregulates the LKB1-AMPK axis. Mechanistically, Fyn controlled AMPK activity by regulating LKB1 localization.</P> <P><B>Conclusions</B></P> <P>The Fyn-regulated LKB1-AMPK axis acts as a novel inhibitory module for mast cell activation, which points to AMPK activators as therapeutic drugs for allergic diseases.</P>
Kiyorni Matsuo,Tornoyuki Murata,Takanori Koga,Atsuko Kubo,Yuichi Yoshida,Yosuke Karakawa,Hiroshi Kawaguchi,Toru Tanaka,Iun Masaki,Toshikatsu Taketomi,Takeo Kitajima 한국재활복지공학회 2009 재활복지공학회논문지 Vol.2 No.1
In this paper.I will report the cases of children who are able to study at kindergarten or elementary school because they learned how to move by themselves using a moving aid before school age, and I will also discuss the development of a mobility device which allows severely disabled preschoolers to practice moving around by themselves safely and easily at home and institutions.